Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Tanya A. Ramsamy"'
Autor:
Jonathan Boucher, Tanya A. Ramsamy, Sylvie Braschi, Daisy Sahoo, Tracey A-M. Neville, Daniel L. Sparks
Publikováno v:
Journal of Lipid Research, Vol 45, Iss 5, Pp 849-858 (2004)
The effect of apolipoprotein A-II (apoA-II) on the structure and stability of HDL has been investigated in reconstituted HDL particles. Purified human apoA-II was incorporated into sonicated, spherical LpA-I particles containing apoA-I, phospholipids
Externí odkaz:
https://doaj.org/article/a200dfe6534b4cd68718feabde749930
Autor:
Robert J. Brown, Joshua R. Schultz, Kerry W.S. Ko, John S. Hill, Tanya A. Ramsamy, Ann L. White, Daniel L. Sparks, Zemin Yao
Publikováno v:
Journal of Lipid Research, Vol 44, Iss 7, Pp 1306-1314 (2003)
Human hepatic lipase (hHL) mainly exists cell surface bound, whereas mouse HL (mHL) circulates in the blood stream. Studies have suggested that the carboxyl terminus of HL mediates cell surface binding. We prepared recombinant hHL, mHL, and chimeric
Externí odkaz:
https://doaj.org/article/98a41152971649969a457b20a49caa8c
Publikováno v:
Journal of Lipid Research, Vol 44, Iss 4, Pp 733-741 (2003)
We have previously shown that hepatic lipase (HL) is inactive when bound to purified heparan sulfate proteoglycans and can be liberated by HDL and apolipoprotein A-I (apoA-I), but not by LDL or VLDL. In this study, we show that HDL is also able to di
Externí odkaz:
https://doaj.org/article/8be3367ecff4475ca4998d9b005edb07
Autor:
Tanya A. Ramsamy, Daniel L. Sparks, Daisy Sahoo, Tracey A-M. Neville, Sylvie Braschi, Jonathan G. Boucher
Publikováno v:
Journal of Lipid Research, Vol 45, Iss 5, Pp 849-858 (2004)
The effect of apolipoprotein A-II (apoA-II) on the structure and stability of HDL has been investigated in reconstituted HDL particles. Purified human apoA-II was incorporated into sonicated, spherical LpA-I particles containing apoA-I, phospholipids
Autor:
Zemin Yao, Robert J. Brown, Tanya A. Ramsamy, Ann L. White, John S. Hill, Daniel L. Sparks, Kerry W.S. Ko, Joshua R. Schultz
Publikováno v:
Journal of Lipid Research, Vol 44, Iss 7, Pp 1306-1314 (2003)
Human hepatic lipase (hHL) mainly exists cell surface bound, whereas mouse HL (mHL) circulates in the blood stream. Studies have suggested that the carboxyl terminus of HL mediates cell surface binding. We prepared recombinant hHL, mHL, and chimeric
Publikováno v:
Journal of Lipid Research, Vol 44, Iss 4, Pp 733-741 (2003)
We have previously shown that hepatic lipase (HL) is inactive when bound to purified heparan sulfate proteoglycans and can be liberated by HDL and apolipoprotein A-I (apoA-I), but not by LDL or VLDL. In this study, we show that HDL is also able to di
Autor:
Daniel L. Sparks, Tanya A. Ramsamy, Sylvie Braschi, Richard Seymour, Tracey A.-M. Neville, Cyrille Maugeais
Publikováno v:
Biochemistry. 39:5441-5449
To evaluate the factors that regulate HDL catabolism in vivo, we have measured the clearance of human apoA-I from rabbit plasma by following the isotopic decay of (125)I-apoA-I and the clearance of unlabeled apoA-I using a radioimmunometric assay (RI
Publikováno v:
Biochemistry. 37:3735-3742
The structural organization and stability of apoB100 in complexes containing triglyceride (TG) and phospholipid have been examined. LDL was delipidated to form aqueous soluble apoB100-TG complexes that retain approximately 70% of LDL TG, but contain
Publikováno v:
Journal of Lipid Research, Vol 38, Iss 11, Pp 2224-2231 (1997)
The hydrolysis of HDL phospholipids (PL) and glycerides by hepatic lipase (HL) has been investigated in native and reconstituted HDL particles (Lp2A-I). Fasting, normolipidemic HDL exhibit total lipid hydrolytic rates of between 10 and 36 nM FA/h per
Autor:
Leah, Rogers, Sarah, Burchat, Jessica, Gage, Mirela, Hasu, Mohamad, Thabet, Lindsay, Willcox, Lindsay, Wilcox, Tanya A, Ramsamy, Stewart C, Whitman
Publikováno v:
Cardiovascular Research
CD1d-restricted natural killer T (NKT) cells function by regulating numerous immune responses during innate and adaptive immunity. Depletion of all populations of CD1d-dependent NKT cells has been shown by several groups to reduce atherosclerosis in