Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Teodor E Yordanov"'
Autor:
Lilian Schimmel, Keng Yih Chew, Claudia J Stocks, Teodor E Yordanov, Patricia Essebier, Arutha Kulasinghe, James Monkman, Anna Flavia Ribeiro dosSantos Miggiolaro, Caroline Cooper, Lucia deNoronha, Kate Schroder, Anne Karine Lagendijk, Larisa I Labzin, Kirsty R Short, Emma J Gordon
Publikováno v:
Clinical & Translational Immunology, Vol 10, Iss 10, Pp n/a-n/a (2021)
Abstract Objectives Thrombotic and microvascular complications are frequently seen in deceased COVID‐19 patients. However, whether this is caused by direct viral infection of the endothelium or inflammation‐induced endothelial activation remains
Externí odkaz:
https://doaj.org/article/fb13a66448274e4d8345aa44e4f79908
Autor:
Teodor E. Yordanov, Mikaela S. Keyser, Marco A. Enriquez Martinez, Tyron Esposito, Juliann B. Tefft, Elysse K. Morris, Larisa I. Labzin, Samantha J. Stehbens, Alan E. Rowan, Benjamin M. Hogan, Christopher S. Chen, Jan Lauko, Anne K. Lagendijk
Publikováno v:
APL Bioengineering, Vol 8, Iss 1, Pp 016108-016108-14 (2024)
Cerebral cavernous malformations (CCMs) are vascular lesions that predominantly form in blood vessels of the central nervous system upon loss of the CCM multimeric protein complex. The endothelial cells within CCM lesions are characterized by overact
Externí odkaz:
https://doaj.org/article/a60ddf22761145ba8d3826e7dbcf0896
CR4 Signaling Contributes to a DC-Driven Enhanced Immune Response Against Complement-Opsonized HIV-1
Autor:
Marta Bermejo-Jambrina, Michael Blatzer, Paula Jauregui-Onieva, Teodor E. Yordanov, Paul Hörtnagl, Taras Valovka, Lukas A. Huber, Doris Wilflingseder, Wilfried Posch
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Dendritic cells (DCs) possess intrinsic cellular defense mechanisms to specifically inhibit HIV-1 replication. In turn, HIV-1 has evolved strategies to evade innate immune sensing by DCs resulting in suboptimal maturation and poor antiviral immune re
Externí odkaz:
https://doaj.org/article/e5e2ae5d2cb3474293c79d483a1f2c7d
Autor:
Larisa I. Labzin, Keng Yih Chew, Kathrin Eschke, Xiaohui Wang, Tyron Esposito, Claudia J. Stocks, James Rae, Ralph Patrick, Helen Mostafavi, Brittany Hill, Teodor E. Yordanov, Caroline L. Holley, Stefan Emming, Svenja Fritzlar, Francesca L. Mordant, Daniel P. Steinfort, Kanta Subbarao, Christian M. Nefzger, Anne K. Lagendijk, Emma J. Gordon, Robert G. Parton, Kirsty R. Short, Sarah L. Londrigan, Kate Schroder
Publikováno v:
Science Signaling. 16
Macrophages are key cellular contributors to the pathogenesis of COVID-19, the disease caused by the virus SARS-CoV-2. The SARS-CoV-2 entry receptor ACE2 is present only on a subset of macrophages at sites of SARS-CoV-2 infection in humans. Here, we
Autor:
Teodor E. Yordanov, Marco A. E. Martinez, Tyron Esposito, Juliann B. Tefft, Larisa I. Labzin, Samantha J. Stehbens, Alan Rowan, Benjamin M. Hogan, Christopher S. Chen, Jan Lauko, Anne K. Lagendijk
Cerebral Cavernous Malformations (CCMs) are vascular lesions that predominantly form in blood vessels of the central nervous system (CNS) upon loss of the CCM multimeric protein complex. The endothelial cells (ECs) within CCM lesions are characterise
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::57617e14dde2d78c2e134afac74b35ed
https://doi.org/10.1101/2023.03.27.534302
https://doi.org/10.1101/2023.03.27.534302
Autor:
Larisa I Labzin, Keng Yih Chew, Kathrin Eschke, Xiaohui Wang, Tyron Esposito, Claudia J Stocks, James Rae, Ralph Patrick, Helen Mostafavi, Brittany Hill, Teodor E. Yordanov, Caroline L Holley, Stefan Emming, Svenja Fritzlar, Francesca L. Mordant, Daniel P. Steinfort, Kanta Subbarao, Christian M. Nefzger, Anne K Lagendijk, Emma Gordon, Robert Parton, Kirsty R. Short, Sarah L. Londrigan, Kate Schroder
Macrophages are key cellular contributors to COVID-19 pathogenesis. Whether SARS-CoV-2 can enter macrophages, replicate and release new viral progeny remains controversial. Similarly, whether macrophages need to sense replicating virus to drive cytok
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::044ba46e4dbba825f16517895671c1ea
https://doi.org/10.1101/2022.03.22.485248
https://doi.org/10.1101/2022.03.22.485248
Autor:
Tevin CY. Chau, Teodor E. Yordanov, Jason A. da Silva, Scott Paterson, Alpha S. Yap, Benjamin M. Hogan, Anne Karine Lagendijk
The assembly of a mature vascular network involves the coordinated control of cell shape changes to regulate morphogenesis of a complex vascular network. Cellular changes include a process of endothelial cell (EC) elongation which is essential for es
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9ec214a044cc394fc4f1c081b3cf7e64
https://doi.org/10.1101/2022.02.01.478747
https://doi.org/10.1101/2022.02.01.478747
Autor:
Tevin C. Y. Chau, Mikaela S. Keyser, Jason A. Da Silva, Elysse K. Morris, Teodor E. Yordanov, Kinga P. Duscyz, Scott Paterson, Alpha S. Yap, Benjamin M. Hogan, Anne Karine Lagendijk
Publikováno v:
Development (Cambridge, England). 149(23)
The assembly of a mature vascular network involves coordinated endothelial cell (EC) shape changes, including the process of EC elongation. How EC elongation is dynamically regulated in vivo is not fully understood. Here, we have generated a zebrafis
Autor:
Keng Yih Chew, Emma Gordon, Claudia J. Stocks, Essebier T, Kate Schroder, Lilian Schimmel, Teodor E. Yordanov, James Monkman, Anne K. Lagendijk, Arutha Kulasinghe, dos Santos Miggiolaro Afr, Kirsty R. Short, de Noronha L, Larisa I. Labzin
ObjectivesThrombotic and microvascular complications are frequently seen in deceased COVID-19 patients. However, whether this is caused by direct viral infection of the endothelium or inflammation-induced endothelial activation remains highly content
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7312926fcc8191d0ee688965d00c5039
https://doi.org/10.1101/2021.02.14.431177
https://doi.org/10.1101/2021.02.14.431177
Autor:
Christiane A. Opitz, Ulrike Rehbein, Laura Brohée, Aurelio A. Teleman, Alexander Martin Heberle, Constantinos Demetriades, Marti Cadena Sandoval, Wilhelm Palm, Jose Miguel Ramos Pittol, Matylda Macias, Saskia Trump, Katharina Kern, Bianca Berdel, Andreas von Deimling, Birgit Holzwarth, Bernadette Carroll, Ines Heiland, Ann-Sofie De Meulemeester, Mirja Tamara Prentzell, Aleksandra Siekierska, Kathrin Thedieck, Jacek Jaworski, Mark Nellist, Hannah West, Mariana E. G. de Araujo, Mathias Bockwoldt, Eduard Stefan, Friederike Reuter, Michèle Reil, Andrii Kopach, Sandra Woltering, Suvagata Roy Chowdhury, Stefan Pusch, Viktor I. Korolchuk, Ralf Baumeister, Magdalena Kedra, Justyna Zmorzynska, Julian R. Sampson, Teodor E. Yordanov, Ineke van 't Land-Kuper, Chloë Scheldeman, Omar Torres-Quesada, Anja Reintjes, Lukas A. Huber, Gianluca Figlia, Peter de Witte, Laura E. Thomas
Publikováno v:
Cell
2021, ' G3BPs tether the TSC complex to lysosomes and suppress mTORC1 signaling ', Cell, vol. 184, no. 3, pp. 655-674.e27 . https://doi.org/10.1016/j.cell.2020.12.024
Cell, 184(3), 655-674.e27. Cell Press
Cell, 184(3), 655-674.e27. CELL PRESS
2021, ' G3BPs tether the TSC complex to lysosomes and suppress mTORC1 signaling ', Cell, vol. 184, no. 3, pp. 655-674.e27 . https://doi.org/10.1016/j.cell.2020.12.024
Cell, 184(3), 655-674.e27. Cell Press
Cell, 184(3), 655-674.e27. CELL PRESS
Summary Ras GTPase-activating protein-binding proteins 1 and 2 (G3BP1 and G3BP2, respectively) are widely recognized as core components of stress granules (SGs). We report that G3BPs reside at the cytoplasmic surface of lysosomes. They act in a non-r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cd1988cff1d6a25e654c51d5e842f355
https://hdl.handle.net/10037/21857
https://hdl.handle.net/10037/21857