Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Susan Canny"'
Autor:
Sonwabile Dzanibe, Aaron J. Wilk, Susan Canny, Thanmayi Ranganath, Berenice Alinde, Florian Rubelt, Huang Huang, Mark M. Davis, Susan P. Holmes, Heather B. Jaspan, Catherine A. Blish, Clive M. Gray
Publikováno v:
Nature Communications, Vol 15, Iss 1, Pp 1-15 (2024)
Abstract While preventing vertical HIV transmission has been very successful, HIV-exposed uninfected infants (iHEU) experience an elevated risk to infections compared to HIV-unexposed and uninfected infants (iHUU). Here we present a longitudinal mult
Externí odkaz:
https://doaj.org/article/44079eeedfed4aefb26588fb0716959c
Autor:
Susan Canny, Elizabeth Mellins
Publikováno v:
F1000Research, Vol 6 (2017)
Systemic juvenile idiopathic arthritis (sJIA) and its most significant complication, macrophage activation syndrome (MAS), have traditionally been treated with steroids and non-steroidal anti-inflammatory medications. However, the introduction of bio
Externí odkaz:
https://doaj.org/article/59ea3eeefcf3470fad64e3e26a5f6c8e
Autor:
Edward I. Cho, Marie E. Egan, Paula A. Cahill, Susan Canny, Judith Glöckner-Pagel, Naomi Balamuth, Catherine A. Ambrose, Carsten A. Wagner, John P. Geibel, Michael J. Caplan, Lamiko Pappoe
Publikováno v:
Nature Medicine. 8:485-492
The most common mutation in cystic fibrosis, Delta F508, results in a cystic fibrosis transmembrane conductance regulator (CFTR) protein that is retained in the endoplasmic reticulum (ER). Retention is dependent upon chaperone proteins, many of which
Publikováno v:
Handbook of Pharmaceutical Public Policy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2940a30220e9a4cc0546df5e9b0b4e85
https://doi.org/10.1201/b14469-3
https://doi.org/10.1201/b14469-3
Autor:
Scott A. Weiner, Gergely L. Lukacs, Vanathy Rajendran, Daniel Rubin, Susan Canny, Marie E. Egan, Michael J. Caplan, Marilyn Pearson, Kai Du, Judith Glöckner-Pagel
Publikováno v:
Science (New York, N.Y.). 304(5670)
Cystic fibrosis is caused by mutations in the gene encoding the cystic fibrosis transmembrane conductance regulator (CFTR). The most common mutation, ΔF508, results in the production of a misfolded CFTR protein that is retained in the endoplasmic re