Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Teresia Goldberg"'
Publikováno v:
Annals of the New York Academy of Sciences. 1043:461-466
Advanced glycation end products (AGEs) are a heterogeneous group of compounds that form continuously in the body. Their rate of endogenous formation is markedly increased in diabetes mellitus, a condition in which AGEs play a major pathological role.
Autor:
Helen Vlassara, Joseph A. Vassalotti, Wejing Cai, Min Lu, Jaime Uribarri, Melpomeni Peppa, Teresia Goldberg, Suresh Baliga
Publikováno v:
American Journal of Kidney Diseases. 42:532-538
Background: Levels of advanced glycation end products (AGEs), well-known proinflammatory compounds, are markedly elevated in patients with renal failure, raising the speculation that they have a role as cardiovascular risk factors in this population.
Autor:
Min Lu, Jaime Uribarri, Helen Vlassara, Melpomeni Peppa, Teresia Goldberg, Weijing Cai, Cijiang He
Publikováno v:
Journal of the American Society of Nephrology. 14:728-731
Advanced glycation endproduct (AGE) levels are elevated in renal failure patients and may contribute to the excessive cardiovascular disease in this population. Diet-derived AGE are major contributors to the total body AGE pool. It was postulated tha
Autor:
Veronique Dardaine, Jill P. Crandall, Helen Vlassara, Melpomeni Peppa, Robert Oberstein, Weijing Cai, Elliot J. Rayfield, Teresia Goldberg
Publikováno v:
Proceedings of the National Academy of Sciences. 99:15596-15601
Diet is a major environmental source of proinflammatory AGEs (heat-generated advanced glycation end products); its impact in humans remains unclear. We explored the effects of two equivalent diets, one regular (high AGE, H-AGE) and the other with 5-f
Autor:
David Pelcovitz, Teresia Goldberg
Publikováno v:
TOPICS IN CLINICAL NUTRITION. 10:73-81
Autor:
Teresia Goldberg, Alfred E. Slonim
Publikováno v:
Journal of the American Dietetic Association. 93:1423-1430
Hepatic glycogen storage diseases (GSD) are a group of rare genetic disorders in which glycogen cannot be metabolized to glucose in the liver because of one of a number of possible enzyme deficiencies along the glycogenolytic pathway. Patients with G
Autor:
Jennifer Minikes, Frank Martiniuk, Linda Bulone, Joseph A. Galanko, Efrat Slonim, Teresia Goldberg, Alfred E. Slonim
Publikováno v:
Musclenerve. 35(1)
Adult-onset acid maltase deficiency is an inherited lysosomal skeletal-muscle disease characterized by progressive myopathy and respiratory failure, for which there is no known therapy. In an uncontrolled, prospective study, we evaluated whether adhe
Publikováno v:
The Journal of pediatrics. 137(2)
Infantile patients with acid maltase deficiency have severe hypertrophic cardiomyopathy, left ventricular outflow obstruction, and generalized muscle weakness and die before 1 year of age. We identified 12 infants with acid maltase deficiency who had
Autor:
Alfred E. Slonim, Linda Bulone, Mark A. Wingertzahn, Teresia Goldberg, Mary Beth Damore, Matthew J. McKinley
Publikováno v:
The New England journal of medicine. 342(22)
Crohn's disease is a chronic inflammatory disorder of the bowel. In a preliminary study, we evaluated whether the administration of growth hormone (somatropin) as well as a high-protein diet would ameliorate the symptoms of the disease.We randomly as
Publikováno v:
Diabetes Care. 25:1898-1899
We have read with great interest the article entitled “Dietary Fat and Meat Intake in Relation to Risk of Type 2 Diabetes in Men,” by Van Dam et al. (1), which suggests a relationship between increased consumption of animal fat and red and proces