Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Suvi Vartiainen"'
Autor:
Harriet Walker, Suvi Vartiainen, Juha Apajalahti, Jules Taylor-Pickard, Ivana Nikodinoska, Colm A. Moran
Publikováno v:
Animals, Vol 14, Iss 2, p 328 (2024)
The importance of enzymes in the poultry industry is ever increasing because they help to extract as many nutrients as possible from the raw material available and reduce environmental impacts. Therefore, an experiment was conducted to examine the ef
Externí odkaz:
https://doaj.org/article/2e61ff7a33cd44e28ee1dfdabb1115a5
Publikováno v:
Toxins, Vol 12, Iss 1, p 37 (2020)
Ochratoxin A (OTA) is a common mycotoxin contaminant in animal feed. When absorbed from the gastrointestinal tract, OTA has a propensity for pathological effects on animal health and deposition in animal tissues. In this study, the potential of yeast
Externí odkaz:
https://doaj.org/article/b355f1613493414e8933c73c03bac2c7
Autor:
Suvi Vartiainen, Shanjun Chen, Jack George, Tea Tuomela, Kaisa R. Luoto, Kevin M. C. O’Dell, Howard T. Jacobs
Publikováno v:
Disease Models & Mechanisms, Vol 7, Iss 6, Pp 635-648 (2014)
A point mutation in the Drosophila gene that codes for the major adult isoform of adenine nuclear translocase (ANT) represents a model for human diseases that are associated with ANT insufficiency [stress-sensitive B1 (sesB1)]. We characterized the o
Externí odkaz:
https://doaj.org/article/dbb7c5b6274140e1b3e8be3b394c9f0a
Publikováno v:
Neurobiology of Disease, Vol 22, Iss 3, Pp 477-486 (2006)
α-Synuclein containing cellular inclusions are a hallmark of Parkinson Disease, Lewy Body Dementia, and Multiple System Atrophy. A genome wide expression screen was performed in C. elegans overexpressing both wild-type and A53T human α-synuclein. 4
Externí odkaz:
https://doaj.org/article/3b15da76342b485bbce3b4f751c3d09f
Publikováno v:
Toxins, Vol 12, Iss 1, p 37 (2020)
Toxins
Volume 12
Issue 1
Toxins
Volume 12
Issue 1
Ochratoxin A (OTA) is a common mycotoxin contaminant in animal feed. When absorbed from the gastrointestinal tract, OTA has a propensity for pathological effects on animal health and deposition in animal tissues. In this study, the potential of yeast
Autor:
Steven P. Gross, Neema Hariri, Babu Reddy Janakaloti Narayanareddy, Diane K. O'Dowd, Suvi Vartiainen
Publikováno v:
Traffic. 15:762-771
There is an increasing interest in factors that can impede cargo transport by molecular motors inside the cell. Although potentially relevant (Yi JY, Ori-McKenney KM, McKenney RJ, Vershinin M, Gross SP, Vallee RB. High-resolution imaging reveals indi
Autor:
Pierre Rustin, Bettina Hutz, Manuel Portero-Otin, Alba Naudí, Takao Yagi, Akemi Matsuno-Yagi, George D. Mcilroy, Howard T. Jacobs, Alberto Sanz, Angela M. Wilson, Essi Kiviranta, Simo Ellilä, Reineld Pamplona, Eric Dufour, Mikko Soikkeli, Matti Lakanmaa, Esko Kemppainen, Mariona Jové, Rhoda Stefanatos, Suvi Vartiainen, Kia K. Kemppainen, Akbar Zeb, Tea Tuomela
Publikováno v:
Proceedings of the National Academy of Sciences. 107:9105-9110
Mutations in mitochondrial oxidative phosphorylation complex I are associated with multiple pathologies, and complex I has been proposed as a crucial regulator of animal longevity. In yeast, the single-subunit NADH dehydrogenase Ndi1 serves as a non-
Autor:
Howard T. Jacobs, Kia K. Kemppainen, Eero Mustalahti, Jongkyeong Chung, Massimo Zeviani, Tea Tuomela, Suvi Vartiainen, Daniel J. M. Fernández-Ayala, Marek Babusiak, Rodolfo Costa, Pierre Rustin, Kevin M.C. O'Dell, Alberto Sanz
Publikováno v:
Digital.CSIC. Repositorio Institucional del CSIC
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Defects in mitochondrial OXPHOS are associated with diverse and mostly intractable human disorders. The single-subunit alternative oxidase (AOX) found in many eukaryotes, but not in arthropods or vertebrates, offers a potential bypass of the OXPHOS c
Publikováno v:
Current Genomics. 6:97-107
Autor:
Jouni Sirviö, Garry Wong, Suvi Vartiainen, Merja Lakso, Richard Nass, James H. Thomas, Anu Maarit Moilanen, Randy D. Blakely
Publikováno v:
Journal of Neurochemistry. 86:165-172
Overexpression of human alpha-synuclein in model systems, including cultured neurons, drosophila and mice, leads to biochemical and pathological changes that mimic synucleopathies including Parkinson's disease. We have overexpressed both wild-type (W