Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Tatyana Vikulina"'
Autor:
Susanne Roser‐Page, Daiana Weiss, Tatyana Vikulina, Mingcan Yu, Roberto Pacifici, M. Neale Weitzmann
Publikováno v:
JBMR Plus, Vol 6, Iss 7, Pp n/a-n/a (2022)
ABSTRACT Cyclic adenosine monophosphate (cAMP)‐dependent phosphodiesterase (PDE) inhibitors such as pentoxifylline (PTX) suppress cAMP degradation and promote cAMP‐dependent signal transduction. PDE inhibitors increase bone formation and bone mas
Externí odkaz:
https://doaj.org/article/3bb095da92144a4a944296c3d9876282
Autor:
Patrice L. Capers, Hyacinth I. Hyacinth, Shayla Cue, Prasanthi Chappa, Tatyana Vikulina, Susanne Roser-Page, M. Neale Weitzmann, David R. Archer, Gale W. Newman, Alexander Quarshie, Jonathan K. Stiles, Jacqueline M. Hibbert
Publikováno v:
Journal of Nutritional Science, Vol 4 (2015)
Key pathophysiology of sickle cell anaemia includes compensatory erythropoiesis, vascular injury and chronic inflammation, which divert amino acids from tissue deposition for growth/weight gain and muscle formation. We hypothesised that sickle mice m
Externí odkaz:
https://doaj.org/article/da0746a8020b4994a82223ed8c970dd7
Autor:
M Neale Weitzmann, Daiana Weiss, Tatyana Vikulina, Susanne Roser-Page, Kanglun Yu, Meghan E McGee-Lawrence, Chia Ling Tu, Wenhan Chang, Ighovwerha Ofotokun
Publikováno v:
J Infect Dis
The Journal of infectious diseases, vol 226, iss 1
The Journal of infectious diseases, vol 226, iss 1
Background Immune reconstitution bone loss (IRBL) is a common side-effect of antiretroviral therapy (ART) in people with human immunodeficiency virus (PWH). Immune reconstitution bone loss acts through CD4+ T-cell/immune reconstitution-induced inflam
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::91037f7edfa3953678395d5af87ee0d1
https://europepmc.org/articles/PMC9373144/
https://europepmc.org/articles/PMC9373144/
Autor:
Li Hao, Tatyana Vikulina, Daiana Weiss, Shannon L. Meeks, Christine L. Kempton, M. Neale Weitzmann, Susanne Roser-Page, Kanglun Yu, Natalia del Mazo Arbona, Meghan E. McGee-Lawrence, W. Hunter Baldwin
Publikováno v:
Blood Advances. 3:288-300
Hemophilia A (HA), a rare X-linked recessive genetic disorder caused by insufficient blood clotting factor VIII, leaves affected individuals susceptible to spontaneous and traumatic hemorrhage. Although males generally exhibit severe symptoms, due to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f3918de0f9ea154c215bdd4a6cce1b28
https://doi.org/10.1182/bloodadvances.2018027557
https://doi.org/10.1182/bloodadvances.2018027557
Autor:
Tatyana Vikulina, M. Neale Weitzmann, Meghan E. McGee-Lawrence, Kanglun Yu, Susanne Roser-Page
Publikováno v:
Rheumatology. 57:1105-1114
Objective Immunosuppressive biologics are used in the management of RA and additional immunomodulators are under investigation including modulators of the CD40/CD40 ligand (CD40L) costimulation pathway. Tampering with immune function can have unantic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a8b3e663c51814302c67762168b6394f
https://doi.org/10.1093/rheumatology/kex525
https://doi.org/10.1093/rheumatology/kex525
Autor:
Roberto Pacifici, Timothy F. Lane, Mark M. Habib, Susanne Roser-Page, Daiana Weiss, M. Neale Weitzmann, George R. Beck, Tatyana Vikulina
Publikováno v:
Annals of the New York Academy of Sciences. 1415:21-33
Activated lymphocytes promote inflammation and bone destruction in rheumatoid arthritis (RA), making T cells and B cells therapeutic targets. Indeed, pharmacological blockade of CD28 costimulation using CTLA-4Ig (abatacept), approved for amelioration
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d95385f48451695cc81e098856419eb7
https://doi.org/10.1111/nyas.13643
https://doi.org/10.1111/nyas.13643
Autor:
Tatyana Vikulina, Susanne Roser-Page, M. Neale Weitzmann, Masayoshi Yamaguchi, Ighovwerha Ofotokun
Publikováno v:
The Journal of infectious diseases. 216(9)
Background Bone loss occurs in human immunodeficiency virus (HIV) infection but paradoxically is intensified by HIV-associated antiretroviral therapy (ART), resulting in an increased fracture incidence that is largely independent of ART regimen. Infl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2dbb91794935fd91f36438901c41aff3
https://pubmed.ncbi.nlm.nih.gov/28968828
https://pubmed.ncbi.nlm.nih.gov/28968828
Publikováno v:
Arthritis & Rheumatology. 66:990-999
RA is a chronic inflammatory autoimmune disease that leads to bone loss around inflamed joints, as well as a generalized systemic osteoporosis (1–3). Lymphocytes play central roles not only in the initiation and progression of the inflammatory stat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8f0b124319fd3919294da5d3c89b7e95
https://doi.org/10.1002/art.38319
https://doi.org/10.1002/art.38319
Publikováno v:
Transfusion Medicine Reviews. 23:13-24
Currently, more than 50 years after its apparent early recognition in case reports, and more than 20 years after its name was coined to denote a distinct entity of pulmonary transfusion reactions, transfusion-related acute lung injury (TRALI) has eme
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2edfcf3babc8e9a224257a22de6ac735
https://doi.org/10.1016/j.tmrv.2008.09.002
https://doi.org/10.1016/j.tmrv.2008.09.002
Autor:
Ighovwerha Ofotokun, Kehmia Titanji, Tatyana Vikulina, Francois Villinger, Susanne Roser-Page, Kenneth A. Rogers, Jeffrey L. Lennox, Cecile D. Lahiri, Aswani Vunnava, Anandi N. Sheth, M. Neale Weitzmann
Publikováno v:
AIDS (London, England). 30(3)
OBJECTIVE Antiretroviral therapy (ART) paradoxically intensifies bone loss in the setting of HIV infection. Although the extent of bone loss varies, it occurs with virtually all ART types, suggesting a common pathway that may be aligned with HIV dise
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5c57258657673850223d800a8486fab1
https://pubmed.ncbi.nlm.nih.gov/26760232
https://pubmed.ncbi.nlm.nih.gov/26760232