Targeting of Intracellular TMEM16 Proteins to the Plasma Membrane and Activation by Purinergic Signaling

Autor: Rainer Schreiber, Jiraporn Ousingsawat, Karl Kunzelmann

Publikováno v: International Journal of Molecular Sciences, Vol 21, Iss 11, p 4065 (2020)

Anoctamins such as TMEM16A and TMEM16B are Ca2+-dependent Cl− channels activated through purinergic receptor signaling. TMEM16A (ANO1), TMEM16B (ANO2) and TMEM16F (ANO6) are predominantly expressed at the plasma membrane and are therefore well acce

Externí odkaz: https://doaj.org/article/01d26a6981f8440ab8a11a212a1f8e2d

Targeting of Intracellular TMEM16 Proteins to the Plasma Membrane and Activation by Purinergic Signaling

Autor: Schreiber, Rainer, Ousingsawat, Jiraporn, Kunzelmann, Karl

Publikováno v: International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 21, Iss 4065, p 4065 (2020)

Anoctamins such as TMEM16A and TMEM16B are Ca2+-dependent Cl− channels activated through purinergic receptor signaling. TMEM16A (ANO1), TMEM16B (ANO2) and TMEM16F (ANO6) are predominantly expressed at the plasma membrane and are therefore well acce

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::65fc2c6a9f88f23c415383711580666c
https://pubmed.ncbi.nlm.nih.gov/32517157

Gain of function of TMEM16E/ANO5 scrambling activity caused by a mutation associated with gnathodiaphyseal dysplasia

Autor: Joachim Scholz-Starke, Anna Boccaccio, Eleonora Di Zanni, Antonella Gradogna

Publikováno v: Cellular and molecular life sciences (Print. ed.) 75 (2018): 1657–1670. doi:10.1007/s00018-017-2704-9
info:cnr-pdr/source/autori:Di Zanni E, Gradogna A, Scholz-Starke J, Boccaccio A/titolo:Gain of function of TMEM16E%2FANO5 scrambling activity caused by a mutation associated with gnathodiaphyseal dysplasia/doi:10.1007%2Fs00018-017-2704-9/rivista:Cellular and molecular life sciences (Print. ed.)/anno:2018/pagina_da:1657/pagina_a:1670/intervallo_pagine:1657–1670/volume:75
Cellular and Molecular Life Sciences

Mutations in the human TMEM16E (ANO5) gene are associated both with the bone disease gnathodiaphyseal dysplasia (GDD; OMIM: 166260) and muscle dystrophies (OMIM: 611307, 613319). However, the physiological function of TMEM16E has remained unclear. We

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f01c6e6b414f9ca31b149946246f489b
https://publications.cnr.it/doc/387156

A role of TMEM16E carrying a scrambling domain in sperm motility

Autor: Gyobu, Sayuri

論文1ページ目の下部に著作権を表示すること。（© 2016, American Society for Microbiology. ）
0048
甲第19634号
医科博第72号
新制||医科||5(附属図書館)
32670
学位規則第4条第1項

Externí odkaz: http://hdl.handle.net/2433/215460

Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
K zobrazení výsledku je třeba se přihlásit.