Zobrazeno 1 - 10
of 49
pro vyhledávání: '"Tamotsu Nishida"'
Autor:
Tamotsu Nishida
Publikováno v:
Biochemical and Biophysical Research Communications. 615:150-156
The transcriptional repressor PARIS, a substrate of the ubiquitin E3 ligase parkin, represses the expression of the transcriptional co-activator, PGC-1α gene, and is involved in several pathological processes, including neurodegenerative disease and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::57531364c21e859eefb11dfd5db4fcf7
https://doi.org/10.1016/j.bbrc.2022.05.065
https://doi.org/10.1016/j.bbrc.2022.05.065
RNF4-mediated SUMO-targeted ubiquitination relieves PARIS/ZNF746-mediated transcriptional repression
Autor:
Tamotsu Nishida, Yoshiji Yamada
Publikováno v:
Biochemical and Biophysical Research Communications. 526:110-116
The transcriptional repressor PARIS, which is a substrate of the ubiquitin E3 ligase parkin, represses the expression of the transcriptional co-activator, PGC-1α. However, little is known about how its repression activity is regulated. We have previ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::61f3985db3dc065de83dc188685781c9
https://doi.org/10.1016/j.bbrc.2020.03.063
https://doi.org/10.1016/j.bbrc.2020.03.063
Autor:
Tamotsu Nishida, Yoshiji Yamada
Publikováno v:
Biochemical and Biophysical Research Communications. 473:1261-1267
Parkin-interacting substrate (PARIS), a member of the family of Krüppel-associated box (KRAB)-containing zinc-finger transcription factors, is a substrate of the ubiquitin E3 ligase parkin. PARIS represses the expression of peroxisome proliferator-a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::532195b48940c58963bc286c01edb6b9
https://doi.org/10.1016/j.bbrc.2016.04.051
https://doi.org/10.1016/j.bbrc.2016.04.051
Autor:
Mitsutoshi Oguri, Hideki Horibe, Yoshiji Yamada, Motoji Sawabe, Kimihiko Kato, Tamotsu Nishida
Publikováno v:
International Journal of Molecular Medicine. 33:1355-1363
Epigenetic modification, particularly changes in DNA methylation at gene promoters, is implicated in the pathogenesis of atherosclerosis. However, the analysis of DNA methylation in atherosclerosis has been limited to a few selected candidate genes.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2c4ade0f77327beef96632a9a1ab8ff5
https://doi.org/10.3892/ijmm.2014.1692
https://doi.org/10.3892/ijmm.2014.1692
Publikováno v:
Genes to Cells. 16:652-669
The small ubiquitin-related modifier (SUMO) system of higher eukaryotes plays important roles in normal cell division, especially in chromosome segregation. However, only a few mitotic SUMO substrates have been identified in mammals. Here, we show th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::47f50d92df2d2eaff38b120430352750
https://doi.org/10.1111/j.1365-2443.2011.01521.x
https://doi.org/10.1111/j.1365-2443.2011.01521.x
Autor:
Sachiko Tsukita, Kousei Ito, Hiroshi Suzuki, Tamotsu Nishida, Yuki Ikebuchi, Satoko Minami, Shuichi Sekine, Yoshikatsu Kanai, Masashi Honma, Naohiko Anzai, Toshiharu Horie
Publikováno v:
American Journal of Physiology-Gastrointestinal and Liver Physiology. 296:G406-G413
The ATP-binding cassette transporter family C 2 (Abcc2) is a member of efflux transporters involved in the biliary excretion of organic anions from hepatocytes. Posttranslational regulation of Abcc2 has been implicated, although the molecular mechani
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::71b3decfd47aa465f26c3bed11c08641
https://doi.org/10.1152/ajpgi.90309.2008
https://doi.org/10.1152/ajpgi.90309.2008
Autor:
Norie Nakahara, Tamotsu Nishida, Ayako Muraoka, Atsuyo Maeda, Tokumi Maruyama, Midori Yokota, Takayuki Ohshima
Publikováno v:
Biochemical and Biophysical Research Communications. 377:1031-1035
It is emerging that covalent modifications of many transcription factors and co-factors by the small ubiquitin-like modifier (SUMO) can have a key role in modulating their transcriptional regulation. As SUMO modification is often associated with tran
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9a7d6b1e41912512370fbaafbbef6c49
https://doi.org/10.1016/j.bbrc.2008.09.149
https://doi.org/10.1016/j.bbrc.2008.09.149
Autor:
Yoshiji Yamada, Tamotsu Nishida
Publikováno v:
Biochemical and Biophysical Research Communications. 374:382-387
Sumoylation is reversibly regulated by SUMO-specific proteases. We characterized a nucleolar SUMO-specific protease, SMT3IP1, which has a preference for SUMO-2/3. To elucidate SMT3IP1 function, we screened for its interacting proteins that may be its
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6a94e200f7ee3aac71b1241afe357a0b
https://doi.org/10.1016/j.bbrc.2008.07.047
https://doi.org/10.1016/j.bbrc.2008.07.047
Publikováno v:
Taiikugaku kenkyu (Japan Journal of Physical Education, Health and Sport Sciences). 53:147-158
The purpose of this study was to reconstruct a cognitive appraisal scale for high school athletes (Shibukura and Mori, 2004), and to examine the scale's reliability and validity. The subjects were 1370 first and second year high school athletes (903
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3b4fde5074628aa158a44e794d970946
https://doi.org/10.5432/jjpehss.a530111
https://doi.org/10.5432/jjpehss.a530111
Publikováno v:
eLS
Myocardial infarction (MI) is an important clinical problem because of its large contribution to mortality. The main causal and treatable risk factors for MI include hypertension, hypercholesterolemia or dyslipidemia, diabetes mellitus, and smoking.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bbe3abd6b9072f98b3325ac0e8d4706e
https://doi.org/10.1007/s11568-008-9025-x
https://doi.org/10.1007/s11568-008-9025-x