Zobrazeno 1 - 10
of 20
pro vyhledávání: '"T J, Kotlar"'
Autor:
Charles N. Rotimi, T. S. Schuh, Richard S. Cooper, Xiaofeng Zhu, T. J. Kotlar, S. Halbach, Amy Luke, Xingyi Guo, Anthony G. Comuzzie, William L. Lowe
Publikováno v:
International Journal of Obesity. 25:54-60
To examine the role of the Trp64Arg polymorphism in the beta 3-adrenergic receptor gene and the beta 3-adrenergic receptor gene locus in obesity-related traits in African Americans.A total of 687 individuals representing 193 African American families
Publikováno v:
Journal of Lipid Research, Vol 29, Iss 8, Pp 1087-1096 (1988)
Rat lymph chylomicrons were treated with rat heparin-releasable hepatic lipase (HL) or with bovine milk lipoprotein lipase (LPL). The ability of the resulting particles to be taken up by the liver in vivo was assessed following their infusion into th
Externí odkaz:
https://doaj.org/article/60dc254ed65942a3b9c50e2e404b58f2
Autor:
T J Kotlar, Jayme Borensztajn
Publikováno v:
Biochemical Journal. 269:539-542
The lipids extracted from chylomicrons, chylomicron remnants generated in vivo and hepatic-lipase-treated chylomicrons were emulsified by sonication. These emulsified particles retained the capacity of the native lipoproteins to be differentiated by
Publikováno v:
European journal of endocrinology. 146(5)
OBJECTIVE: To test further the hypothesis that autosomal dominant neurohypophyseal diabetes insipidus (adFNDI) is caused by heterozygous mutations in the vasopressin-neurophysin II (AVP-NPII) gene that exert a dominant negative effect by producing a
Publikováno v:
Journal of investigative medicine : the official publication of the American Federation for Clinical Research. 48(4)
Previous studies have demonstrated a role for tumor necrosis factor-alpha (TNF-alpha) in insulin resistance. A polymorphic variant of the TNF-alpha gene, the TNF2 allele, which is a guanine to adenine polymorphism at position -308 in the TNF-alpha pr
Autor:
J C, Achermann, W X, Gu, T J, Kotlar, J J, Meeks, L P, Sabacan, S B, Seminara, R L, Habiby, P C, Hindmarsh, D P, Bick, R J, Sherins, W F, Crowley, L C, Layman, J L, Jameson
Publikováno v:
The Journal of clinical endocrinology and metabolism. 84(12)
Although delayed puberty is relatively common and often familial, its molecular and pathophysiologic basis is poorly understood. In contrast, the molecular mechanisms underlying some forms of hypogonadotropic hypogonadism (HH) are clearer, following
Publikováno v:
Molecular genetics and metabolism. 67(1)
Autosomal-dominant familial neurohypophyseal diabetes insipidus (adFNDI) is caused by heterozygous mutations in the gene encoding vasopressin-neurophysin II (AVP-NPII) on chromosome 20p13. We analyzed the AVP-NP II gene in a family with adFNDI by dir
Publikováno v:
The Biochemical journal. 279
Rat lymph chylomicrons and chylomicron remnants were treated with trypsin or Pronase. The ability of the resulting apoprotein-free lipoproteins to be taken up by the isolated perfused rat liver, and to bind to isolated rat liver membranes, was examin
Publikováno v:
Biochemical Journal. 192:845-851
1. Rat lymph chylomicrons were depleted of their surface phospholipids by treatment with pure phospholipase A2 from Crotalus adamanteus venom. 2. About 80% of the phospholipids could be removed from the chylomicrons without any apparent effect on the
Publikováno v:
Journal of Lipid Research, Vol 29, Iss 8, Pp 1087-1096 (1988)
Rat lymph chylomicrons were treated with rat heparin-releasable hepatic lipase (HL) or with bovine milk lipoprotein lipase (LPL). The ability of the resulting particles to be taken up by the liver in vivo was assessed following their infusion into th