Zobrazeno 1 - 10
of 98
pro vyhledávání: '"T, Lejemtel"'
Autor:
S C, Wong, T, Sanborn, L A, Sleeper, J G, Webb, R, Pilchik, D, Hart, S, Mejnartowicz, T A, Antonelli, R, Lange, J K, French, G, Bergman, T, LeJemtel, J S, Hochman
Publikováno v:
Journal of the American College of Cardiology. 36(3 Suppl)
We sought to delineate the angiographic findings, clinical correlates and in-hospital outcomes in patients with cardiogenic shock (CS) complicating acute myocardial infarction.Patients with CS complicating acute myocardial infarction carry a grave pr
Autor:
J S, Hochman, C E, Buller, L A, Sleeper, J, Boland, V, Dzavik, T A, Sanborn, E, Godfrey, H D, White, J, Lim, T, LeJemtel
Publikováno v:
Journal of the American College of Cardiology. 36(3 Suppl)
This SHOCK Study report seeks to provide an overview of patients with cardiogenic shock (CS) complicating acute myocardial infarction (MI) and the outcome with various treatments. The outcome of patients undergoing revascularization in the SHOCK Tria
Autor:
A K, Jacobs, J K, French, J, Col, L A, Sleeper, J N, Slater, L, Carnendran, J, Boland, X, Jiang, T, LeJemtel, J S, Hochman
Publikováno v:
Journal of the American College of Cardiology. 36(3 Suppl)
We sought to determine the outcomes of patients with cardiogenic shock (CS) complicating non-ST-segment elevation acute myocardial infarction (MI).Such patients represent a high-risk (ST-segment depression) or low-risk (normal or nonspecific electroc
Publikováno v:
Journal of the American College of Cardiology. 36(3 Suppl)
We sought to evaluate the frequency of pulmonary congestion and associated clinical and hemodynamic findings in patients with suspected cardiogenic shock (CS).The prevalence of pulmonary congestion in the setting of CS is uncertain.The 571 SHOCK Tria
Publikováno v:
American heart journal. 137(2)
Cardiogenic shock (CS) is the leading cause of death in patients hospitalized with acute myocardial infarction (MI). Nonrandomized studies suggest reduced mortality rate with revascularization.The SHOCK trial is a multicenter, randomized, and unblind
Autor:
J N, Cohn, M B, Fowler, M R, Bristow, W S, Colucci, E M, Gilbert, V, Kinhal, S K, Krueger, T, Lejemtel, K A, Narahara, M, Packer, S T, Young, T L, Holcslaw, M A, Lukas
Publikováno v:
Journal of cardiac failure. 3(3)
Many patients remain markedly symptomatic despite optimal current therapy for heart failure. Beta-blockers have often been viewed as contraindicated in this group because of their potential adverse short-term effects on cardiac function.One hundred t
Publikováno v:
Journal of hypertension. Supplement : official journal of the International Society of Hypertension. 12(2)
Losartan is a specific angiotensin II receptor antagonist with no agonist properties. This agent permits evaluation of the response to selective angiotensin II antagonism in patients with congestive heart failure.A study was designed to assess the ac
Publikováno v:
Yearbook of Intensive Care and Emergency Medicine 1994 ISBN: 9783540576136
Cardiogenic shock remains the leading cause of death in patients hospitalized with acute myocardial infarction (AMI). The incidence (7.5%) and mortality (80%) were unchanged from 1975 to 1988 in a report of the Worcester, Massachusetts Community [1].
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0ee18624f648b52f61448f9da32169bf
https://doi.org/10.1007/978-3-642-85068-4_28
https://doi.org/10.1007/978-3-642-85068-4_28
Autor:
W S, Colucci, E H, Sonnenblick, K F, Adams, M, Berk, S C, Brozena, A J, Cowley, J M, Grabicki, S A, Kubo, T, LeJemtel, W A, Littler
Publikováno v:
Journal of the American College of Cardiology. 22(4 Suppl)
We describe the results of two placebo-controlled trials (MIL-1077 and MIL-1078) designed to evaluate the clinical efficacy of oral milrinone administered together with converting enzyme inhibitors to patients with congestive heart failure. Although
Publikováno v:
Circulation. 88(4 Pt 1)
BACKGROUND Losartan is a new specific angiotensin II receptor antagonist with no agonist properties that provides the opportunity to study the consequences of angiotensin II blockade. The objective of the present study was to evaluate the hemodynamic