Zobrazeno 1 - 7
of 7
pro vyhledávání: '"T. G. P. Saldeen"'
Publikováno v:
Current Research: Cardiology.
Publikováno v:
British Journal of Pharmacology. 120:305-311
Department of Medicine, University of Florida College of Medicine and the V.A. Medical Center, Gainesville, FL, U.S.A. and*Department of Forensic Medicine, University of Uppsala, Sweden1 The role of nitric oxide (NO) in ischaemia-reperfusion injury t
Publikováno v:
Journal of Cardiovascular Pharmacology. 27:283-289
Recombinant tissue-type plasminogen activator (rt-PA) administration rapidly restores blood flow in thrombosed coronary arteries, but coronary arteries often reocclude after initial thrombolysis. This occurs because of the short half-life of rt-PA an
Publikováno v:
Journal of Cardiovascular Pharmacology. 18:231-236
Thrombolytic effects of tissue-type plasminogen activator (t-PA) are limited by in vivo platelet activation and dynamic coronary vasoconstriction. To examine if the concurrent administration of a fibrin(ogen)-degradation product, pentapeptide 6A (Ala
Autor:
Wilmer W. Nichols, M. F. Ter Riet, Jawahar L. Mehta, F. A. Nicolini, V. K. Chandna, T. G. P. Saldeen, Daniel Lawson
Publikováno v:
Journal of Cardiovascular Pharmacology. 16:112-120
Release of superoxide radicals during the early phase of coronary reperfusion may result in degradation of endothelium-derived relaxing factor (EDRF), extension of myocardial injury, precipitation of arrhythmias, and stimulation of platelet aggregati
Publikováno v:
Circulation Research. 64:43-54
Previous studies indicate impairment of coronary arterial ring relaxation in response to acetylcholine (ACh) following coronary reperfusion, mediated via loss of endothelium-derived relaxing factor (EDRF). To examine if coronary vasodilator reserve i
Autor:
M. F. Ter Riet, Jawahar L. Mehta, W. W. Nichols, D. L. Lawson, W. H. Donnelly, T. G. P. Saldeen, L. Thompson
Publikováno v:
Circulation research. 65(5)
Previous studies indicate impairment of coronary arterial ring relaxation and loss of coronary vasodilator reserve after coronary artery occlusion and reperfusion. These changes are mediated in part through loss of endothelium-derived relaxing factor