Zobrazeno 1 - 10
of 648
pro vyhledávání: '"T lymphocyte antigen"'
Autor:
Kanatbek Mukantayev, Kanat Tursunov, Zhansaya Adish, Darkhan Kanayev, Laura Tokhtarova, Malika Nurtleu, Bisultan Abirbekov
Publikováno v:
Veterinary World, Vol 17, Iss 9, Pp 2096-2103 (2024)
Background and Aim: Bovine interleukin 15 (bIL15) is a potential immunotherapy that can block the spread of bovine leukemia virus (BLV). However, immune checkpoints that maintain body homeostasis may reduce their effectiveness. Thus, an analysis of t
Externí odkaz:
https://doaj.org/article/6c75c2480b5d4eccb2add94dd4ca3d0c
Publikováno v:
Frontiers in Immunology, Vol 15 (2024)
Alveolar echinococcosis (AE) is a zoonotic parasitic disease caused by the infection of Echinococcus multilocularis (E. multilocularis) larvae. Cytotoxic T-lymphocyte antigen 4 (CTLA-4) produces inhibitory signals and induces T cell exhaustion, there
Externí odkaz:
https://doaj.org/article/82346988063a4cee85f46a1fe9a41cc9
Publikováno v:
BMC Urology, Vol 23, Iss 1, Pp 1-7 (2023)
Abstract Background Sarcomatoid carcinoma is a rare, high-grade malignancy with epithelial and mesenchymal components. It may be a good candidate for immunotherapy because it is associated with overexpression of programmed cell death ligand 1. Sarcom
Externí odkaz:
https://doaj.org/article/1131940943ec4ba98101b80be45cec3c
Autor:
Hossam El Idrissi, Ibtissam Balar
Publikováno v:
Middle East Journal of Cancer, Vol 14, Iss 1, Pp 1-16 (2023)
The aim of this research is to use bibliometric analysis to investigate the status and patterns of the 100 most frequently cited publications regarding the cytotoxic Tlymphocyte- associated protein (CTLA-4) research for cancer. The articles published
Externí odkaz:
https://doaj.org/article/c57500800fe643688b8dcba7f5549da1
Autor:
Yuki Yoshikawa, Michio Imamura, Masami Yamauchi, C. Nelson Hayes, Hiroshi Aikata, Wataru Okamoto, Yoshihiro Miyata, Morihito Okada, Noboru Hattori, Kazuhiko Sugiyama, Yukio Yoshioka, Shigeaki Toratani, Masaaki Takechi, Tatsuo Ichinohe, Tsutomu Ueda, Sachio Takeno, Tsuyoshi Kobayashi, Hideki Ohdan, Jun Teishima, Michihiro Hide, Yasushi Nagata, Yoshiki Kudo, Koji Iida, Kazuaki Chayama
Publikováno v:
BMC Cancer, Vol 22, Iss 1, Pp 1-9 (2022)
Abstract Background While immune checkpoint inhibitors (ICIs) occasionally cause immune-related adverse events (irAEs) in various organs, the prevalence of irAEs and potential risk factors have not been clarified. We identified irAE predictive factor
Externí odkaz:
https://doaj.org/article/9db6981ec3ff4d57acfc8ee546284f44
Autor:
Máté Krausz, Noriko Mitsuiki, Valeria Falcone, Johanna Komp, Sara Posadas-Cantera, Hanns-Martin Lorenz, Jiri Litzman, Daniel Wolff, Maria Kanariou, Anita Heinkele, Carsten Speckmann, Georg Häcker, Hartmut Hengel, Laura Gámez-Díaz, Bodo Grimbacher
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
Externí odkaz:
https://doaj.org/article/4811e267db8b4a0f951d41d027cdeb75
Publikováno v:
Frontiers in Oncology, Vol 12 (2022)
Externí odkaz:
https://doaj.org/article/d17947320c7841cbb4c11418c27cf358
Autor:
Máté Krausz, Noriko Mitsuiki, Valeria Falcone, Johanna Komp, Sara Posadas-Cantera, Hanns-Martin Lorenz, Jiri Litzman, Daniel Wolff, Maria Kanariou, Anita Heinkele, Carsten Speckmann, Georg Häcker, Hartmut Hengel, Laura Gámez-Díaz, Bodo Grimbacher
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
PurposeHeterozygous mutations in CTLA4 lead to an inborn error of immunity characterized by immune dysregulation and immunodeficiency, known as CTLA-4 insufficiency. Cohort studies on CTLA4 mutation carriers showed a reduced penetrance (around 70%) a
Externí odkaz:
https://doaj.org/article/64f0ae1228b94a4eaeab35fff84eeb9a
Akademický článek
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Publikováno v:
BMC Immunology, Vol 22, Iss 1, Pp 1-9 (2021)
Abstract Background The cytotoxic T-lymphocyte-associated antigen 4 (CTLA-4) is widely considered as a pivotal immune checkpoint molecule to suppress antitumor immunity. However, the significance of soluble CTLA-4 (sCTLA-4) remains unclear in the pat
Externí odkaz:
https://doaj.org/article/794355d19c7042e59461e86a8a16e2e1