Zobrazeno 1 - 10
of 14
pro vyhledávání: '"T M A, Peters"'
Autor:
Agnieszka Strzelecka, L. H. T. M. Vervoort-Peters, W. Matthijs Blankesteijn, Jacques Debets, Ben J. A. Janssen, Evangelos P. Daskalopoulos, Peter Leenders, Kevin C.M. Hermans
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 6 (2019)
Frontiers in cardiovascular medicine, 6:118. Frontiers Media S.A.
Frontiers in Cardiovascular Medicine
Frontiers in cardiovascular medicine, 6:118. Frontiers Media S.A.
Frontiers in Cardiovascular Medicine
Introduction: An inadequate wound healing following myocardial infarction (MI) is one of the main etiologies of heart failure (HF) development. Interventions aiming at improving this process may contribute to preserving cardiac function after MI. Our
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bfb7619e40b016470b0143d4aea81a9f
https://doi.org/10.3389/fcvm.2019.00118
https://doi.org/10.3389/fcvm.2019.00118
Autor:
S L, Smit, T M A, Peters, I A M, Gisbertz, W, Moolenaar, Y, Hendriks, H H, Vincent, D, Houtsma, O J L, Loosveld, A E, van Herwaarden, A J M, Rennings, D W, Swinkels
Publikováno v:
The Netherlands journal of medicine. 76(8)
Background: Type 2A hereditary haemochromatosis (type 2A HH) is a rare iron-loading disorder caused by mutations in the HFE2 gene, which encodes the HJV protein. We present characteristics, treatment and follow-up of subjects diagnosed with type 2A H
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::40209f7e739c6190ae7673f0ad8607a0
https://pubmed.ncbi.nlm.nih.gov/30362946
https://pubmed.ncbi.nlm.nih.gov/30362946
Autor:
T M A, Peters, A F M, Meulders, K, Redert, M L H, Cuijpers, A J M, Rennings, M C H, Janssen, N M A, Blijlevens, D W, Swinkels
Publikováno v:
The Netherlands journal of medicine. 75(2)
Type 3 hereditary haemochromatosis (HH) is a rare iron overload disorder caused by variants in the transferrin 2 receptor (TFR2) gene. We aim to present characteristics of patients diagnosed with TFR2-HH in the Netherlands, in order to increase knowl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c720b9591336ed46701ca544009b6d30
https://pubmed.ncbi.nlm.nih.gov/28276324
https://pubmed.ncbi.nlm.nih.gov/28276324
Autor:
Jack P.M. Cleutjens, Ben J. A. Janssen, H. T. M. Vervoort‐Peters, Jos F.M. Smits, W. Matthijs Blankesteijn, Mat J.A.P. Daemen, Agnieszka Strzelecka, Susanne W.M. van den Borne, Veerle A.M. van de Schans, Peter Lijnen
Publikováno v:
Cardiovascular research, 84(2), 273-282. Oxford University Press
Aims Our objective was to study the effect of the genetic background on the wound healing process after myocardial infarction (MI) in mice. Methods and results MI was induced in five different mouse strains (BalbC, C57Bl6, FVB, 129S6, and Swiss). At
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9ff2b1057594a7b7e9f1261ef64713b4
https://pubmed.ncbi.nlm.nih.gov/19542177
https://pubmed.ncbi.nlm.nih.gov/19542177
Autor:
W. Matthijs Blankesteijn, Mat J.A.P. Daemen, Peter Lijnen, J. Willem Voncken, Jos F.M. Smits, H. T. M. Vervoort‐Peters, Jagat Narula, Susanne W.M. van den Borne, Vivian E. H. Dahlmans
Publikováno v:
Journal of the American College of Cardiology, 51(22), 2184-2192. Elsevier USA
Objectives Our goal was to evaluate intercellular adhesion complex proteins in myocardium in human infarct rupture. Background Infarct rupture, a fatal complication of myocardial infarction (MI), has been attributed to a defective cell adhesion compl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa637ccfdcf715a75d2cab91ae961fc1
https://pure.amc.nl/en/publications/defective-intercellular-adhesion-complex-in-myocardium-predisposes-to-infarct-rupture-in-humans(6779bf4c-1517-4be5-908f-78522b884646).html
https://pure.amc.nl/en/publications/defective-intercellular-adhesion-complex-in-myocardium-predisposes-to-infarct-rupture-in-humans(6779bf4c-1517-4be5-908f-78522b884646).html
Autor:
G. J. Blauw, P. van Brummelen, P. Vermeij, P. A. van Zvieten, H. W. C. M. Boddeke, H. N. Doods, P. A. van Zwieten, A. H. Bom, D. J. Duncker, P. D. Verdouw, P. R. Saxena, M. J. A. P. Daemen, H. T. M. Vervoort-Peters, H. H. W. Thijssen, D. Davidesko, H. D. Batink, H. D. Bangik, K. J. van Charldrop, S. F. de Boer, J. L. Slangen, J. van der Gugten, W. S. De Loos, B. Bohus, W. De Jong, D. De Wied, M. A. H. de Zwart, H. van der Goot, H. Timmerman, A. Garritsen, A. P. IJzerman, W. Soudijn, J. B. Heynis, H. W. G. M. Boddeke, B. J. A. Janssen, H. van Essen, H. A. J. Struyker Boudier, J. F. M. Smits, T. J. W. Jap, C. E. J. Ketelaars, J. Bruinvels, M. J. Krielaart, D. M. J. Veenstra, G. J. T. H. Stolze, L. M. L. le Noble, D. W. Slaaf, G. J. Tangelder, H. C. Neijt, I. J. te Duits, J. J. Plomp, H. P. M. Vijverberg, J. A. M. Raaijmakers, R. J. E. van den Eijnde, F. S. Radhakishun, J. C. Stoof, A. H. Mulder, D. H. G. Versteeg, J. M. van Ree, F. A. M. Redegeld, J. Noordhoek, J. M. H. M. Reul, J. A. D. M. Tonnaer, E. R. de Kloet, J. Riezebos, A. de Jonge, C. G. J. Sweep, V. M. Wiegant, F. Th. M. van Amsterdam, M. Haas, J. Zaagsma, R. G. M. van Amsterdam, F. Brouwer, K. J. van Charldorp, R. van de Straat, J. de Vries, N. P. E. Vermeulden, P. L. M. van Giersbergen, J van Harten, RRECM Zeegers, P van Brumnelen, DD Breimer, P. N. M. van Heiningen, E. J. van Hoogdalem, A. G. de Boer, D. D. Breimer, J. van Wijngaarden, H. Wesseling
Publikováno v:
Pharmaceutisch Weekblad. 8:270-278
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4fba487c10f884e8c4f524c81f871237
https://doi.org/10.1007/bf01960072
https://doi.org/10.1007/bf01960072
Autor:
H. A. J. Struyker-Boudier, Ben J. A. Janssen, L. H. T. M. Vervoort-Peters, Henk H.W. Thijssen, F. H. M. Derkx, Jos F.M. Smits, H. van Essen
Publikováno v:
Journal of Hypertension. 7:447-455
To test the hypothesis that continuous intrarenal norepinephrine (NE) infusions produce hypertension via activation of afferent renal nerves (ARN), rats were subjected to complete renal denervation (RN-x), selective renal deafferentation (ARN-x) or s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ded298ab833e17bbbf864efbf1222972
https://doi.org/10.1097/00004872-198906000-00003
https://doi.org/10.1097/00004872-198906000-00003
Autor:
H. A. J. Struyker Boudier, C. M. Kasbergen, L. H. T. M. Vervoort-Peters, Jos F.M. Smits, J. C. S. Kleinjans
Publikováno v:
Life Sciences. 29:509-514
Nonadrenaline was infused intravenously for 5 days into conscious, unrestrained normotensive rats. Infusion of 40 μg per kg body weight per hour caused a chronic elevation of blood pressure, reaching a maximum increase of 28 ± 5 mm Hg on the fifth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6fe9477692f9ef7e53fda7dffc38ffca
https://doi.org/10.1016/0024-3205(81)90218-6
https://doi.org/10.1016/0024-3205(81)90218-6
Autor:
Jos F.M. Smits, Math J.M. Rousch, L. H. T. M. Vervoort-Peters, H. A. J. Struyker-Boudier, Henk H.W. Thijssen
Publikováno v:
Life Sciences. 38:137-145
Beta-adrenoceptor binding characteristics were determined in different fractions of rat kidney tubules using a [125Iodo]-(-)-cyanopindolol (ICYP) binding assay. The highest amount of binding sites was found in a fraction containing predominantly dist
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0466d3875832ef247d89e2966255210e
https://doi.org/10.1016/0024-3205(86)90005-6
https://doi.org/10.1016/0024-3205(86)90005-6
Autor:
H. A. J. Struyker-Boudier, Jos F.M. Smits, L. H. T. M. Vervoort-Peters, H. van Essen, Ben J. A. Janssen
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 13(4)
In the present study we examined sympathetic function and baroreceptor reflex sensitivity in adult spontaneously hypertensive rats (SHR) after a selective transection of afferent renal nerves in the prehypertensive and established phases of hypertens
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::011eedbef6eeee520abcbdea998b45b8
https://pubmed.ncbi.nlm.nih.gov/2564373
https://pubmed.ncbi.nlm.nih.gov/2564373