Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Syed Shamsh Tabrez"'
Autor:
Manish Chamoli, Anita Goyala, Syed Shamsh Tabrez, Atif Ahmed Siddiqui, Anupama Singh, Adam Antebi, Gordon J. Lithgow, Jennifer L. Watts, Arnab Mukhopadhyay
Publikováno v:
Nature Communications, Vol 11, Iss 1, Pp 1-13 (2020)
Metabolic reprogramming during Dietary Restriction (DR) activates cytoprotective gene expression. Here the authors show that PUFAs generated during DR signal via the p38-MAPK pathway to enhance cytoprotective gene expression, contributing to increase
Externí odkaz:
https://doaj.org/article/3c37c1fd9a7048f1bb216609bd91f5e7
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-13 (2017)
Alternative splicing coupled to nonsense-mediated decay (AS-NMD) is a conserved mechanism for post-transcriptional gene regulation. Here, the authors provide evidence that AS-NMD is enhanced during dietary restriction (DR) and is required for DR-medi
Externí odkaz:
https://doaj.org/article/f44c3f8b8fa7410ea1bd00d3f0cd9658
Autor:
Manish Chamoli, Gordon J. Lithgow, Anita Goyala, Arnab Mukhopadhyay, Jennifer L. Watts, Syed Shamsh Tabrez, Anupama Singh, Adam Antebi, Atif Ahmed Siddiqui
Publikováno v:
Nature Communications
Nature Communications, Vol 11, Iss 1, Pp 1-13 (2020)
Nat Commun
Nature Communications, Vol 11, Iss 1, Pp 1-13 (2020)
Nat Commun
The metabolic state of an organism instructs gene expression modalities, leading to changes in complex life history traits, such as longevity. Dietary restriction (DR), which positively affects health and life span across species, leads to metabolic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a86a9a9c97ae5fc401ab94b0857b0ea
https://pubmed.ncbi.nlm.nih.gov/32978396
https://pubmed.ncbi.nlm.nih.gov/32978396
Autor:
Sandeep B. Golegaonkar, Mashanipalya G. Jagadeeshaprasad, Srinivasa-Gopalan Sampathkumar, Mahesh J. Kulkarni, Sneha B. Bansode, Shalini Sethurathinam, Syed Shamsh Tabrez, Awadhesh Pandit, Arnab Mukhopadhyay
Publikováno v:
Aging Cell
Advanced glycation end products (AGEs) are formed when glucose reacts nonenzymatically with proteins; these modifications are implicated in aging and pathogenesis of many age-related diseases including type II diabetes, atherosclerosis, and neurodege
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a183990c37437da41ad594a06886bc00
http://europepmc.org/articles/PMC4406675
http://europepmc.org/articles/PMC4406675