Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Suzette Maynard"'
Autor:
Stella M. Davies, Susan R. Rose, Jared Sipple, Suzette Maynard, Parinda A. Mehta, Jie Li, Qishen Pang
Publikováno v:
Antioxidants & Redox Signaling. 17:1083-1098
Aims: Insulin resistance is a hallmark of obesity and type 2 diabetes. Reactive oxygen species (ROS) have been proposed to play a causal role in insulin resistance. However, evidence linking ROS to insulin resistance in disease settings has been scan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2686e11791c981426f157da2b897af5
https://doi.org/10.1089/ars.2011.4417
https://doi.org/10.1089/ars.2011.4417
Publikováno v:
Blood. 115(8)
The molecular pathway by which Fanconi anemia (FA) proteins function in oxidative stress response has not been defined. Here we report the functional interaction of the FA protein Fanconi anemia complementation group D2 (FANCD2) and the forkhead tran
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d16f39fc041bd78830232b1e9a88c86f
https://pubmed.ncbi.nlm.nih.gov/20040763
https://pubmed.ncbi.nlm.nih.gov/20040763
Publikováno v:
Blood. 114:1089-1089
Abstract 1089 Poster Board I-111 Fanconi anemia (FA) is a human genomic instability syndrome that is uniquely sensitive to oxidative stress. Members of the FA protein family are involved in repair of genetic damage caused by DNA cross-linkers. The mo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d54c30f2ade608a814b0b5b215b0d374
https://doi.org/10.1182/blood.v114.22.1089.1089
https://doi.org/10.1182/blood.v114.22.1089.1089
Publikováno v:
Web of Science
Abstract 3098 Poster Board III-35 In a subset of AML with a normal karyotype, a frame-shift mutation in the extreme C-terminal of the nucleophosmin (NPM) gene results in the creation of a nuclear export signal, generating a mutant NPM protein (NPMc)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3bb430e15ff9d30ae74682e376c5ff1e
https://publons.com/wos-op/publon/34981474/
https://publons.com/wos-op/publon/34981474/
