Zobrazeno 1 - 10
of 12
pro vyhledávání: '"T. G. Karevina"'
Publikováno v:
Rossiiskii fiziologicheskii zhurnal imeni I.M. Sechenova. 84(4)
I.M. administration of L-thyroxin activated hepatic secretory function and changed the bile chemical contents. The substance P intensified production of bile and increased the absolute contents of free and conjugated bile acids, cholesterine, bilirub
Publikováno v:
Fiziologicheskii zhurnal imeni I.M. Sechenova. 79(6)
Lactine was found to depress the level of the histamine secretion of the gastric juice and to reduce the debit of hydrochloric acid in dogs. This peptide suppressed mainly the spontaneous choleresis and alters the compound composition of the bile. La
Autor:
T G, Karevina
Publikováno v:
Patologicheskaia fiziologiia i eksperimental'naia terapiia. (5)
It was demonstrated in experiments on guinea pigs with the use of a histamine model of ulcerogenesis that blocking agents of H1-histamine receptors produced a more marked gastro-protective effect than H2-antagonists. The results of the study confirm
Publikováno v:
Bulletin of Experimental Biology and Medicine. 108:1553-1556
Publikováno v:
Fiziologicheskii zhurnal. 31(3)
Autor:
S D, Groĭsman, T G, Karevina
Publikováno v:
Fiziologicheskii zhurnal. 28(3)
Publikováno v:
Biulleten' eksperimental'noi biologii i meditsiny. 99(3)
Changes in adenyl nucleotide and glycogen content in rat gastric tissue under the action of stressogenic factor and injection of the dopamine blocker metoclopramide and the dopamine precursor L-DOPA were investigated with the use of the "social stres
Publikováno v:
Biulleten' eksperimental'noi biologii i meditsiny. 108(11)
In experiments on vagotomized and intact rats with the use of two models of experimental gastric ulceration (injection of serotonin and stress) it was demonstrated that the inhibitory action of vagotomy on haemorrhagic gastric effectiveness was more
Publikováno v:
Fiziologicheskii zhurnal. 33(6)
Publikováno v:
Biulleten' eksperimental'noi biologii i meditsiny. 97(1)
Experiments on rats with the use of different exposures to stress (generalized electrization and "social stress") have demonstrated that stimulation of dopamine receptors localized in the central nervous system is one of the reasons for stress-induce