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pro vyhledávání: '"Teodora Mateeva"'
Autor:
Kirsten Jenkins, Teodora Mateeva, István Szabó, Andre Melnik, Paola Picotti, Attila Csikász-Nagy, Edina Rosta
Publikováno v:
Computational and Structural Biotechnology Journal, Vol 18, Iss , Pp 1032-1042 (2020)
Parkinson’s disease (PD), Alzheimer’s disease (AD) and Amyotrophic lateral sclerosis (ALS) are neurodegenerative diseases hallmarked by the formation of toxic protein aggregates. However, targeting these aggregates therapeutically have thus far s
Externí odkaz:
https://doaj.org/article/7e2ec4eee7964fa3bd2acef156c3f96c
Autor:
Paola Picotti, Kirsten Jenkins, István Szabó, Andre Melnik, Edina Rosta, Teodora Mateeva, Attila Csikász-Nagy
Publikováno v:
Computational and Structural Biotechnology Journal, Vol 18, Iss, Pp 1032-1042 (2020)
Mateeva, T, Rosta, E, Csikasz-Nagy, A & Jenkins, K 2020, ' Combining data integration and molecular dynamics for target identification in α-synuclein-aggregating neurodegenerative diseases : Structural insights on Synaptojanin-1 (Synj1) ', Journal of Computational and Structural Biotechnology, vol. 18, pp. 1032-1042 . https://doi.org/10.1016/j.csbj.2020.04.010
Computational and Structural Biotechnology Journal
Computational and Structural Biotechnology Journal, 18
Mateeva, T, Rosta, E, Csikasz-Nagy, A & Jenkins, K 2020, ' Combining data integration and molecular dynamics for target identification in α-synuclein-aggregating neurodegenerative diseases : Structural insights on Synaptojanin-1 (Synj1) ', Journal of Computational and Structural Biotechnology, vol. 18, pp. 1032-1042 . https://doi.org/10.1016/j.csbj.2020.04.010
Computational and Structural Biotechnology Journal
Computational and Structural Biotechnology Journal, 18
Parkinson's disease (PD), Alzheimer's disease (AD) and Amyotrophic lateral sclerosis (ALS) are neurodegenerative diseases hallmarked by the formation of toxic protein aggregates. However, targeting these aggregates therapeutically have thus far shown
ATP13A2 is a gene encoding a protein of the P5B subfamily of ATPases and is a PARK gene. Molecular defects of the gene are mainly associated with variations of Parkinson’s Disease (PD). Despite the established importance of the protein in regulatin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7abc1dd826c6d4324d88a14078a5efd2
https://doi.org/10.1101/2021.06.01.446648
https://doi.org/10.1101/2021.06.01.446648