Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Tamara A Kashiwada"'
Autor:
Pavel Krejci, Lisa Salazar, Tamara A Kashiwada, Katarina Chlebova, Alena Salasova, Leslie Michels Thompson, Vitezslav Bryja, Alois Kozubik, William R Wilcox
Publikováno v:
PLoS ONE, Vol 3, Iss 12, p e3961 (2008)
Activating mutations in FGFR3 tyrosine kinase cause several forms of human skeletal dysplasia. Although the mechanisms of FGFR3 action in cartilage are not completely understood, it is believed that the STAT1 transcription factor plays a central role
Externí odkaz:
https://doaj.org/article/05d9a687bded42dbbc7fcef01db7e7b0
Autor:
Malcolm Casale, Lisa Salazar, April N. Meyer, Daniel J. Donoghue, William R. Wilcox, Matthew Hallowell, Tamara A. Kashiwada, Leslie M. Thompson, Pavel Krejci
Publikováno v:
PLoS ONE, Vol 9, Iss 1, p e86470 (2014)
Salazar, L; Kashiwada, T; Krejci, P; Meyer, AN; Casale, M; Hallowell, M; et al.(2014). Fibroblast growth factor receptor 3 interacts with and activates TGFβ-activated kinase 1 tyrosine phosphorylation and NFκB signaling in multiple myeloma and bladder cancer. PLoS ONE, 9(1). doi: 10.1371/journal.pone.0086470. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/09n6212c
PLoS ONE
Salazar, L; Kashiwada, T; Krejci, P; Meyer, AN; Casale, M; Hallowell, M; et al.(2014). Fibroblast growth factor receptor 3 interacts with and activates TGFβ-activated kinase 1 tyrosine phosphorylation and NFκB signaling in multiple myeloma and bladder cancer. PLoS ONE, 9(1). doi: 10.1371/journal.pone.0086470. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/09n6212c
PLoS ONE
Cancer is a major public health problem worldwide. In the United States alone, 1 in 4 deaths is due to cancer and for 2013 a total of 1,660,290 new cancer cases and 580,350 cancer-related deaths are projected. Comprehensive profiling of multiple canc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::915a1fab7ad91a5846135d4f15fb9dec
https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/24466111/pdf/?tool=EBI
https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/24466111/pdf/?tool=EBI
Autor:
Pavel Krejci, William R. Wilcox, Daniel J. Donoghue, Paul J. Muchowski, Leslie M. Thompson, Lisa Salazar, Tamara A. Kashiwada
Ectopic activation of fibroblast growth factor receptor 3 (FGFR3) is associated with several cancers, including multiple myeloma (MM). FGFR3 inhibition in these cells inhibits proliferation and induces apoptosis, validating FGFR3 signaling as a thera
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::232ac79636a79e8f8d5a1cf026488c79
https://europepmc.org/articles/PMC2902846/
https://europepmc.org/articles/PMC2902846/
Autor:
Katarina Chlebova, Vitezslav Bryja, Pavel Krejci, Leslie M. Thompson, Alois Kozubík, William R. Wilcox, Tamara A. Kashiwada, Alena Salašová, Lisa Salazar
Publikováno v:
Krejci, Pavel; Salazar, Lisa; Kashiwada, Tamara A.; Chlebova, Katarina; Salasova, Alena; Thompson, Leslie Michels; et al.(2008). Analysis of STAT1 Activation by Six FGFR3 Mutants Associated with Skeletal Dysplasia Undermines Dominant Role of STAT1 in FGFR3 Signaling in Cartilage. PLoS One, 3(12), e3961. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/6qh76229
PLoS ONE
PLoS ONE, Vol 3, Iss 12, p e3961 (2008)
PLoS ONE
PLoS ONE, Vol 3, Iss 12, p e3961 (2008)
Activating mutations in FGFR3 tyrosine kinase cause several forms of human skeletal dysplasia. Although the mechanisms of FGFR3 action in cartilage are not completely understood, it is believed that the STAT1 transcription factor plays a central role
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::40032f5c7ffe8fbc41b378215fb909a2
http://www.escholarship.org/uc/item/6qh76229
http://www.escholarship.org/uc/item/6qh76229
Autor:
Tamara A. Kashiwada, Petra Jelínková, Helen S. Goodridge, Leslie M. Thompson, William R. Wilcox, Lisa Salazar, Pavel Krejci, Matthew J. Schibler
Publikováno v:
Journal of cell science. 121(Pt 3)
Activating mutations in fibroblast growth factor receptor 3 (FGFR3) cause several human skeletal dysplasias as a result of attenuation of cartilage growth. It is believed that FGFR3 inhibits chondrocyte proliferation via activation of signal transduc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dc7a411150f7aa81716368061c198c9e
https://pubmed.ncbi.nlm.nih.gov/18198189
https://pubmed.ncbi.nlm.nih.gov/18198189