Zobrazeno 1 - 10
of 26
pro vyhledávání: '"Teresa Wrońska-Nofer"'
Autor:
Nofer, Jolanta Jajte, Elżbieta Dziubałtowska, Wojciech Krajewski, Wojciech Wąsowicz, Konrad Rydzyński, Teresa Wrońska-Nofer, Wiesław Szymczak
Publikováno v:
Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 731:58-63
Objectives Occupational exposure to nitrous oxide (N 2 O) and/or halogenated hydrocarbons has been suggested to induce damage of genetic material, but the underlying mechanisms remain obscure. This study investigated the role of oxidative processes i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e47b0e51c14898510afab77390d082d6
https://doi.org/10.1016/j.mrfmmm.2011.10.010
https://doi.org/10.1016/j.mrfmmm.2011.10.010
Autor:
Jan Stetkiewicz, Wojciech Krajewski, Teresa Wrońska-Nofer, Małgorzata Kucharska, Wiktor Wesołowski
Publikováno v:
International Journal of Hygiene and Environmental Health. 210:133-138
Objectives The aim of this study was to assess the level of occupational exposure to nitrous oxide (N 2 O) in operating rooms (ORs), as related to different ventilation and scavenging systems used to remove waste anaesthetic gases from the work envir
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f1d7f7e7a0f2813de051db588ea4285
https://doi.org/10.1016/j.ijheh.2006.07.004
https://doi.org/10.1016/j.ijheh.2006.07.004
Autor:
Teresa Wrońska-Nofer, Wojciech Wąsowicz, Małgorzata Trzcinka-Ochocka, Jerzy-Roch Nofer, Anna Pisarska, Janusz Braziewicz, Tadeusz Hałatek, Jan Stetkiewicz
OBJECTIVES Occupational exposure to lead may produce kidney damage, but existing data on the dose range associated with nephrotoxicity are inconclusive. We here assessed renal function under conditions of low to moderate lead exposure using renal sci
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2d93c4247272f0bbef5ab89595a9e1ce
Autor:
W. Krajewski, Dobrosława Lewińska, Teresa Wrońska-Nofer, Małgorzata Stańczyk, Maciej Stępnik, Joanna Arkusz
Publikováno v:
Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 581:1-9
It has been postulated that exposure to nitrous oxide and halogenated anaesthetics is associated with various adverse health effects such as neurological and reproductive abnormalities or impairment of hepatic functions. In spite of the quite well kn
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5dc519f45f5dcc59cbdc983d288ed300
https://doi.org/10.1016/j.mrgentox.2004.10.018
https://doi.org/10.1016/j.mrgentox.2004.10.018
Autor:
Wojciech Wasowicz, Konrad Rydzyński, Wojciech Krajewski, Teresa Wrońska-Nofer, Jan Stetkiewicz, Jolanta Jajte, Jadwiga Palus, Małgorzata Kucharska
Publikováno v:
Mutation research. 666(1-2)
Occupational exposure to anaesthetics such as nitrous oxide (N(2)O) and halogenated hydrocarbons has been suggested to increase risk of genetic damage. However, the dose-dependency of genotoxic effects has not been unequivocally established and their
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2544b62df1147c047c9ce953a43c1492
https://pubmed.ncbi.nlm.nih.gov/19439331
https://pubmed.ncbi.nlm.nih.gov/19439331
Autor:
Małgorzata Kucharska, Jan Stetkiewicz, B. Pilacik, Wojciech Krajewski, J.-R. Nofer, M. Fobker, Teresa Wrońska-Nofer
Publikováno v:
British journal of anaesthesia. 99(6)
Previous studies demonstrated inactivation of vitamin B12 by nitrous oxide (N(2)O). The intraoperative exposure to N(2)O was shown to induce megaloblastic anaemia and myelopathy in subjects with subclinical vitamin B12 deficiency. In contrast, no dat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7bf000d595d07aba303816369a13ea00
https://pubmed.ncbi.nlm.nih.gov/17951609
https://pubmed.ncbi.nlm.nih.gov/17951609
Autor:
Malgorzata Wierzbicka, Teresa Wrońska-Nofer, Jerzy-Roch Nofer, Halina Bolińska, Gerd Assmann, Jan Stetkiewicz, Manfred Fobker, Helmut Schulte, Arnold von Eckardstein
Objectives Oxidative stress in plasma may be promoted by plasma thiols such as homocysteine. However, other thiols such as glutathione may also exert antioxidant effects in vitro and in vivo. To further investigate whether plasma thiols act as prooxi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7e027daf215d87dfbdcabbef85efda93
https://www.zora.uzh.ch/id/eprint/81724/
https://www.zora.uzh.ch/id/eprint/81724/
Publikováno v:
International journal of occupational medicine and environmental health. 16(4)
Ethanol may be a significant combined factor in human solvent toxicity. Lipid peroxidation has been suggested to be an important contributing mechanism involved in experimental alcohol-induced liver injury. The aim of the study was to investigate whe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8178152a620d5e558c1e6b97dd756c23
https://pubmed.ncbi.nlm.nih.gov/14964644
https://pubmed.ncbi.nlm.nih.gov/14964644
Publikováno v:
International journal of occupational medicine and environmental health. 15(1)
Isoprostanes are prostaglandin-like compounds that are produced by free radical mediated peroxidation of polyunsaturated fatty acids. There is a direct evidence showing that F2-isoprostanes can be utilized as a marker of lipid peroxidation due to the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::8b7534dc849b4b0e98b379e30fc6f222
https://pubmed.ncbi.nlm.nih.gov/12038860
https://pubmed.ncbi.nlm.nih.gov/12038860
Autor:
Jerzy-Roch Nofer, Julita Chojnowska-Jezierska, Justyna M Wisniewska-Knypl, Tadeusz Hałatek, Teresa Wrońska-Nofer
Publikováno v:
Archives of toxicology. 76(3)
There is considerable epidemiological evidence that workers exposed to carbon disulfide (CS2) develop premature atherosclerosis leading to increased rates of coronary heart disease (CHD), but mechanisms underlying this association remain obscure. The
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f4eb8660d6b9de0cd07c92000356c00b
https://pubmed.ncbi.nlm.nih.gov/11967620
https://pubmed.ncbi.nlm.nih.gov/11967620