Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Takako Ishida"'
Publikováno v:
The Journal of Dermatology. 49
Autor:
Miyako Obuchi-Shimoji, Takashi Kato, Takako Ishida-Iwata, Ayaka Tahara-Mogi, Yuta Tanizaki, Mizue Meguro-Ishikawa, Megumi Ichisugi
Publikováno v:
Scientific Reports
The development of mammalian megakaryocytes (MKs) and platelets, which are thought to be absent in non-mammals, is primarily regulated by the thrombopoietin (TPO)/Mpl system. Although non-mammals possess nucleated thrombocytes instead of platelets, t
Autor:
Satoshi Tashiro, Yoichi Nakao, Rob W. M. Van Soest, Takako Ishida-Takaku, Hidekazu Suzuki, Motoki Takaku, Hitoshi Kurumizaka, Shintaro Ishigami, Takashi Kainuma
Publikováno v:
Genes to Cells. 16:427-436
Mutations and single-nucleotide polymorphisms affecting RAD51 gene function have been identified in several tumors, suggesting that the inappropriate expression of RAD51 activity may cause tumorigenesis. RAD51 is an essential enzyme for the homologou
Autor:
Jin Inoue, Hidekazu Suzuki, Takehiko Shibata, Takashi Kainuma, Yoshimasa Takizawa, Hitoshi Kurumizaka, Satoshi Tashiro, Tsutomu Mikawa, Takako Ishida
Publikováno v:
Nucleic Acids Research
RAD51, an essential eukaryotic DNA recombinase, promotes homologous pairing and strand exchange during homologous recombination and the recombinational repair of double strand breaks. Mutations that up- or down-regulate RAD51 gene expression have bee
Autor:
Yasutaka Ando, Mami Ishihara, Mitsuteru Akahoshi, Nobuhisa Mizuki, Nobuyoshi Kitaichi, Shinobu Takenaka, Shigeaki Ohno, Hitoshi Nakashima, Kenichi Namba, Takako Ishida, T. Shirakawa
Publikováno v:
Tissue Antigens. 71:564-567
CARD15 was first identified as a susceptibility gene for Crohn's disease. More recently, CARD15 mutations were shown to be associated with the pediatric granulomatous inflammatory diseases, Blau syndrome and early-onset sarcoidosis (EOS). The aim of
Publikováno v:
Genes to Cells. 13:91-103
The Rad51 protein, which catalyzes homologous-pairing and strand-exchange reactions, is an essential enzyme for homologous recombinational repair (HRR) and meiotic homologous recombination in eukaryotes. In humans, the conventional Rad51 (HsRad51) pr
Autor:
Emiko Isogai, Satoshi Kotake, Mami Ishihara, Youmei Hiraga, Shigeaki Ohno, Katsuya Suzuki, Hiromitsu Ono, Koh'ichi Kimura, Hiroshi Isogai, Takako Ishida, Koki Aoki
Publikováno v:
Graefe's Archive for Clinical and Experimental Ophthalmology. 236:280-284
· Background: Sarcoidosis is a multisystemic granulomatous disease of unknown etiology, while Lyme borreliosis is a multisystemic disorder caused by Borrelia burgdorferi. The purpose of this study is to evaluate the relationship between sarcoidosis
Publikováno v:
Tissue Antigens. 50:650-653
Genetic polymorphism in intron 6 of the LMP7 gene was investigated using polymerase chain reaction-restriction fragment length polymorphism in 90 unrelated healthy Japanese controls and 66 Japanese patients with sarcoidosis. Four alleles, including t
Autor:
Mami Ishihara, Etsuko Maruya, Hidetoshi Inoko, Taeko Naruse, M. Kagiya, Shigeaki Ohno, Takako Ishida, Nobuhisa Mizuki, Hiroo Saji
Publikováno v:
Tissue Antigens. 49:107-110
Sarcoidosis is a systemic granulomatous disease and the DRB1 gene of the DR subregion has been implicated for determining the genetic susceptibility to the disease. We evaluated the allelic variation of the TAP2 gene using the PCR-RFLP method as well
Autor:
Mami Ishihara, Hiroshi Isogai, Emiko Isogai, Takako Ishida, Yasuo Matsui, Shigeaki Ohno, Koh'ichi Kimura, Masaru Oritsu
Publikováno v:
Graefe's Archive for Clinical and Experimental Ophthalmology. 234:770-773
Lyme disease is a multisystemic disorder caused by the spirochete Borrelia burgdorferi, while sarcoidosis is a multisystemic granulomatous disease of unknown etiology. The purpose of this study was to evaluate the relationship between Lyme disease an