Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Szilvia Juhasz"'
Autor:
Lili Hegedus, Agnes Toth, Gabor M. Harami, Janos Palinkas, Nargis Karatayeva, Eniko Sajben-Nagy, Szabolcs Bene, Sara Afzali Jaktajdinani, Mihaly Kovacs, Szilvia Juhasz, Peter Burkovics
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-12 (2024)
Abstract Genome replication is frequently impeded by highly stable DNA secondary structures, including G-quadruplex (G4) DNA, that can hinder the progression of the replication fork. Human WRNIP1 (Werner helicase Interacting Protein 1) associates wit
Externí odkaz:
https://doaj.org/article/ef8693911000466fb2cdcfe50eeb72f6
Genome stability in human cells relies on the efficient repair of double-stranded DNA breaks, which is mainly achieved by homologous recombination (HR). Among the regulators of various cellular functions, Protein Phosphatase 4 (PP4) plays a pivotal r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a4e2bf2a2aa82cef8262fa2187b7102b
https://doi.org/10.1101/2023.05.31.542826
https://doi.org/10.1101/2023.05.31.542826
Autor:
David Balogh, Česlovas Venclovas, Dante Neculai, Himabindu Gali, Yathish Jagadheesh Achar, Szilvia Juhasz, Lajos Haracska, Sirano Dhe-Paganon, Mónika Mórocz
Publikováno v:
Nucleic Acids Research
Nucleic acids research, Oxford : Oxford University Press, 2015, Vol. 43, No. 21, p. 10277-10291
Nucleic acids research, Oxford : Oxford University Press, 2015, Vol. 43, No. 21, p. 10277-10291
Defects in the ability to respond properly to an unrepaired DNA lesion blocking replication promote genomic instability and cancer. Human HLTF, implicated in error-free replication of damaged DNA and tumour suppression, exhibits a HIRAN domain, a RIN
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::33c7cdd0a4ea717264d18b8d4622ce02
http://europepmc.org/articles/PMC4666394
http://europepmc.org/articles/PMC4666394
Autor:
Bill Jelen, Szilvia Juhasz
The 40 essential tips that all Excel users need to know. Crowdsourced by more than 300 contributors who collaborated on choosing the 40 best Excel tips, MrExcel XL provides users with a concise book that can be absorbed in under an hour. Includes a b
Autor:
Lajos Haracska, Peter Burkovics, Szilvia Juhasz, Sufang Zhang, Lumir Krejci, Marietta Y.W.T. Lee, Marek Sebesta, Judit Szabó
Publikováno v:
DNA Repair
Homologous recombination (HR) is essential for maintaining genomic integrity, which is challenged by a wide variety of potentially lethal DNA lesions. Regardless of the damage type, recombination is known to proceed by RAD51-mediated D-loop formation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::32c88cb1ec50206126e6b9a49b50cbd2
Autor:
Szilvia Juhasz, Zhihao Zhuang, Ildiko Hajdu, David Balogh, Mark A. Villamil, Lajos Haracska, Peter Burkovics
Publikováno v:
Nucleic Acids Research
Unrepaired DNA damage may arrest ongoing replication forks, potentially resulting in fork collapse, increased mutagenesis and genomic instability. Replication through DNA lesions depends on mono- and polyubiquitylation of proliferating cell nuclear a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2969aab2f35c489a03bf5d6bdcfda3ce
https://doi.org/10.1093/nar/gks850
https://doi.org/10.1093/nar/gks850
Autor:
Szilvia Juhasz, Valeria Szukacsov, Mónika Mórocz, Lajos Haracska, Himabindu Gali, Ildiko Hajdu, Károly Fátyol, Peter Burkovics
Publikováno v:
Nucleic Acids Research
DNA double-strand breaks (DSBs) can be generated not only by reactive agents but also as a result of replication fork collapse at unrepaired DNA lesions. Whereas ubiquitylation of proliferating cell nuclear antigen (PCNA) facilitates damage bypass, m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b9f7b05867a75e97f60f86dd0cb3bce9
http://europepmc.org/articles/PMC3401441
http://europepmc.org/articles/PMC3401441
Publikováno v:
Molecular & Cellular Oncology, Vol 5, Iss 5 (2018)
Chromatin remodeling is critical for the regulation of the DNA damage response. We highlight findings from our recent study showing that the deposition of the histone variant H3.3 by the alpha-thalassemia mental retardation X-linked protein (ATRX) an
Externí odkaz:
https://doaj.org/article/d9c5afd83c454a02b1027a6f7e8a658d