Zobrazeno 1 - 10 of 10 pro vyhledávání: '"Taryn A. Cass"'
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Akademický článek
The metabolic bone disease associated with the Hyp mutation is independent of osteoblastic HIF1α expression
Autor: Julia M. Hum, Erica L. Clinkenbeard, Colin Ip, Taryn A. Cass, Matt Allen, Kenneth E. White
Publikováno v: Bone Reports, Vol 6, Iss C, Pp 38-43 (2017)
Fibroblast growth factor-23 (FGF23) controls key responses to systemic phosphate increases through its phosphaturic actions on the kidney. In addition to stimulation by phosphate, FGF23 positively responds to iron deficiency anemia and hypoxia in rod
Externí odkaz: https://doaj.org/article/5ae83a4a382f4e56a5a7ca85add00fd1
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The metabolic bone disease associated with the Hyp mutation is independent of osteoblastic HIF1α expression
Autor: Matthew R. Allen, Taryn A. Cass, Kenneth E. White, Erica L. Clinkenbeard, Colin S. Ip, Julia M. Hum
Publikováno v: Bone Reports, Vol 6, Iss C, Pp 38-43 (2017)
Fibroblast growth factor-23 (FGF23) controls key responses to systemic phosphate increases through its phosphaturic actions on the kidney. In addition to stimulation by phosphate, FGF23 positively responds to iron deficiency anemia and hypoxia in rod
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2676c62b3a3741003cabc96dc7cbe3d3
http://www.sciencedirect.com/science/article/pii/S2352187217300037
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4
Conditional Deletion of MurineFgf23: Interruption of the Normal Skeletal Responses to Phosphate Challenge and Rescue of Genetic Hypophosphatemia
Autor: Teresita Bellido, Erica L. Clinkenbeard, Taryn A. Cass, Matthew R. Allen, Kenneth E. White, Julia M. Hum, Pu Ni
Publikováno v: Journal of Bone and Mineral Research. 31:1247-1257
The transgenic and knockout (KO) animals involving Fgf23 have been highly informative in defining novel aspects of mineral metabolism, but are limited by shortened lifespan, inability of spatial/temporal FGF23 control, and infertility of the global K
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::c30678eed9ff75e69d87153c9ef06346
https://doi.org/10.1002/jbmr.2792
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6
Neonatal Iron Deficiency Causes Abnormal Phosphate Metabolism by Elevating FGF23 in Normal and ADHR Mice
Autor: Kenneth E. White, Tim Lahm, Lelia J. Summers, Christine A Bayt, Marjorie Albrecht, Emily G. Farrow, Erica L. Clinkenbeard, Taryn A. Cass, Munro Peacock, Jessica L. Roberts, Matthew R. Allen
Publikováno v: Journal of Bone and Mineral Research. 29:361-369
Fibroblast growth factor 23 (FGF23) gain of function mutations can lead to autosomal dominant hypophosphatemic rickets (ADHR) disease onset at birth, or delayed onset following puberty or pregnancy. We previously demonstrated that the combination of
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::bc5b278d1218918e8ce65a2a15ac686f
https://doi.org/10.1002/jbmr.2049
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7
The metabolic bone disease associated with the
Autor: Julia M, Hum, Erica L, Clinkenbeard, Colin, Ip, Taryn A, Cass, Matt, Allen, Kenneth E, White
Publikováno v: Bone Reports
Fibroblast growth factor-23 (FGF23) controls key responses to systemic phosphate increases through its phosphaturic actions on the kidney. In addition to stimulation by phosphate, FGF23 positively responds to iron deficiency anemia and hypoxia in rod
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::1161dd0fadc69ca82f90317ea5726aee
https://pubmed.ncbi.nlm.nih.gov/28377980
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8
Chronic Hyperphosphatemia and Vascular Calcification Are Reduced by Stable Delivery of Soluble Klotho
Autor: Arun K. Tatiparthi, Robert L. Johnson, Erica L. Clinkenbeard, Martin S. Cramer, Manoj Bhaskaran, Linda Maureen O'bryan, Rosamund Carol Smith, Kenneth E. White, Jonathan M. Wilson, Taryn A. Cass, Julia M. Hum
Publikováno v: Journal of the American Society of Nephrology : JASN. 28(4)
αKlotho (αKL) regulates mineral metabolism, and diseases associated with αKL deficiency are characterized by hyperphosphatemia and vascular calcification (VC). αKL is expressed as a membrane-bound protein (mKL) and recognized as the coreceptor fo
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6d2efb5613c3426da0aac844c074e86c
https://pubmed.ncbi.nlm.nih.gov/27837149
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9
Neonatal iron deficiency causes abnormal phosphate metabolism by elevating FGF23 in normal and ADHR mice
Autor: Erica L, Clinkenbeard, Emily G, Farrow, Lelia J, Summers, Taryn A, Cass, Jessica L, Roberts, Christine A, Bayt, Tim, Lahm, Marjorie, Albrecht, Matthew R, Allen, Munro, Peacock, Kenneth E, White
Publikováno v: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. 29(2)
Fibroblast growth factor 23 (FGF23) gain of function mutations can lead to autosomal dominant hypophosphatemic rickets (ADHR) disease onset at birth, or delayed onset following puberty or pregnancy. We previously demonstrated that the combination of
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::efa719e76c0ba51c8da3e53abbb84af4
https://pubmed.ncbi.nlm.nih.gov/23873717
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