Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Sumasri Guntupalli"'
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 10 (2017)
The molecular mechanisms underlying plastic changes in the strength and connectivity of excitatory synapses have been studied extensively for the past few decades and remain the most attractive cellular models of learning and memory. One of the major
Externí odkaz:
https://doaj.org/article/f7a7328a03c243f0a468ce0c177e3853
Publikováno v:
Neural Plasticity, Vol 2016 (2016)
Evidence from neuropathological, genetic, animal model, and biochemical studies has indicated that the accumulation of amyloid-beta (Aβ) is associated with, and probably induces, profound neuronal changes in brain regions critical for memory and cog
Externí odkaz:
https://doaj.org/article/d7733ef04d874ecd8bb677704603ab55
Autor:
Sumasri Guntupalli, Pojeong Park, Dae Hee Han, Mitchell Ringuet, Daniel G. Blackmore, Dhanisha J. Jhaveri, Frank Koentgen, Jocelyn Widagdo, Bong-Kiun Kaang, Victor Anggono
SUMMARYActivity-dependent changes in the number of AMPA-type glutamate receptors (AMPARs) at the synapse underpin the expression of long-term potentiation (LTP) and long-term depression (LTD), cellular correlates of learning and memory. Post-translat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8860d7a88bee2192acf6566ab70c1dcf
https://doi.org/10.1101/2022.07.27.501670
https://doi.org/10.1101/2022.07.27.501670
Publikováno v:
Cell Mol Neurobiol
Excitatory neurotransmission relies on the precise targeting of α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors to the neuronal plasma membrane. Activity-dependent ubiquitination of AMPA receptor (AMPAR) subunits
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e3fe50ce687ec563165005f0f20949cb
https://doi.org/10.1007/s10571-020-00809-2
https://doi.org/10.1007/s10571-020-00809-2
Autor:
Tianyi Zhu, Richard L. Huganir, Sumasri Guntupalli, Jocelyn Widagdo, Se Eun Jang, Victor Anggono
Publikováno v:
The Journal of Biological Chemistry
The accumulation of soluble amyloid-β (Aβ) peptides produces profound neuronal changes in the brain during the pathogenesis of Alzheimer's disease. Excessive levels of Aβ disrupt excitatory synaptic transmission by promoting the removal of synapti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2393eca9ddcda37d26a156372aba9ad7
http://europepmc.org/articles/PMC5437227
http://europepmc.org/articles/PMC5437227
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 10 (2017)
Frontiers in Molecular Neuroscience
Frontiers in Molecular Neuroscience
The molecular mechanisms underlying plastic changes in the strength and connectivity of excitatory synapses have been studied extensively for the past few decades and remain the most attractive cellular models of learning and memory. One of the major
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8e2dc4802dbec7cc5bfaf404201c92cd
http://journal.frontiersin.org/article/10.3389/fnmol.2017.00347/full
http://journal.frontiersin.org/article/10.3389/fnmol.2017.00347/full
Autor:
Swati Maitra, Bhanu Chandra Karisetty, Abhipradnya Bipin Wahul, Arvind Kumar, Ramya Garikapati, Nitin Khandelwal, Anusha Musalamadugu, Thatiparthi Jhansyrani, Sumasri Guntupalli, Sumana Chakravarty
Publikováno v:
Behavioural brain research. 318
Molecular mechanisms of depression-like pathophysiology in female rodent models are less reported compared to males, despite its higher prevalence in human females. Moreover, the stress-response in brain circuitries including reward and cognition cir
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3ef35596f1cb88f29cf3f99f2f5654e2
https://pubmed.ncbi.nlm.nih.gov/27780721
https://pubmed.ncbi.nlm.nih.gov/27780721
Publikováno v:
Neural Plasticity, Vol 2016 (2016)
Neural Plasticity
Neural Plasticity
Evidence from neuropathological, genetic, animal model, and biochemical studies has indicated that the accumulation of amyloid-beta (Aβ) is associated with, and probably induces, profound neuronal changes in brain regions critical for memory and cog
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::decffc289ac84c2a2f7c6b3e5e58d5f3
https://doaj.org/article/d7733ef04d874ecd8bb677704603ab55
https://doaj.org/article/d7733ef04d874ecd8bb677704603ab55