Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Tanja Schreiber"'
Autor:
Skadi Kull, K Melanie Schulz, Jasmin Weisemann, Sebastian Kirchner, Tanja Schreiber, Alexander Bollenbach, P Wojtek Dabrowski, Andreas Nitsche, Suzanne R Kalb, Martin B Dorner, John R Barr, Andreas Rummel, Brigitte G Dorner
Publikováno v:
PLoS ONE, Vol 10, Iss 2, p e0116381 (2015)
Botulism is a severe neurological disease caused by the complex family of botulinum neurotoxins (BoNT). Based on the different serotypes known today, a classification of serotype variants termed subtypes has been proposed according to sequence divers
Externí odkaz:
https://doaj.org/article/cfd6ec23d336421fb0b2a67143b2e8a2
Autor:
Tanja Schreiber
Tod und Trauer am Arbeitsplatz treten oft plötzlich und unvorhersehbar auf; besonders gravierend ist dies dann, wenn Kollegen sterben. Der Tod durchbricht den Führungsalltag und sorgt für Krisenzustände und Chaos. Die tiefgreifende Veränderung d
Autor:
Christoph Hölscher, Hanna Erdmann, Mahin Abad Dar, Lisa Heitmann, Stefan Ehlers, Andrew N. J. McKenzie, Frank Brombacher, Jochen Behrends, Tanja Schreiber
Publikováno v:
The Journal of Pathology
Human tuberculosis (TB) is a leading global health threat and still constitutes a major medical challenge. However, mechanisms governing tissue pathology during post-primary TB remain elusive, partly because genetically or immunologically tractable a
Autor:
Martin B. Dorner, Marc-André Avondet, Conny Mascher, Oliver G. Weingart, Charlotte Egger, Tanja Schreiber, Thomas F. H. Berger, Martin J. Loessner, Diana Pauly, Frank Gessler, Brigitte G. Dorner
Publikováno v:
Applied and Environmental Microbiology. 76:3293-3300
Botulinum neurotoxin (BoNT) is the most toxic substance known to man and the causative agent of botulism. Due to its high toxicity and the availability of the producing organism Clostridium botulinum , BoNT is regarded as a potential biological warfa
Autor:
Stefan Ehlers, Alexandra Rausch, Peter J. Murray, Jörg Mages, Tanja Schreiber, Christoph Hölscher, Roland Lang, Lisa Heitmann
Publikováno v:
The Journal of Immunology. 183:1301-1312
Elevated IL-10 has been implicated in reactivation tuberculosis (TB). Since macrophages rather than T cells were reported to be the major source of IL-10 in TB, we analyzed the consequences of a macrophage-specific overexpression of IL-10 in transgen
Autor:
Manuela Heßmann, Alexandra Rausch, Christoph Hölscher, Silvia Bulfone-Paus, Alexandra Hölscher, Stefan Ehlers, Tanja Schreiber
Publikováno v:
European Journal of Immunology. 36:1156-1167
CD8+ T cells are involved in protection against Mycobacterium tuberculosis infection and represent a promising target for new vaccine strategies. Because IL-15 is important for the homeostasis of CD8+ T cells, we studied the immune response in IL-15-
Autor:
Sahar Aly, Christoph Hölscher, Stefan Ehlers, Sven Hartmann, Alexandra Hölscher, Tanja Schreiber, Martin Lipp, John B. Lowe
Publikováno v:
The Journal of Immunology. 176:1131-1140
Immunity to Mycobacterium tuberculosis infection is critically dependent on the timely priming of T effector lymphocytes and their efficient recruitment to the site of mycobacterial implantation in the lung. E-, P-, and L-selectin counterreceptors co
Autor:
Andreas Rummel, Skadi Kull, Sebastian Kirchner, J. Strotmeier, Martin B. Dorner, Alexander Bollenbach, P. Wojtek Dabrowski, Tanja Schreiber, Andreas Nitsche, K. Melanie Schulz, Brigitte G. Dorner, Suzanne R. Kalb, John R. Barr
Publikováno v:
PLoS ONE
PLoS ONE, Vol 10, Iss 2, p e0116381 (2015)
PLoS ONE, Vol 10, Iss 2, p e0116381 (2015)
Botulism is a severe neurological disease caused by the complex family of botulinum neurotoxins (BoNT). Based on the different serotypes known today, a classification of serotype variants termed subtypes has been proposed according to sequence divers
Publikováno v:
Immunobiology. 214(8)
In order to determine the relative importance of fucosyltransferase (Fuc-T)-directed functionalization of selectin ligands for chronic inflammatory and antibacterially protective responses in vivo, mice selectively deficient for Fuc-TIV, Fuc-TVII or