Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Talia Levy"'
Publikováno v:
PLoS ONE, Vol 17, Iss 10, p e0276579 (2022)
Metabolic reprogramming is now considered a hallmark of cancer cells. KRas-driven cancer cells use glutaminolysis to generate the tricarboxylic acid cycle intermediate α-ketoglutarate via a transamination reaction between glutamate and oxaloacetate.
Externí odkaz:
https://doaj.org/article/f1e75bd3e5584ca3baf10df019cae074
Autor:
Vered H. Eisenberg, Ortal Sharlin, Merav Barbi, Talia Levy, Ayala Kobo-Greenhut, Nitza Peer, Izhar Ben Shlomo, Zimlichman Eyal, Yael Adler
Publikováno v:
International Journal for Quality in Health Care
Background Preventing medical errors is crucial, especially during crises like the COVID-19 pandemic. Failure Modes and Effects Analysis (FMEA) is the most widely used prospective hazard analysis in healthcare. FMEA relies on brainstorming by multi-d
Autor:
Jane McGlade, Orly Reiner, Irina Shin, Vanessa Zamor, Rebecca Haffner-Krausz, Noga Kozer, Tamar Sapir, Talia Levy
Publikováno v:
Cerebral Cortex. 28:3115-3128
The evolutionarily conserved Notch pathway plays an important role in regulation of stem cell renewal and cell fate determination in numerous organs, and as such is a key pathway in normal health and disease processes. Canonical Notch signaling is us
Autor:
Talia, Levy, Salim, Bader, Kay-Geert, Hermann, Gal, Yaniv, Gahl, Grinberg, Oshry, Mozes, Merav, Lidar, Iris, Eshed
Publikováno v:
The Israel Medical Association journal : IMAJ. 19(11)
Enthesopathy may lead to calcification of the stylohyoid ligament and can cause elongation of the styloid process (SP).To evaluate whether SP elongation is associated with two common enthesitis-related diseases: ankylosing spondylitis (AS) and diffus
Autor:
Tamar Sapir, Anna Kaplan, Michal Segal-Salto, Peter James, Karin Hansson, Talia Levy, Orly Reiner, Michael Frotscher, Michaela Schweizer
Publikováno v:
Human molecular genetics. 26(9)
Mutations in the depalmitoylation enzyme, palmitoyl protein thioesterase (PPT1), result in the early onset neurodegenerative disease known as Infantile Neuronal Ceroid Lipofuscinosis. Here, we provide proteomic evidence suggesting that PPT1 deficienc
Publikováno v:
Human molecular genetics 21(8), 1681-1692 (2011). doi:10.1093/hmg/ddr603
Human Molecular Genetics
Human Molecular Genetics
Microdeletions encompassing the MAPT (Tau) locus resulting in intellectual disability raised the hypothesis that Tau may regulate early functions in the developing brain. Our results indicate that neuronal migration was inhibited in mouse brains foll
Autor:
Sven Bergmann, Frédéric M. Coquelle, Irit Orr, Naama Barkai, Orly Reiner, Yehuda Brody, Sharon G. Wolf, Gregor Eichele, Daniela Bar-El, Tamar Sapir, Talia Levy
Publikováno v:
Cell Cycle. 5:976-983
The doublecortin-like (DCX) domains serve as protein-interaction platforms. DCX tandem domains appear in the product of the X-linked doublecortin (DCX) gene, in retinitis pigmentosa-1 (RP1), as well as in other gene products. Mutations in the human D
Autor:
Orly Reiner, Michal Caspi, Cynthia Koifman, Junken Aoki, Jan De Mey, Hiroyuki Arai, Frédéric M. Coquelle, Talia Levy
Publikováno v:
Journal of Biological Chemistry. 278:38740-38748
Mutations in one allele of the human LIS1 gene cause a severe brain malformation, lissencephaly. Although most LIS1 mutations involve deletions, several point mutations with a single amino acid alteration were described. Patients carrying these mutat
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 33(29)
Shootin1 has been ascribed a role in regulating polarization of primary hippocampal neurons. To better understand the possible role of Shootin1 in the developing brain, we identified a member of the kinesin superfamily, KIF20B, as a novel Shootin1 in
Autor:
Talia Levy, Orly Reiner, Michael Elbaum, Eva-Maria Mandelkow, Tamar Sapir, Thomas Timm, Anat Shmueli
Abnormal neuronal migration is manifested in brain malformations such as lissencephaly. The impairment in coordinated cell motility likely reflects a faulty mechanism of cell polarization or coupling between polarization and movement. Here we report
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e9f2c0dbfd738bf6f1c1fb45573ae728
https://europepmc.org/articles/PMC6671805/
https://europepmc.org/articles/PMC6671805/