Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Susan Longar"'
Autor:
Vicki Woolworth, Shigeki Imaizumi, Bryan M. Pereira, Sylvia F. Chen, Robert A. Fishman, Pak H. Chan, Lillian Chu, Susan Longar, Ki Moore, Lars Hillered, Albert C. H. Yu
Publikováno v:
Annals of the New York Academy of Sciences. 559:237-247
Publikováno v:
Brain Research. 277:329-337
Development of brain edema following various pathological insults occurs after some delay. The mechanism of the delay is poorly understood. Using an in vivo model of cold-injury to study the time course of edema development, the present study indicat
Publikováno v:
Journal of neurochemistry. 48(6)
Traumatic spinal cord injury has recently been shown to cause a rapid increase in free fatty acids (FFAs) and lipid degradation in cats. The present studies report a more delayed, time-dependent increase in FFAs and a concomitant decrease in phosphol
Publikováno v:
Neural Membranes ISBN: 9781461256380
Cerebral edema is defined as an increase in brain volume due to an increase in its water and sodium content. Based on neuropathological and experimental observations, brain edema has been divided into three major categories (Klatzo, 1967: Fishman, 19
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::29d5ea5afe626260bdfe14d20e1853dc
https://doi.org/10.1007/978-1-4612-5636-6_6
https://doi.org/10.1007/978-1-4612-5636-6_6
Publisher Summary This chapter explains that free polyenoic fatty acids (PUFAs), especially arachidonic acid and docosahexaenoic acid are rapidly released following ischemia, electroconvulsive seizures, and various pathological insults. Free PUFAs an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::87c6036f6c68cefeb66682723aafefdb
https://doi.org/10.1016/s0079-6123(08)61986-x
https://doi.org/10.1016/s0079-6123(08)61986-x
Publikováno v:
Annals of neurology. 21(6)
Oxygen-derived free radicals and membrane lipid peroxidation have been postulated to be involved in brain edema and cell death, secondary to ischemia and traumatic injury. Using a model of brain edema induced by cold-induced injury, we have demonstra
Publikováno v:
Neurology. 34:315-315
We studied the cerebral effects of oxygen-derived free radicals generated from the xanthine oxidase/hypoxanthine/ADP-Fe3+ system. Xanthine oxidase/hypoxanthine/ADP-Fe3+ solution (0.1 ml) was infused into caudate putamen, and brain was frozen rapidly