Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Taslima T Lina"'
Publikováno v:
PLoS ONE, Vol 15, Iss 10, p e0240789 (2020)
In a recent study, using an in vitro model to study intravaginal nanoparticle exposure during yeast infections, we demonstrated that C. albicans exposure suppressed apoptotic gene expression and induced oxidative stress and pyroptosis in vaginal epit
Externí odkaz:
https://doaj.org/article/29123554345e43d691103109e8cc3a1b
Autor:
Taslima T Lina, Shatha Alzahrani, Jennifer House, Yoshio Yamaoka, Arlene H Sharpe, Bill A Rampy, Irina V Pinchuk, Victor E Reyes
Publikováno v:
PLoS ONE, Vol 10, Iss 3, p e0121841 (2015)
During Helicobacter pylori (H. pylori) infection CD4+ T cells in the gastric lamina propria are hyporesponsive and polarized by Th1/Th17 cell responses controlled by Treg cells. We have previously shown that H. pylori upregulates B7-H1 expression on
Externí odkaz:
https://doaj.org/article/a598c471666d4b8697cfbf0b6d89ca0b
Autor:
Taslima T Lina, Bijay K Khajanchi, Ishrat J Azmi, Mohammad Aminul Islam, Belal Mahmood, Mahmuda Akter, Atanu Banik, Rumana Alim, Armando Navarro, Gabriel Perez, Alejandro Cravioto, Kaisar A Talukder
Publikováno v:
PLoS ONE, Vol 9, Iss 10, p e108735 (2014)
BackgroundResistance to cephalosporins in Enterobacteriaceae is mainly due to the production of extended-spectrum beta-lactamase (ESBL). Little is known about ESBL-producing bacteria in Bangladesh. Therefore, the study presents results of phenotypic
Externí odkaz:
https://doaj.org/article/abbf45f7ac1b47f3bba8aff56e480cc2
Helicobacter pylori (H. pylori) is a gram-negative bacterium that infects more than 50% of humanity and is associated with gastritis, peptic ulcer and gastric cancer. Although CD4+ T cells are recruited to the gastric mucosa, the host is unable to cl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7233e4d0ab763fc103027d272be0f27e
https://europepmc.org/articles/PMC6988449/
https://europepmc.org/articles/PMC6988449/
Publikováno v:
PLoS ONE
PLoS ONE, Vol 15, Iss 10, p e0240789 (2020)
PLoS ONE, Vol 15, Iss 10, p e0240789 (2020)
In a recent study, using an in vitro model to study intravaginal nanoparticle exposure during yeast infections, we demonstrated that C. albicans exposure suppressed apoptotic gene expression and induced oxidative stress and pyroptosis in vaginal epit
Publikováno v:
Infection and immunity. 85(12)
In multicellular organisms, autophagy is induced as an innate defense mechanism. Notably, the obligate intracellular bacterium Ehrlichia chaffeensis resides in early endosome-like vacuoles and circumvents lysosomal fusion through an unknown mechanism
Autor:
Taslima T. Lina, Irina V. Pinchuk, Yoshio Yamaoka, Ellen J. Beswick, Jennifer House, David Y. Graham, Victor E. Reyes
Publikováno v:
The Journal of Immunology. 191:3838-3846
Gastric epithelial cells (GECs) are the primary target for Helicobacter pylori infection and may act as APCs regulating local T cell responses. We previously reported that H. pylori infection of GECs induces the expression of the T cell coinhibitory
Publikováno v:
mBio, Vol 7, Iss 4, p e00672-16 (2016)
mBio, Vol 7, Iss 4 (2016)
mBio
mBio, Vol 7, Iss 4 (2016)
mBio
Ehrlichia chaffeensis preferentially targets mononuclear phagocytes and survives through a strategy of subverting innate immune defenses, but the mechanisms are unknown. We have shown E. chaffeensis type 1 secreted tandem repeat protein (TRP) effecto
Publikováno v:
Frontiers in Cellular and Infection Microbiology, Vol 6 (2016)
Frontiers in Cellular and Infection Microbiology
Frontiers in Cellular and Infection Microbiology
Ehrlichia chaffeensis is a small, gram negative, obligately intracellular bacterium that preferentially infects mononuclear phagocytes. It is the etiologic agent of human monocytotropic ehrlichiosis (HME), an emerging life-threatening tick-borne zoon
Ehrlichia chaffeensis invades and survives in phagocytes by modulating host cell processes and evading innate defenses, but the mechanisms are not fully defined. Recently we have determined that E. chaffeensis tandem repeat proteins (TRPs) are type 1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6fe46e6bf89fbddfbe8f2e6bfde9cb98
https://europepmc.org/articles/PMC4771358/
https://europepmc.org/articles/PMC4771358/