Zobrazeno 1 - 8 of 8 pro vyhledávání: '"Susana, Lucea"'
1
Inhibition of phosphate transport by NAD+/NADH in brush border membrane vesicles
Autor: Susana Lucea, Natalia Guillén, Cecilia Sosa, Víctor Sorribas
Publikováno v: Zaguán. Repositorio Digital de la Universidad de Zaragoza
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Nicotinamide is an important regulator of Pi homeostasis after conversion into NAD+/NADH. In this work, we have studied the classical inhibition of Pi transport by these compounds in the brush border membrane vesicles (BBMV) of rat kidney and rat int
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c0bf3f0ba6be76c62b5394c17bd06e13
https://doi.org/10.1152/ajpcell.00404.2021
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2
Inhibition of phosphate transport by NAD
Autor: Susana, Lucea, Natalia, Guillén, Cecilia, Sosa, Víctor, Sorribas
Publikováno v: American journal of physiology. Cell physiology. 322(5)
Nicotinamide is an important regulator of Pi homeostasis after conversion into NAD
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::30fe9e5821928d05f5550fb8cd5ce3dd
https://pubmed.ncbi.nlm.nih.gov/35264014
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6
Effects of oral exposure to arsenite on arsenic metabolism and transport in rat kidney
Autor: Victor Sorribas, Susana Lucea, Cecilia Sosa, Natalia Guillén
Publikováno v: Zaguán. Repositorio Digital de la Universidad de Zaragoza
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Nephrotoxicity is within the recognized toxic effects of arsenic. In this study we assessed the effect of arsenite on the renal capacity to metabolize and handle arsenicals in rats exposed to drinking water with 0, 1, 5, or 10 ppm sodium arsenite for
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::18a8d769557acea6ab861282b3888715
https://pubmed.ncbi.nlm.nih.gov/32736004
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7
Identifying early pathogenic events during vascular calcification in uremic rats
Autor: Luis Hortells, Cecilia Sosa, Susana Lucea, Natalia Guillén, Victor Sorribas, Ángel Millán
Publikováno v: Digital.CSIC. Repositorio Institucional del CSIC
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Vascular calcification in chronic kidney disease is a very complex process traditionally explained in multifactorial terms. Here we sought to clarify relevance of the diverse agents acting on vascular calcification in uremic rats and distinguish betw
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d696054c5f6d983db03d11e0e3801268
http://hdl.handle.net/10261/182014
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8
CALHM1 and its polymorphism P86L differentially control Ca2+ homeostasis, mitogen-activated protein kinase signaling, and cell vulnerability upon exposure to amyloid β
Autor: Javier Egea, Lamia Mouhid, Ana J. Moreno-Ortega, Manuela G. López, Susana Lucea, Izaskun Buendia, Ana Ruiz-Nuño, María F. Cano-Abad
Publikováno v: Biblos-e Archivo. Repositorio Institucional de la UAM
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Aging Cell
The mutated form of the Ca2+ channel CALHM1 (Ca2+ homeostasis modulator 1), P86L-CALHM1, has been correlated with early onset of Alzheimer’s disease (AD). P86L-CALHM1 increases production of amyloid beta (Ab) upon extracellular Ca2+ removal and its
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b7267996589ee5fb613199141ac30861
http://hdl.handle.net/10486/671438
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