Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Svetislav B, Tatić"'
Autor:
Héctor, Vázquez-Lorente, Duško M, Dundjerović, Svetislav B, Tatić, Sara, Rodríguez-Menéndez, Héctor, González-Iglesias, Cláudio M, Gomes, Ivan R, Paunović, Vesna V, Dragutinović
Publikováno v:
Biological trace element research.
Medullary Thyroid Carcinoma (MTC) constitutes around 5% of all thyroid cancers. Trace elements assessment has emerged as a useful strategy in the diagnostics of MTC combined with Matrix Metalloproteinases (MMPs) and Tissue Inhibitors of Matrix Metall
Autor:
Héctor Vázquez-Lorente, Duško M. Dunđerović, Svetislav B. Tatić, Sanja Radojević-Škodrić, Cláudio M. Gomes, Ivan R. Paunović, Vesna Dragutinović
Publikováno v:
Applied immunohistochemistrymolecular morphology : AIMM.
Medullary Thyroid Carcinoma (MTC) is a tumor of the neuroendocrine system. In recent years, the need to assess the MTC diagnostic-related parameters has emerged with the aim to elucidate the mechanisms involved in this pathology. The objective of thi
Autor:
Héctor Vázquez-Lorente, Duško M. Dundjerović, Svetislav B. Tatić, Sara Rodríguez-Menéndez, Héctor González-Iglesias, Cláudio M. Gomes, Ivan R. Paunović, Vesna V. Dragutinović
Publikováno v:
Scopus
Supplementary Information The online version contains supplemen- tary material available at https://doi.org/10.1007/s12011-022-03431-z.
Funding Funding for open access charge: Universidad de Granada / CBUA. This study was supported by Zinc-Net
Funding Funding for open access charge: Universidad de Granada / CBUA. This study was supported by Zinc-Net
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ba78325b8450acea2ab37bde923e4f78
http://hdl.handle.net/10651/67421
http://hdl.handle.net/10651/67421
Autor:
Valentina B, Ignjatović, Jelena R, Janković Miljuš, Jelena V, Rončević, Svetislav B, Tatić, Tijana M, Išić Denčić, Ilona Đ, Đorić, Sonja A, Šelemetjev
Publikováno v:
Histochemistry and cell biology. 157(2)
Papillary thyroid carcinoma (PTC), a common endocrine malignancy, presents a challenge from a prognostic standpoint. Molecular alterations underlying PTC progression include deregulation of focal adhesion kinase (FAK) at post-transcriptional and post