Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Sven Bos"'
Autor:
Florian Kronenberg, Monique T. Mulder, Frank P.J. Leijten, Leonie C. van Vark-van der Zee, Jeanine E. Roeters van Lennep, Joyce Malfliet, Sven Bos, Shirley Uitte de Willige, Judith J. de Vries, Dingeman C. Rijken
Publikováno v:
Thrombosis Research, 198, 210-212. Elsevier Ltd.
Autor:
Swasti Indhirajanti, Monique T. Mulder, Sven Bos, Paul L A van Daele, Jeanine E. Roeters van Lennep, Ilze Bot
Publikováno v:
Atherosclerosis, 268, 152-156
Atherosclerosis, 268, 152-156. Elsevier Ireland Ltd
Atherosclerosis, 268, 152-156. Elsevier Ireland Ltd
Background and aims: Mast cells have been implicated in the development and progression of atherosclerosis in animal models and human autopsy studies. However, it is unknown whether long-term exposure to excess of mast cells is associated with cardio
Autor:
Natalie C. Ward, Sven Bos, Adrie J.M. Verhoeven, Michael Phillips, Gerald F. Watts, Eric J.G. Sijbrands
Publikováno v:
Journal of Clinical Lipidology, 11(3), 682-693. Elsevier
Background Familial hypercholesterolemia (FH) is the most common and serious monogenic disorder of lipid metabolism. The incidence of coronary artery disease (CAD) varies among both treated and untreated FH patients. Objective The aim of the study wa
Autor:
Jeanine E. Roeters van Lennep, Sven Bos, Koen Nieman, Arend F.L. Schinkel, Gerald F. Watts, Adrie J.M. Verhoeven, Martijne H C Duvekot, Gert Jan R ten Kate, Monique T. Mulder, Eric J.G. Sijbrands
Publikováno v:
Atherosclerosis, 256, 62-66. Elsevier Ireland Ltd
Background and aims Statins reduce subclinical atherosclerosis and premature atherosclerotic cardiovascular disease (ASCVD) in patients with familial hypercholesterolemia (FH). However, some FH patients still develop ASCVD despite statin therapy. We
Autor:
Meizi Jiang, Adrie J.M. Verhoeven, Sven Bos, Jeanine E. Roeters van Lennep, Ranitha Vongpromek, Gert-Jan R. ten Kate, Koen Nieman, Hideaki Bujo, Wolfgang J. Schneider, Eric J.G. Sijbrands, Monique T. Mulder
Publikováno v:
Atherosclerosis, 265, 299-304. Elsevier Ireland Ltd
Background and aims Despite statin treatment, a high prevalence of severe vascular calcification is found in patients with familial hypercholesterolemia (FH). We assessed the relation between the circulating soluble form of low-density lipoprotein re
Autor:
Mahtab, Sharifi, Elizabeth, Higginson, Sven, Bos, Angela, Gallivan, Darren, Harvey, Ka Wah, Li, Amali, Abeysekera, Angela, Haddon, Helen, Ashby, Kate E, Shipman, Jackie A, Cooper, Marta, Futema, Jeanine E, Roeters van Lennep, Eric J G, Sijbrands, Mourad, Labib, Devaki, Nair, Steve E, Humphries
Publikováno v:
Atherosclerosis
Atherosclerosis, 263, 405-411. Elsevier Ireland Ltd
Atherosclerosis, 263, 405-411. Elsevier Ireland Ltd
Background and aims Familial hypercholesterolemia (FH) is a common inherited disorder of low density lipoprotein-cholesterol (LDL-C) metabolism. It is associated with higher risk of premature coronary heart disease. Around 60% of patients with a clin
Autor:
Gerald F. Watts, Adriana C. Touw-Blommesteijn, Eric J.G. Sijbrands, Adrie J.M. Verhoeven, Martijne H C Duvekot, Jeanine E. Roeters van Lennep, Monique T. Mulder, Sven Bos
Publikováno v:
Atherosclerosis, 242(1), 226-229. Elsevier Ireland Ltd
Background: Lipoprotein (a), also called Lp(a), is a cardiovascular disease (CVD) risk factor. Statins do not lower Lp(a), this may at least partly explain residual CVD risk in statin-treated patients with familial hypercholesterolemia (FH). We inves
Autor:
Aswin Verhoeven, P. J. De Feyter, Monique T. Mulder, Ranitha Vongpromek, R. Yahya, Sven Bos, J.E. Roeters van Lennep, Florian Kronenberg, G. J. R. Ten Kate, Eric J.G. Sijbrands
Publikováno v:
Journal of Internal Medicine, 278(2), 166-173. Wiley-Blackwell Publishing Ltd
Objectives Lipoprotein(a) [Lp(a)] is an independent risk factor for aortic valve stenosis and aortic valve calcification (AVC) in the general population. In this study, we determined the association between AVC and both plasma Lp(a) levels and apolip
Autor:
Sven Bos, Adriaan Moelker, Gert-Jan R. ten Kate, Koen Nieman, Janneke G. Langendonk, Akira Kurata, Admir Dedic, Jeanine E. Roeters van Lennep, Lisan A. Neefjes, Pim J. de Feyter, Eric J.G. Sijbrands, Anho Liem, Gabriel P. Krestin
Publikováno v:
Journal of the American College of Cardiology, 66(24), 2687-2695. Elsevier Inc.
BACKGROUND Familial hypercholesterolemia is typically caused by LDL receptor (LDLR) mutations that result in elevated levels of LDL cholesterol (LDL-C). In homozygous FH, the prevalence of aortic valve calcification (AoVC) reaches 100% and is often s
Autor:
Sven Bos, Willem J. Kop, Kadir Caliskan, Corline Brouwers, Jeanine E. Roeters van Lennep, Susanne S. Pedersen, Eric J.G. Sijbrands
Publikováno v:
International Journal of Behavioral Medicine, 22(6), 717-725. Springer
International Journal of Behavioral Medicine, 22(6), 717-725. Routledge
Brouwers, C, Caliskan, K, Bos, S, Van Lennep, J E R, Sijbrands, E J, Kop, W J & Pedersen, S S 2015, ' Health Status and Psychological Distress in Patients with Non-compaction Cardiomyopathy : The Role of Burden Related to Symptoms and Genetic Vulnerability ', International Journal of Behavioral Medicine, vol. 22, no. 6, pp. 717-725 . https://doi.org/10.1007/s12529-015-9475-1
International Journal of Behavioral Medicine, 22(6), 717-725. Routledge
Brouwers, C, Caliskan, K, Bos, S, Van Lennep, J E R, Sijbrands, E J, Kop, W J & Pedersen, S S 2015, ' Health Status and Psychological Distress in Patients with Non-compaction Cardiomyopathy : The Role of Burden Related to Symptoms and Genetic Vulnerability ', International Journal of Behavioral Medicine, vol. 22, no. 6, pp. 717-725 . https://doi.org/10.1007/s12529-015-9475-1
BackgroundNon-compaction cardiomyopathy (NCCM) is a cardiomyopathy characterized by left ventricular tribeculae and deep intertrabecular recesses. Because of its genetic underpinnings and physical disease burden, noncompaction cardiomyopathy is expec