Zobrazeno 1 - 10
of 47
pro vyhledávání: '"Sylvie Dussault"'
Autor:
Sylvie Dussault, Michel Desjarlais, Nozha Raguema, Eric Boilard, Sylvain Chemtob, Alain Rivard
Publikováno v:
Cells, Vol 13, Iss 15, p 1243 (2024)
MicroRNAs (miRs) regulate physiological and pathological processes, including ischemia-induced angiogenesis and neovascularization. They can be transferred between cells by extracellular vesicles (EVs). However, the specific miRs that are packaged in
Externí odkaz:
https://doaj.org/article/d4281f5eaa49431e80ee70acc3c9a436
Autor:
Kadidia Diallo, Sylvie Dussault, Christophe Noll, Angel F. Lopez, Alain Rivard, André C. Carpentier, Gareth E. Lim
Publikováno v:
Molecular Metabolism, Vol 41, Iss , Pp 101052- (2020)
Objective: Adaptive thermogenesis, which is partly mediated by sympathetic input on brown adipose tissue (BAT), is a mechanism of heat production that confers protection against prolonged cold exposure. Various endogenous stimuli, for example, norepi
Externí odkaz:
https://doaj.org/article/cbb61bedabbe455ba3b0501e1ff26d2b
Publikováno v:
Frontiers in Genetics, Vol 11 (2020)
BackgroundClassical cardiovascular risk factors (CRFs) are associated with impaired angiogenic activities of bone marrow–derived proangiogenic cells (PACs) related to peripheral artery diseases (PADs) and ischemia-induced neovascularization. MicroR
Externí odkaz:
https://doaj.org/article/0356c733f27a4237a1d3977eb7b88125
Autor:
Raphael Mathieu, Sylvie Dussault, Michel Desjarlais, François Rivard, Wahiba Dhahri, Anik Cloutier, Anne-Monique Nuyt, Alain Rivard
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-10 (2017)
Abstract Adverse perinatal conditions can lead to developmental programming of cardiovascular diseases. Prematurely born infants are often exposed to high oxygen levels, which in animal models has been associated with endothelial dysfunction, hyperte
Externí odkaz:
https://doaj.org/article/f82ba77dde434355ab36b34a1d84dc4e
Publikováno v:
PLoS ONE, Vol 6, Iss 8, p e23308 (2011)
Aging is associated with increased oxidative stress levels and impaired neovascularization following ischemia. CuZnSOD has an important role to limit oxidative stress in the vasculature. Here we investigated the role of CuZnSOD for the modulation of
Externí odkaz:
https://doaj.org/article/9dc4b1ebd967439190a60bba697e948e
Publikováno v:
Atherosclerosis. 372:32-40
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2bca74064d56886711027a6aa89a00fa
https://doi.org/10.1016/j.atherosclerosis.2023.03.017
https://doi.org/10.1016/j.atherosclerosis.2023.03.017
The protein tyrosine phosphatase PTPRJ/DEP-1 has been implicated in negative growth regulation in endothelial cells, where its expression varies at transitions between proliferation and contact inhibition. However, in the same cells, DEP-1 has also b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f37c4496faf4f529b9d50b5453fb456
https://doi.org/10.1158/0008-5472.c.6508170.v1
https://doi.org/10.1158/0008-5472.c.6508170.v1
Legend to Supp. Fig. S1
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d17aae9cd5336cfd45b480de8e5ed728
https://doi.org/10.1158/0008-5472.22411179
https://doi.org/10.1158/0008-5472.22411179
Fig. S1. VEGF expression in tumors collected from WT or DEP-1 KO mice.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::15bc53556763e8d2bd8207278ca67122
https://doi.org/10.1158/0008-5472.22411182.v1
https://doi.org/10.1158/0008-5472.22411182.v1
Autor:
Veronica Sanchez, Alain Rivard, Michel Desjarlais, Sabah N. A. Hussain, Sharon Harel, Sylvie Dussault, François Rivard
Publikováno v:
Atherosclerosis. 289:73-84
Background and aims MicroRNA (miR)-146 is a key regulator of inflammation, endothelial activation and atherosclerosis. This study sought to define its potential role for the modulation of ischemia-induced neovascularization in atherosclerotic conditi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::56a43f702ad6e37b6013672b6adb5c5c
https://doi.org/10.1016/j.atherosclerosis.2019.08.010
https://doi.org/10.1016/j.atherosclerosis.2019.08.010