Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Tatsuro Zeze"'
Autor:
Mio Imagawa, Takanori Shinjo, Kohei Sato, Kentaro Kawakami, Tatsuro Zeze, Yuki Nishimura, Masaaki Toyoda, Shuang Chen, Naoaki Ryo, Al-kafee Ahmed, Misaki Iwashita, Akiko Yamashita, Takao Fukuda, Terukazu Sanui, Fusanori Nishimura
Publikováno v:
Frontiers in Physiology, Vol 14 (2023)
Drug-induced gingival overgrowth (DIGO), induced by certain immunosuppressive drugs, antihypertensive agents, and antiepileptic drugs, may contribute to the formation of deeper periodontal pockets and intractableness in periodontitis. To date, multip
Externí odkaz:
https://doaj.org/article/9d1c5e5e5f1f4fda8fbe1420134ce008
Autor:
Yuki Nishimura, Misaki Iwashita, Masato Hayashi, Takanori Shinjo, Yukari Watanabe, Tatsuro Zeze, Akiko Yamashita, Takao Fukuda, Terukazu Sanui, Tomomi Sano, Tomoichiro Asano, Fusanori Nishimura
Publikováno v:
Acta Diabetologica. 59:1275-1286
Aims Pancreatic β-cell apoptosis may be involved in the onset and progression of type 2 diabetes mellitus, although its mechanism remains unclear. We previously demonstrated that macrophage-derived interferon (IFN) β induced X-linked inhibitor of a
Autor:
George L King, Fusanori Nishimura, Thomas E Van Dyke, Hatice Hasturk, Koji Mizutani, Wu I-Hsien, Jialin Fu, Ronald St-Louis, Kohei Sato, Tatsuro Zeze, Hisashi Yokomizo, Qian Li, Kyoungmin Park, Atsushi Ishikado, Yumi Zaitsu, Satoru Onizuka, Takanori Shinjo
Publikováno v:
Diabetes.
Insulin resistance and hyperglycemia are risk factors for periodontitis and poor wound healing in diabetes, which have been associated with selective loss of insulin- activation of the PI3K/Akt pathway in the gingiva. This study showed that insulin r
Autor:
Fusanori Nishimura, Akiko Yamashita, Terukazu Sanui, Masato Hayashi, Takanori Shinjo, Tatsuro Zeze, Rehab Alshargabi, Yuki Nishimura, Takao Fukuda, Misaki Iwashita, Tomomi Sano
Publikováno v:
Biochemical and biophysical research communications. 533(4)
SPOCK1 is a calcium-binding matricellular proteoglycan that has been extensively studied in several cancer cells. Previously, we generated a mouse line overexpressing SPOCK1 (Spock1-Tg mouse) and showed that SPOCK1 might play an important role in dru