Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Takatoshi Uchida"'
Autor:
Takatoshi Uchida, Shota Shimizu, Reiko Yamagishi, Suzumi M. Tokuoka, Yoshihiro Kita, Megumi Honjo, Makoto Aihara
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-12 (2021)
Abstract The trabecular meshwork (TM) constitutes the main pathway for aqueous humor drainage and is exposed to complex intraocular pressure fluctuations. The mechanism of homeostasis in which TM senses changes in intraocular pressure and leads to no
Externí odkaz:
https://doaj.org/article/3a790bb7932047738f10802992a9fe8f
Autor:
Takatoshi Uchida, Shota Shimizu, Reiko Yamagishi, Suzumi M. Tokuoka, Yoshihiro Kita, Rei Sakata, Megumi Honjo, Makoto Aihara
Publikováno v:
PLoS ONE, Vol 16, Iss 10 (2021)
Trabecular meshwork constitutes the conventional outflow pathway and controls intraocular pressure by regulating aqueous outflow. Mechanical stimulation has been studied as one of the triggers to regulate aqueous outflow in trabecular meshwork, but i
Externí odkaz:
https://doaj.org/article/22ef1f2aa6244fa88337b567288ff18d
Publikováno v:
FEBS Open Bio, Vol 6, Iss 12, Pp 1238-1247 (2016)
Oxidative stress is involved in the pathologies of corneal epithelial cells. However, the importance of specific antioxidant enzymes in corneal epithelial cells is not fully understood. The purpose of this study is to elucidate the role of glutathion
Externí odkaz:
https://doaj.org/article/8cf231968a9948beac1493f673b13d1c
Publikováno v:
PLoS ONE, Vol 10, Iss 6, p e0130467 (2015)
The purpose of the present study was to investigate the role of glutathione peroxidase 4 (GPx4) in glutamate-induced oxytosis in the retina.For in vitro studies, an immortalized rat retinal precursor cell line R28 was used. Cells were transfected wit
Externí odkaz:
https://doaj.org/article/8736cc49a2664d538b1907a487b61dda
Autor:
Reiko Yamagishi, Suzumi M. Tokuoka, Makoto Aihara, Shota Shimizu, Yoshihiro Kita, Megumi Honjo, Takatoshi Uchida
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-12 (2021)
The trabecular meshwork (TM) constitutes the main pathway for aqueous humor drainage and is exposed to complex intraocular pressure fluctuations. The mechanism of homeostasis in which TM senses changes in intraocular pressure and leads to normal leve
Publikováno v:
Journal of Ocular Pharmacology and Therapeutics. 35:535-541
Purpose: To determine whether the adiponectin receptor (AdipoR) agonist AdipoRon inhibits glutamate-induced neuronal cell death and to investigate the neuroprotective mechanism of AdipoRon in rat primary retinal ganglion cells (RGCs). Methods: The ex
Autor:
Takatoshi, Uchida, Shota, Shimizu, Reiko, Yamagishi, Suzumi M, Tokuoka, Yoshihiro, Kita, Megumi, Honjo, Makoto, Aihara
Publikováno v:
Scientific Reports
The trabecular meshwork (TM) constitutes the main pathway for aqueous humor drainage and is exposed to complex intraocular pressure fluctuations. The mechanism of homeostasis in which TM senses changes in intraocular pressure and leads to normal leve
Autor:
Suzumi M. Tokuoka, Shota Shimizu, Takatoshi Uchida, Rei Sakata, Reiko Yamagishi, Yoshihiro Kita, Makoto Aihara, Megumi Honjo
Publikováno v:
PLoS ONE, Vol 16, Iss 10, p e0258911 (2021)
PLoS ONE, Vol 16, Iss 10 (2021)
PLoS ONE
PLoS ONE, Vol 16, Iss 10 (2021)
PLoS ONE
Trabecular meshwork constitutes the conventional outflow pathway and controls intraocular pressure by regulating aqueous outflow. Mechanical stimulation has been studied as one of the triggers to regulate aqueous outflow in trabecular meshwork, but i
Publikováno v:
FEBS Open Bio
Oxidative stress is involved in the pathologies of corneal epithelial cells. However, the importance of specific antioxidant enzymes in corneal epithelial cells is not fully understood. The purpose of this study is to elucidate the role of glutathion
Publikováno v:
Current Eye Research. 42:380-385
To investigate the role of glutathione peroxidase 4 (GPx4) in corneal endothelial cells.An immortalized human corneal endothelial cell line was used. Cells were transfected with either siRNA specifically silencing GPx4 or scrambled control siRNA. Kno