Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Teri Naismith"'
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 11 (2023)
Plastin-3 (PLS3) is a calcium-sensitive actin-bundling protein that has recently been linked to the development of childhood-onset osteoporosis. Clinical data suggest that PLS3 mutations lead to a defect in osteoblast function, however the underlying
Externí odkaz:
https://doaj.org/article/38f9f057efb6490cba2c215e65599369
Autor:
Brian G. Petrich, Daved H. Fremont, Hua Pan, Madhav Subramanian, Phyllis I. Hanson, Ju Young Kim, Hae Chul Park, Karen Krchma, Fadi E. Pulous, Carmen M. Halabi, Ashraf-ul Kabir, Jun Wu, Samuel A. Wickline, Dong Hun Lee, Changwon Park, Kyunghee Choi, Xiaoli Wang, Suhyun Kim, Teri Naismith
Publikováno v:
Sci Transl Med
The crosstalk between angiogenesis and immunity within the tumor microenvironment (TME) is critical for tumor prognosis. While pro-angiogenic and immunosuppressive TME promote tumor growth, anti-angiogenic and immune stimulatory TME inhibit tumor pro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::df3815771294273cc4790a554d1619bb
https://europepmc.org/articles/PMC8252962/
https://europepmc.org/articles/PMC8252962/
Autor:
Christoph Kamm, Roberta L. Beauchamp, Laurie J. Ozelius, Phyllis I. Hanson, Jeffrey W. Hewett, Xandra O. Breakefield, Heather Boston, Vijaya Ramesh, Teri Naismith
Publikováno v:
Journal of Neurochemistry. 89:1186-1194
The torsins comprise a four-member family of AAA+ chaperone proteins, including torsinA, torsinB, torp2A and torp3A in humans. Mutations in torsinA underlie early onset torsion dystonia, an autosomal dominant, neurologically based movement disorder.
Autor:
Laurie J. Ozelius, Xandra O. Breakefield, Sara M. Camp, Daniele Bergeron, Nicole A. Smith, Teri Naismith, Damien Slater, Vijaya Ramesh, Heather Boston, Jeremy D. Wilbur, Christoph Kamm, Deborah E. Schuback, Philipp Ziefer, Jeffrey W. Hewett, Phyllis I. Hanson
Publikováno v:
Journal of Neuroscience Research. 72:158-168
Most cases of early-onset torsion dystonia are caused by deletion of GAG in the coding region of the DYT1 gene encoding torsinA. This autosomal dominant neurologic disorder is characterized by abnormal movements, believed to originate from neuronal d
Autor:
Jeffrey Hewett, Philipp Ziefer, Daniele Bergeron, Teri Naismith, Heather Boston, Damien Slater, Jeremy Wilbur, Deborah Schuback, Christoph Kamm, Nicole Smith, Sara Camp, Laurie J. Ozelius, Vijaya Ramesh, Phyllis I. Hanson, Xandra O. Breakefield
Publikováno v:
Journal of Neuroscience Research; 4/15/2003, Vol. 72 Issue 2, p158-168, 11p