Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Tawfik Addi"'
Autor:
Guillaume Lano, Manon Laforêt, Clarissa Von Kotze, Justine Perrin, Tawfik Addi, Philippe Brunet, Stéphane Poitevin, Stéphane Burtey, Laetitia Dou
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 7, p 2392 (2020)
Endogenous agonists of the transcription factor aryl hydrocarbon receptor (AHR) such as the indolic uremic toxin, indoxyl sulfate (IS), accumulate in patients with chronic kidney disease. AHR activation by indolic toxins has prothrombotic effects on
Externí odkaz:
https://doaj.org/article/6569bcafdeb34c26807de67fa9c83420
Publikováno v:
Toxins, Vol 10, Iss 10, p 412 (2018)
Patients with chronic kidney disease (CKD) display an elevated risk of thrombosis. Thrombosis occurs in cardiovascular events, such as venous thromboembolism, stroke, and acute coronary syndrome, and is a cause of hemodialysis vascular access dysfunc
Externí odkaz:
https://doaj.org/article/b03c47e258004c7295f3bc3d5529bb6c
Autor:
Bertrand Gondouin, Françoise Dignat-George, Omar Kheroua, Kamel Eddine El Mecherfi, Nathalie McKay, Tawfik Addi, Stéphane Burtey, Alix de Macedo, Noémie Jourde-Chiche, Claire Cerini, Stéphane Poitevin, Laetitia Dou, Philippe Brunet
Publikováno v:
Archives of Toxicology
Archives of Toxicology, Springer Verlag, 2019, 93 (1), pp.121-136. ⟨10.1007/s00204-018-2328-3⟩
Archives of Toxicology, 2019, 93 (1), pp.121-136. ⟨10.1007/s00204-018-2328-3⟩
Archives of Toxicology, Springer Verlag, 2019, 93 (1), pp.121-136. ⟨10.1007/s00204-018-2328-3⟩
Archives of Toxicology, 2019, 93 (1), pp.121-136. ⟨10.1007/s00204-018-2328-3⟩
International audience; Chronic kidney disease (CKD) is associated with high risk of thrombosis. Indole-3 acetic acid (IAA), an indolic uremic toxin, induces the expression of tissue factor (TF) in human umbilical vein endothelial cells (HUVEC) via t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::354d0643d3192b4d7d624e541547f219
https://hal-amu.archives-ouvertes.fr/hal-02060898
https://hal-amu.archives-ouvertes.fr/hal-02060898
Autor:
Marion Sallée, Tawfik Addi, Stéphane Burtey, Françoise Dignat-George, Claire Cerini, Philippe Brunet, A. Duval-Sabatier, Michael S. Denison, Bertrand Gondouin, Stéphane Poitevin, Laetitia Dou, Nathalie McKay, Noémie Jourde-Chiche
Publikováno v:
Kidney International
Kidney International, 2018, 93 (4), pp.986-999. ⟨10.1016/j.kint.2017.11.010⟩
Kidney International, Nature Publishing Group, 2018, 93 (4), pp.986-999. ⟨10.1016/j.kint.2017.11.010⟩
Kidney International, 2018, 93 (4), pp.986-999. ⟨10.1016/j.kint.2017.11.010⟩
Kidney International, Nature Publishing Group, 2018, 93 (4), pp.986-999. ⟨10.1016/j.kint.2017.11.010⟩
Patients with chronic kidney disease (CKD) are exposed to uremic toxins and have an increased risk of cardiovascular disease. Some uremic toxins, like indoxyl sulfate, are agonists of the transcription factor aryl hydrocarbon receptor (AHR). These to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9a1cf56bfcf5743bf4c9a57a811de44f
https://amu.hal.science/hal-02060310
https://amu.hal.science/hal-02060310
Publikováno v:
Néphrologie & Thérapeutique. 14:404
Introduction La maladie renale chronique (MRC) est associee a une augmentation du risque thrombotique. La comprehension des mecanismes moleculaires de ce risque est indispensable pour identifier de nouveaux traitements. La toxine uremique indole-3 ac
Autor:
Bataille, Stanislas, Chermiti, Rania, Giaime, Philippe, Pedinielli, Nathalie, kay, Nathalie Mc, Lano, Guillaume, Tawfik, Addi, Mancini, Julien, Burtey, Stephane, Dou, Laetitia
Publikováno v:
Journal of the American Society of Nephrology; November 2023, Vol. 34 Issue: 11, Number 11 Supplement 11 p532-532, 1p
Autor:
Marion Sallée, Philippe Brunet, Claire Cerini, Bertrand Gondouin, Tawfik Addi, Noémie Jourde-Chiche, Laetitia Dou, Stéphane Burtey
Publikováno v:
Néphrologie & Thérapeutique. 11:264-265
Introduction Aryl hydrocarbon receptor (AhR) est un facteur de transcription active par certains polluants environnementaux, notamment la dioxine. Cette activation s’accompagne d’une augmentation du risque cardiovasculaire. AhR est active par les