Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Sven Liebig"'
Autor:
Sven Liebig, Martin Neumann, Patricia Silva, Jutta Ortiz-Tanchez, Veronika Schulze, Konstandina Isaakidis, Cornelia Schlee, Michael P. Schroeder, Thomas Beder, Luc G. T. Morris, Timothy A. Chan, Lorenz Bastian, Thomas Burmeister, Stefan Schwartz, Nicola Gökbuget, Liliana H. Mochmann, Claudia D. Baldus
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-12 (2023)
Abstract FAT atypical cadherin 1 (FAT1), a transmembrane protein, is frequently mutated in various cancer types and has been described as context-dependent tumor suppressor or oncogene. The FAT1 gene is mutated in 12–16% of T-cell acute leukemia (T
Externí odkaz:
https://doaj.org/article/73aaef34737c42fdadc2a0d1ed2b4094
Autor:
Guus J. J. E. Heynen, Francis Baumgartner, Michael Heider, Upayan Patra, Maximilian Holz, Jan Braune, Melanie Kaiser, Isabell Schäffer, Stefanos A. Bamopoulos, Evelyn Ramberger, Arunima Murgai, Yuen Lam Dora Ng, Uta Margareta Demel, Dominik Laue, Sven Liebig, Josefine Krüger, Martin Janz, Axel Nogai, Markus Schick, Philipp Mertins, Stefan Müller, Florian Bassermann, Jan Krönke, Ulrich Keller, Matthias Wirth
Publikováno v:
Blood Advances
Proteasome inhibition is a highly effective treatment for multiple myeloma (MM). However, virtually all patients develop proteasome inhibitor resistance, which is associated with a poor prognosis. Hyperactive small ubiquitin-like modifier (SUMO) sign
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::62e3785fe11be986954c96a6487cb6c9
http://edoc.mdc-berlin.de/21976/2/21976suppl.zip
http://edoc.mdc-berlin.de/21976/2/21976suppl.zip
Autor:
T. Stroh, Marie Sittig, Lukas Poralla, Britta Siegmund, Sven Liebig, Rainer Glauben, Ulrike Erben
Publikováno v:
Journal of Cellular and Molecular Medicine
Modifying the chromatin structure and interacting with non-histone proteins, histone deacetylases (HDAC) are involved in vital cellular processes at different levels. We here specifically investigated the direct effects of HDAC5 in macrophage activat