Zobrazeno 1 - 10
of 26
pro vyhledávání: '"Tatsuru Togo"'
Autor:
Yoko Fujita, Daisuke Ichikawa, Takeshi Sugaya, Keiichi Ohata, Jun Tanabe, Kazuho Inoue, Seiko Hoshino, Tatsuru Togo, Minoru Watanabe, Kenjiro Kimura, Yugo Shibagaki, Atsuko Kamijo-Ikemori
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-13 (2021)
Abstract We investigate whether suppressing the activation of the angiotensin II type 1a receptor (AT1a) can ameliorate severe chronic tubulointerstitial damage (TID) after renal ischemia reperfusion (IR) using AT1a knockout homozygous (AT1a−/−)
Externí odkaz:
https://doaj.org/article/18eb73f97dd64954b6068a75ce099b1d
Autor:
Tatsuru Togo
Publikováno v:
Biology Open, Vol 6, Iss 12, Pp 1814-1819 (2017)
Disruption of cellular plasma membranes is a common event in many animal tissues, and the membranes are usually rapidly resealed. Moreover, repeated membrane disruptions within a single cell reseal faster than the initial wound in a protein kinase A
Externí odkaz:
https://doaj.org/article/1d39417d5ef240aaa64ebeaf38f7916b
Autor:
Tatsuru Togo
Publikováno v:
PLoS ONE, Vol 7, Iss 8, p e42885 (2012)
Resealing of a disrupted plasma membrane at the micron-diameter range requires Ca(2+)-regulated exocytosis. Repeated membrane disruptions reseal more quickly than the initial wound, and this potentiation of membrane resealing persists for at least 24
Externí odkaz:
https://doaj.org/article/66c6a00aa91f4aa6a688f3bf18d6472e
Autor:
Takeshi Sugaya, Yugo Shibagaki, Seiko Hoshino, Kazuho Inoue, Atsuko Kamijo-Ikemori, Yoko Fujita, Daisuke Ichikawa, Minoru Watanabe, Kenjiro Kimura, Tatsuru Togo, Jun Tanabe, Keiichi Ohata
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-13 (2021)
Scientific Reports
Scientific Reports
We investigate whether suppressing the activation of the angiotensin II type 1a receptor (AT1a) can ameliorate severe chronic tubulointerstitial damage (TID) after renal ischemia reperfusion (IR) using AT1a knockout homozygous (AT1a−/−) male mice
Autor:
Tatsuru, Togo
Publikováno v:
Current topics in membranes. 84
Plasma membrane disruption occurs frequently in many animal tissues. Cell membrane disruption induces not only a rapid and massive influx of Ca
Autor:
Tatsuru Togo
Publikováno v:
Biochemical and biophysical research communications. 511(1)
Disruption and repair of plasma membranes is normally observed in many animal tissues. Recent studies demonstrated that wounding of Madin-Darby canine kidney cells potentiates membrane repair in cells adjacent to wounded cells via paracrine purinergi
Autor:
Tatsuru Togo
Plasma membrane disruption occurs frequently in many animal tissues. Cell membrane disruption induces not only a rapid and massive influx of Ca2+ into the cytosol but also an efflux or release of various signaling molecules, such as ATP, from the cyt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6967af2d16ec4b586d96537d8ae041ff
https://doi.org/10.1016/bs.ctm.2019.07.003
https://doi.org/10.1016/bs.ctm.2019.07.003
Autor:
Luigia Santella, Toko Tsurugaya, Gabriele Amore, Koji Akasaka, Shinichiro Kusunoki, Ikuko Yazaki, Akiko Asada, Jong Tai Chun, Tatsuru Togo
Publikováno v:
Zygote (Cambridge, England)
SummarySea urchin embryos initiate cell specifications at the 16-cell stage by forming the mesomeres, macromeres and micromeres according to the relative position of the cells in the animal–vegetal axis. The most vegetal cells, micromeres, autonomo
Autor:
Tatsuru Togo
Publikováno v:
Purinergic Signalling. 10:283-290
Resealing of a disrupted plasma membrane in the micron-size range requires Ca(2+)-regulated exocytosis. When cells are wounded twice, the second membrane disruption reseals more quickly than the initial wound. This response is protein kinase C (PKC)-
Autor:
Tatsuru Togo
Publikováno v:
Biochemical and Biophysical Research Communications. 388:12-16
Microtubule (MT) plus-end tracking proteins (+TIPs) are involved in the regulation of MT plus-end dynamics and stabilization. It was reported previously that an increase in intracellular Ca(2+) concentration ([Ca(2+)](i)) induced by disruption of the