Zobrazeno 1 - 10
of 87
pro vyhledávání: '"Tae H. Oh"'
Publikováno v:
Neurobiology of Disease, Vol 39, Iss 3, Pp 272-282 (2010)
Here, we first demonstrated the neuroprotective effect of acupuncture after SCI. Acupuncture applied at two specific acupoints, Shuigou (GV26) and Yanglingquan (GB34) significantly alleviated apoptotic cell death of neurons and oligodendrocytes, ther
Externí odkaz:
https://doaj.org/article/015865eafda64b3192d53ebe3a82ae8a
Publikováno v:
PLoS ONE, Vol 8, Iss 9, p e73948 (2013)
Acupuncture (AP) has been used worldwide to relieve pain. However, the mechanism of action of AP is poorly understood. Here, we found that AP relieved neuropathic pain (NP) by inhibiting Jun-N-terminal kinase (JNK) activation in astrocytes after spin
Externí odkaz:
https://doaj.org/article/ffa705ad130c4d87bf93af5b6e358937
Publikováno v:
Nerve-Muscle Cell Trophic Communication ISBN: 9780429282430
Nerve-Muscle Cell Trophic Communication
Nerve-Muscle Cell Trophic Communication
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c1bf067162ace0c203904f93cd31d0a1
https://doi.org/10.1201/9780429282430-6
https://doi.org/10.1201/9780429282430-6
Publikováno v:
Glia. 61:1807-1821
Inflammation induced by microglial activation plays a pivotal role in progressive degeneration after traumatic spinal cord injury (SCI). Voltage-gated sodium channels (VGSCs) are also implicated in microglial activation following injury. However, dir
Publikováno v:
Brain. 135:2375-2389
After spinal cord injury, the disruption of blood-spinal cord barrier by activation of matrix metalloprotease is a critical event leading to infiltration of blood cells, inflammatory responses and neuronal cell death, contributing to permanent neurol
Publikováno v:
Endocrinology. 153:3815-3827
A delayed oligodendrocyte cell death after spinal cord injury (SCI) contributes to chronic demyelination of spared axons, leading to a permanent neurological deficit. Therefore, therapeutic approaches to prevent oligodendrocyte cell death after SCI s
Publikováno v:
Journal of Neurochemistry. 121:818-829
The disruption of blood-spinal cord barrier (BSCB) after spinal cord injury (SCI) elicits an intensive local inflammation by the infiltration of blood cells such as neutrophils and macrophages, leading to cell death and permanent neurological disabil
Publikováno v:
Endocrinology. 152:2377-2386
Here, we examined the protective effect of ghrelin on apoptotic cell death induced by hydrogen peroxide (H₂O₂) in primary oligodendrocyte cultures. Ghrelin receptor, growth hormone secretagogue receptor 1a, was expressed in mature oligodendrocyte
Publikováno v:
Neurobiology of Disease, Vol 39, Iss 3, Pp 272-282 (2010)
Here, we first demonstrated the neuroprotective effect of acupuncture after SCI. Acupuncture applied at two specific acupoints, Shuigou (GV26) and Yanglingquan (GB34) significantly alleviated apoptotic cell death of neurons and oligodendrocytes, ther
Autor:
Young Sook Yoo, Hyunju Chung, Young J. Oh, Tae Young Yune, Seungjoon Park, Jee Youn Lee, Tae H. Oh
Publikováno v:
Endocrinology. 151:3815-3826
Spinal cord injury (SCI) induces massive cell death, leading to permanent neurological disability. No satisfactory treatment is currently available. Ghrelin, a gastric hormone, is known to stimulate GH release from the hypothalamus and pituitary glan