TGFβ2‐Driven Ferritin Degradation and Subsequent Ferroptosis Underlie Salivary Gland Dysfunction in Postmenopausal Conditions.

Autor: Oh, Su‐Jeong^1,2,3 (AUTHOR), Shin, Ye Young^1,4 (AUTHOR), Ahn, Ji‐Su¹ (AUTHOR), Park, Hee‐Jeong^1,2,3 (AUTHOR), Kang, Min‐Jung¹ (AUTHOR), Shin, Tae‐Hoon⁵ (AUTHOR), Lee, Byung‐Chul^6,7 (AUTHOR), Kim, Won Kyu^8,9,10 (AUTHOR), Oh, Jung‐Min¹ (AUTHOR), Lee, Dongjun¹¹ (AUTHOR), Kim, Yun Hak¹² (AUTHOR), Kim, Ji Min¹³ (AUTHOR), Sung, Eui‐Suk¹⁴ (AUTHOR), Lee, Eun‐Woo^15,16 (AUTHOR), Jeong, Jee‐Heon¹⁷ (AUTHOR), Lee, Byung‐Joo¹³ (AUTHOR) voicelee@pusan.ac.kr, Seo, Yoojin¹ (AUTHOR) amaicat@pusan.ac.kr, Kim, Hyung‐Sik^1,2,3 (AUTHOR) hskimcell@pusan.ac.kr

Publikováno v: Advanced Science. 12/18/2024, Vol. 11 Issue 47, p1-18. 18p.

Modulation of Wnt Signaling Pathway in the Absence of Endogenous FGF2

Autor: Y Fei, L Xiao, T Doetschman, JD Coffin, MM Hurley

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::b2b064d06e289b8813f3f54d587f72c7
https://doi.org/10.1210/endo-meetings.2010.part2.p3.p2-130

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Transforming growth factor beta1 suppresses nonmetastatic colon cancer at an early stage of tumorigenesis

Autor: S J, Engle, J B, Hoying, G P, Boivin, I, Ormsby, P S, Gartside, T, Doetschman

Publikováno v: Cancer research. 59(14)

The transforming growth factor beta (TGF-beta) pathway is known to play an important role in both human and urine colon cancer. However, the staging, ligand specificity, and mechanism underlying the tumor suppressive activity of this pathway are unkn

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::adc3aa9f977e068358b18a912682d27b
https://pubmed.ncbi.nlm.nih.gov/10416598

Strain dependency of TGFbeta1 function during embryogenesis

Autor: S, Kallapur, I, Ormsby, T, Doetschman

Publikováno v: Molecular reproduction and development. 52(4)

There is incomplete penetrance to Tgfb1 knockout phenotypes. About 50% of Tgfb1 homozygous mutant (Tgfb1-/-) and 25% of Tgfb1 heterozygous (Tgfb1+/-) embryos die during embryogenesis. In a mixed NIH/Ola x C57BL/6J/Ola x 129 background partial embryon

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::f3b03c1e7e10b4f526168caf4ef2ab85
https://pubmed.ncbi.nlm.nih.gov/10092113