Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Th17 Cells/pathology"'
Autor:
Mylonas, A., Conrad, C.
Publikováno v:
Frontiers in immunology, vol. 9, pp. 2746
Chronic plaque psoriasis is a common debilitating skin disease. The identification of the pathogenic role of the TNF/IL-23/T H 17 pathway has enabled the development of targeted therapies used in the clinic today. Particularly, TNF inhibitors have be
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______1900::1c42725342dd78d0a9c3d838fe45d153
https://serval.unil.ch/notice/serval:BIB_57A9835FCAA8
https://serval.unil.ch/notice/serval:BIB_57A9835FCAA8
Autor:
Stephanie Zwicker, D. Bureik, Aleksandra Batycka-Baran, Thomas Ruzicka, Simon Rothenfusser, Andreas Schmidt, Ronald Wolf, Michel Gilliet, Eva Hattinger, Peter-Arne Gerber
Publikováno v:
PLoS ONE, Vol 12, Iss 4, p e0175153 (2017)
PloS one, vol. 12, no. 4, pp. e0175153
PLoS ONE
PloS one, vol. 12, no. 4, pp. e0175153
PLoS ONE
IL-1β is a potent player in cutaneous inflammation and central for the development of a Th17 micro-milieu in autoinflammatory diseases including psoriasis. Its production is controlled at the transcriptional level and by subsequent posttranslational
Autor:
Fatima-Ezzahra L’Faqihi, Pierre Delobel, Jacques Izopet, Christophe Pasquier, Mary Requena, Jacques Amar, Martine Dubois, Patrice Massip, Maud Mavigner, Bruno Marchou, Pascale Klopp, Karl Barange, Laurent Alric, Michelle Cazabat, Jean-Pierre Vinel
Publikováno v:
Journal of Clinical Investigation
Journal of Clinical Investigation, American Society for Clinical Investigation, 2012, 122 (1), pp.62-69. ⟨10.1172/JCI59011⟩
Journal of Clinical Investigation, 2012, 122 (1), pp.62-69. ⟨10.1172/JCI59011⟩
Journal of Clinical Investigation, American Society for Clinical Investigation, 2012, 122 (1), pp.62-69. ⟨10.1172/JCI59011⟩
Journal of Clinical Investigation, 2012, 122 (1), pp.62-69. ⟨10.1172/JCI59011⟩
International audience; Depletion of CD4+ T cells from the gut occurs rapidly during acute HIV-1 infection. This has been linked to systemic inflammation and disease progression as a result of translocation of microbial products from the gut lumen in