Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Tatjana Zake"'
Autor:
Sabine Upmale-Engela, Ieva Vaivode, Raitis Peculis, Helena Litvina, Tatjana Zake, Andrejs Skesters, Deniss Gogins, Vita Rovite, Ilze Konrade
Publikováno v:
Current Issues in Molecular Biology, Vol 46, Iss 3, Pp 2553-2565 (2024)
The interplay of genetic, immune and environmental factors strongly contributes to the development of autoimmune thyroid disease (AITD), which can be classified as Graves’ disease (GD) or Hashimoto thyroiditis (HT). One of the most studied exogenou
Externí odkaz:
https://doaj.org/article/5bd8a024e63640179f73cd69d81b28be
Autor:
Tatjana Zake, Ieva Kalere, Sabine Upmale‐Engela, Simons Svirskis, Gita Gersone, Andrejs Skesters, Valerija Groma, Ilze Konrade
Publikováno v:
Immunity, Inflammation and Disease, Vol 9, Iss 3, Pp 792-803 (2021)
Abstract Introduction The contribution of Th17 cytokines to autoimmune thyroid disease (AITD) is generally accepted. However, the roles of Th17 cells in the initiation and progression of Hashimoto's thyroiditis (HT) and Graves' disease (GD) remain un
Externí odkaz:
https://doaj.org/article/04e689f0324446f2aa313f450191cbb4
Autor:
Ieva Vaivode, Tatjana Zake, Ieva Strele, Sabine Upmale-Engela, Deniss Gogins, Gita Gersone, Andrejs Skesters, Maija Dambrova, Ilze Konrade
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 3, p 2440 (2023)
Autoimmune thyroid disease (AITD), including Graves’ disease (GD) or Hashimoto’s thyroiditis (HT), occurs due to genetic susceptibility and environmental factors, among which the role of stressful events remains controversial. This study investig
Externí odkaz:
https://doaj.org/article/56283aee54f348a38a5454ba51ef93e4
Autor:
Vija Veisa, Ieva Kalere, Tatjana Zake, Ieva Strele, Marina Makrecka-Kuka, Sabine Upmale-Engela, Andrejs Skesters, Dace Rezeberga, Aivars Lejnieks, Iveta Pudule, Daiga Grinberga, Biruta Velika, Maija Dambrova, Ilze Konrade
Publikováno v:
Medicina, Vol 57, Iss 11, p 1211 (2021)
Background and Objectives: Adequate dietary intake of iodine and selenium is essential during pregnancy. While iodine is vital for maternal thyroid function and fetal development, selenium contributes to the regulation of thyroid function and thyroid
Externí odkaz:
https://doaj.org/article/a2b51c3528c84689853c99b193da5793
Autor:
Sabine Upmale-Engela, Gita Gersone, Valerija Groma, Ilze Konrade, Ieva Kalere, Andrejs Skesters, Simons Svirskis, Tatjana Zake
Publikováno v:
Immunity, Inflammation and Disease, Vol 9, Iss 3, Pp 792-803 (2021)
Immunity, Inflammation and Disease
Immunity, Inflammation and Disease
Introduction The contribution of Th17 cytokines to autoimmune thyroid disease (AITD) is generally accepted. However, the roles of Th17 cells in the initiation and progression of Hashimoto's thyroiditis (HT) and Graves' disease (GD) remain unclear. Se
Autor:
Tatjana Zake, Daiga Grinberga, Vija Veisa, Ieva Strele, Ilze Konrade, Ieva Kalere, Aivars Lejnieks, Sabine Upmale-Engela, Biruta Velika, Iveta Pudule, Dace Rezeberga, Maija Dambrova, Marina Makrecka-Kuka, Andrejs Skesters
Publikováno v:
Medicina
Medicina, Vol 57, Iss 1211, p 1211 (2021)
Volume 57
Issue 11
Medicina, Vol 57, Iss 1211, p 1211 (2021)
Volume 57
Issue 11
Background and Objectives: Adequate dietary intake of iodine and selenium is essential during pregnancy. While iodine is vital for maternal thyroid function and fetal development, selenium contributes to the regulation of thyroid function and thyroid
Publikováno v:
Endocrine Journal. 66:423-430
T helper (Th) 17 cells and interleukin (IL)-17 play a significant role in the pathogenesis of autoimmune thyroid disease (AITD). However, it has recently become clear that Th17 cells are more heterogeneous and exhibit two different phenotypes, wherea
Publikováno v:
Endocrine Abstracts.
Publikováno v:
Medicine
Th17 cells together with their hallmark cytokine interleukin (IL)-17 were identified as crucial contributing factors in the pathogenesis of thyroid autoimmunity. The cytokine-regulated tight junction (Tj) disruption is thought to be essential in the
Publikováno v:
Endocrine Abstracts.