Zobrazeno 1 - 10 of 493 pro vyhledávání: '"T D Lee"'
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Akademický článek
Primary structure of apolipoprotein A-II from inbred mouse strain BALB/c.
Autor: C G Miller, T D Lee, R C LeBoeuf, J E Shively
Publikováno v: Journal of Lipid Research, Vol 28, Iss 3, Pp 311-319 (1987)
The primary structure of apolipoprotein A-II (apoA-II) isolated from the plasma high density lipoprotein (HDL) fraction of the inbred mouse strain BALB/c is described in this work. The complete 78 amino acid protein sequence was determined by proteol
Externí odkaz: https://doaj.org/article/ad079913662d4cab8283925a15b1d2f6
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5
Deviations of the lepton mapping matrix from the Harrison-Perkins-Scott form
Autor: T. D. Lee, R. Friedberg
Publikováno v: Chinese Physics C. 34:1547-1555
We propose a simple set of hypotheses governing the deviations of the leptonic mapping matrix from the Harrison-Perkins-Scott (HPS) form. These deviations are supposed to arise entirely from a perturbation of the mass matrix in the charged lepton sec
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::61056e3bdb038480e7d48a23f8557729
https://doi.org/10.1088/1674-1137/34/10/001
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6
A 'new' HLA-C specificity, CSH-2, restricted to Oriental populations
Autor: S. Q. Zheng, G. L. Zhang, T. M. Zhao, T. D. Lee, K. J. Bu, Pablo Rubinstein, P. Y. Lee, W. J. Gu
Publikováno v: Tissue Antigens. 29:77-82
The high frequency of C-locus blank alleles in different races suggests the existence of an undetected antigen. We have identified a new C-locus antigen, tentatively named CSH-2, which is present in Oriental populations, but is apparently not present
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::42f4a31ec2895a6bb9b3af15be6dbb34
https://doi.org/10.1111/j.1399-0039.1987.tb01555.x
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7
HLA-A, B and HLA-DR phenotypes in Mainland Chinese patients with diabetes mellitus
Autor: Glenn E. Rodey, M Shen, H Wong, Z Chi, T. D. Lee, T Zhao
Publikováno v: Tissue Antigens. 22:92-95
HLA-A, B and DR antigens were studied in Mainland Chinese diabetes mellitus patients and controls (31 Type I DM, 50 Type II DM, 105 controls). HLA-DR3 and HLA-A9 were increased in Type I diabetics only. An increase in HLA-DR4 was noted in Type I diab
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3946b583fc950d6005c1f85bec8f595a
https://doi.org/10.1111/j.1399-0039.1983.tb01173.x
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8
Model with strong γ 4 T -violation
Autor: R. Friedberg, T. D. Lee
Publikováno v: Chinese Physics C. 32:939-944
We extend the $T$ violating model of the paper on "Hidden symmetry of the CKM and neutrino-mapping matrices" by assuming its $T$-violating phases $\chi_\uparrow$ and $\chi_\downarrow$ to be large and the same, with $\chi=\chi_\uparrow=\chi_\downarrow
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::3b6a7f5e3cdba638f0fc586d7b9af1d4
https://doi.org/10.1088/1674-1137/32/12/001
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9
New insights to old problems
Autor: T. D. Lee
Publikováno v: Chinese Physics. 15:1125-1133
From the history of the $\theta$-$\tau$ puzzle and the discovery of parity non-conservation in 1956, we review the current status of discrete symmetry violations in the weak interaction. Possible origin of these symmetry violations are discussed.
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::efa402debfdfe85ebcb26d4efc44eceb
https://doi.org/10.1088/1009-1963/15/6/001
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10
HLA-DRB1 genotyping in patients with juvenile idiopathic arthritis in Taiwan
Autor: Li Chen Chen, T.-D. Lee, Tsung-Chieh Yao, Ming-Ling Kuo, C.-C. Yeh, C.-K. Shaw, Jing-Long Huang
Publikováno v: European Journal of Immunogenetics. 31:185-188
The object of this study was to investigate whether there is an association between HLA-DRB1 alleles and the development of juvenile idiopathic arthritis (JIA) in Taiwan. HLA-DRB1 alleles were studied in 60 patients with JIA and 200 healthy controls
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c25a233b7b355e410dc63b66e177c32
https://doi.org/10.1111/j.1365-2370.2004.00467.x
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