Výsledky vyhledávání - "Takashima, Miyuki"

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Additional file 7 of S-1-propenylcysteine improves TNF-α-induced vascular endothelial barrier dysfunction by suppressing the GEF-H1/RhoA/Rac pathway

Autor: Kunimura, Kayo, Miki, Satomi, Takashima, Miyuki, Jun-Ichiro Suzuki

Additional file 6: Figure S6. Effects of GEF-H1 knockdown on downstream signaling of TNF-α, RhoA and Rac, in HUVECs. Effects of GEF-H1 knockdown on TNF-α-induced MLC2 phosphorylation (a), MYPT phosphorylation (b), ERK1/2 phosphorylation (c) and MLC

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Additional file 4 of S-1-propenylcysteine improves TNF-α-induced vascular endothelial barrier dysfunction by suppressing the GEF-H1/RhoA/Rac pathway

Autor: Kunimura, Kayo, Miki, Satomi, Takashima, Miyuki, Jun-Ichiro Suzuki

Additional file 3: Figure S3. Effects of S1PC on protein levels of TNF receptors and TRAF2 in HUVECs. a Effects of S1PC on the protein levels of TNFR1 and TNFR2 in HUVECs. The membrane and cytoplasmic proteins were extracted after the stimulation wit

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Additional file 6 of S-1-propenylcysteine improves TNF-α-induced vascular endothelial barrier dysfunction by suppressing the GEF-H1/RhoA/Rac pathway

Autor: Kunimura, Kayo, Miki, Satomi, Takashima, Miyuki, Jun-Ichiro Suzuki

Additional file 5: Figure S5. Effects of S1PC on the cell proliferation and morphology of HUVECs. a Effect of S1PC on the cell proliferation in HUVECs. Cell viability was measured with cell proliferation assay system after the treatment with S1PC (75

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Additional file 2 of S-1-propenylcysteine improves TNF-α-induced vascular endothelial barrier dysfunction by suppressing the GEF-H1/RhoA/Rac pathway

Autor: Kunimura, Kayo, Miki, Satomi, Takashima, Miyuki, Jun-Ichiro Suzuki

Additional file 1: Figure S1. Effects of AGE and its major sulfur-constituents on TNF-α-induced hyperpermeability of HUVECs. a The concentration-dependent effect of AGE on TNF-α-induced hyperpermeability of HUVECs. Vascular permeability assay was c

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Additional file 3 of S-1-propenylcysteine improves TNF-α-induced vascular endothelial barrier dysfunction by suppressing the GEF-H1/RhoA/Rac pathway

Autor: Kunimura, Kayo, Miki, Satomi, Takashima, Miyuki, Jun-Ichiro Suzuki

Additional file 2: Figure S2. The concentration-dependent effect of S1PC on thrombin-induced hyperpermeability of HUVECs. Vascular permeability assay was conducted after the stimulation with thrombin (1 U/mL) in the presence or absence of S1PC (75, 1

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Additional file 8 of S-1-propenylcysteine improves TNF-α-induced vascular endothelial barrier dysfunction by suppressing the GEF-H1/RhoA/Rac pathway

Autor: Kunimura, Kayo, Miki, Satomi, Takashima, Miyuki, Jun-Ichiro Suzuki

Additional file 7: Figure S7. Effects of ROCK inhibitor on downstream signaling of TNF-α, RhoA and Rac, in HUVECs. Effects of ROCK inhibitor on TNF-α-induced phosphorylation of MYPT (upper graph) and ERK1/2 (lower graph) were examined in HUVECs. Af

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Additional file 5 of S-1-propenylcysteine improves TNF-α-induced vascular endothelial barrier dysfunction by suppressing the GEF-H1/RhoA/Rac pathway

Autor: Kunimura, Kayo, Miki, Satomi, Takashima, Miyuki, Jun-Ichiro Suzuki

Additional file 4: Figure S4. Effects of major sulfur-containing constituents of AGE on TNF-α-induced GEF-H1 phosphorylation in HUVECs. Cell lysates were obtained after the stimulation with TNF-α (50 ng/mL) in the presence or absence of sulfur-cont

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