Zobrazeno 1 - 10
of 37
pro vyhledávání: '"T F Meyer"'
Autor:
D. Eletto, F. Mentucci, M. Vllahu, A. Voli, A. Petrella, F. Boccellato, T. F. Meyer, A. Porta, A. Tosco
Publikováno v:
Open Biology, Vol 12, Iss 12 (2022)
Chronic Helicobacter pylori infection is the leading cause of intestinal-type adenocarcinoma, as prolonged Helicobacter colonization triggers chronic active gastritis, which may evolve into adenocarcinoma of the intestinal type. In this environment,
Externí odkaz:
https://doaj.org/article/220002c5e2884a1bbb3014cf6a42668d
Autor:
null D. Eletto, null F. Mentucci, null M. Vllahu, null A. Voli, null A. Petrella, null F. Boccellato, null T. F. Meyer, null A. Porta, null A. Tosco
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8182a3f20dbfb6cdab72d2d5bea3cc85
https://doi.org/10.1098/rsob.220278/v2/response1
https://doi.org/10.1098/rsob.220278/v2/response1
Autor:
T. F. Meyer, J. Elias
Publikováno v:
Springer-Lehrbuch ISBN: 9783662486771
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::81e94262778b87f5342e0d111527cab1
https://doi.org/10.1007/978-3-662-48678-8_27
https://doi.org/10.1007/978-3-662-48678-8_27
Autor:
T. F. Meyer
Publikováno v:
Springer-Lehrbuch ISBN: 9783642241666
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5e1d5fe436deef9badd8c37d09b9af55
https://doi.org/10.1007/978-3-642-24167-3_27
https://doi.org/10.1007/978-3-642-24167-3_27
Publikováno v:
The Journal of Immunology. 152:735-742
Repeated low dose infections of the jird Meriones unguiculatus, with the filarial parasite Acanthocheilonema viteae cause a substantial reduction of the total worm burden, suggesting a parasite-driven immune mechanism that controls super-infections.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0e2a2c24a893e070747bda6db5c01923
https://doi.org/10.4049/jimmunol.152.2.735
https://doi.org/10.4049/jimmunol.152.2.735
Autor:
K. S. Bhat, C. P. Gibbs, O. Barrera, S. G. Morrison, F. Jähnig, A. Stem, E.-M Kupsch, T. F. Meyer, J. Swanson
Publikováno v:
Molecular Microbiology. 6:1073-1076
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::558e513dff54867785fe6feb6b1c68b4
https://doi.org/10.1111/j.1365-2958.1992.tb02172.x
https://doi.org/10.1111/j.1365-2958.1992.tb02172.x
Publikováno v:
Molecular microbiology. 42(3)
Helicobacter pylori colonizes the human stomach and is the causative agent of a variety of gastric diseases. After bacterial attachment, the H. pylori CagA protein is translocated into gastric epithelial cells and tyrosine phosphorylated. This proces
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::d2e7782aa560de1bd5ffde59a4d9b89e
https://pubmed.ncbi.nlm.nih.gov/11722731
https://pubmed.ncbi.nlm.nih.gov/11722731
Publikováno v:
Proteomics. 1(4)
The common human pathogen Helicobacter pylori is the major cause of gastric and duodenal ulcers. Based on the complete genome sequences of two independent isolates more than 1800 protein species have been resolved by two-dimensional gel electrophores
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::58330bb003de0569bf0fc17d176357a9
https://pubmed.ncbi.nlm.nih.gov/11681201
https://pubmed.ncbi.nlm.nih.gov/11681201
Publikováno v:
Proteomics. 1(4)
Helicobacter pylori is one of the most common bacterial pathogens that causes a variety of gastric diseases. During infection, the immuno-dominant H. pylori CagA protein is translocated and tyrosine-phosphorylated in gastric epithelial cells. We comp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::3fb4cb6fd47183d0221aa336500a7302
https://pubmed.ncbi.nlm.nih.gov/11681213
https://pubmed.ncbi.nlm.nih.gov/11681213
Autor:
I R, Henderson, J P, Nataro, J B, Kaper, T F, Meyer, S K, Farrand, D L, Burns, B B, Finlay, J W, St Geme
Publikováno v:
Trends in microbiology. 8(8)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::d4c69489442ec0dfdfe8860f4e7b9597
https://pubmed.ncbi.nlm.nih.gov/11041650
https://pubmed.ncbi.nlm.nih.gov/11041650