Quantitative assessment of glomerular basement membrane collagen IV α chains in paraffin sections from patients with focal segmental glomerulosclerosis and Alport gene variants.

Autor: Puapatanakul P; Division of Nephrology, Department of Medicine, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Isaranuwatchai S; Division of Nephrology, Department of Medicine, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand; Division of Nephrology, Department of Internal Medicine, Chulabhorn Hospital, Chulabhorn Royal Academy, Bangkok, Thailand., Chanakul A; Division of Nephrology, Department of Pediatrics, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand., Surintrspanont J; Department of Pathology, King Chulalongkorn Memorial Hospital, The Thai Red Cross Society, Bangkok, Thailand; Special Task Force for Activating Research, Department of Pathology, Chulalongkorn University, Bangkok, Thailand., Iampenkhae K; Department of Pathology, King Chulalongkorn Memorial Hospital, The Thai Red Cross Society, Bangkok, Thailand., Kanjanabuch T; Division of Nephrology, Department of Medicine, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand; Center of Excellence in Kidney Metabolic Disorders, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand., Suphapeetiporn K; Division of Medical Genetics and Metabolism, Center of Excellence for Medical Genomics, Department of Pediatrics, Medical Genomic Cluster, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand; Excellence Center for Genomics and Precision Medicine, King Chulalongkorn Memorial Hospital, The Thai Red Cross Society, Bangkok, Thailand., Charu V; Department of Pathology, Institute of Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, California, USA., Suleiman HY; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Praditpornsilpa K; Division of Nephrology, Department of Medicine, Faculty of Medicine, Chulalongkorn University, Bangkok, Thailand., Miner JH; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA. Electronic address: jeffminer@wustl.edu.

Publikováno v: Kidney international [Kidney Int] 2024 May; Vol. 105 (5), pp. 1049-1057. Date of Electronic Publication: 2024 Feb 22.

An ex vivo culture model of kidney podocyte injury reveals mechanosensitive, synaptopodin-templating, sarcomere-like structures.

Autor: Jiang S; NSF Science and Technology Center for Engineering Mechanobiology, Washington University in St. Louis, St. Louis, MO, USA.; Department of Mechanical Engineering and Materials Science, Washington University in St. Louis, St. Louis, MO, USA., Alisafaei F; NSF Science and Technology Center for Engineering Mechanobiology, Washington University in St. Louis, St. Louis, MO, USA.; Department of Mechanical and Industrial Engineering, New Jersey Institute of Technology, Newark, NJ, USA., Huang YY; Department of Mechanical Engineering and Materials Science, Washington University in St. Louis, St. Louis, MO, USA., Hong Y; NSF Science and Technology Center for Engineering Mechanobiology, Washington University in St. Louis, St. Louis, MO, USA.; Department of Mechanical Engineering and Materials Science, Washington University in St. Louis, St. Louis, MO, USA., Peng X; NSF Science and Technology Center for Engineering Mechanobiology, Washington University in St. Louis, St. Louis, MO, USA.; Department of Mechanical Engineering and Materials Science, Washington University in St. Louis, St. Louis, MO, USA., Qu C; NSF Science and Technology Center for Engineering Mechanobiology, Washington University in St. Louis, St. Louis, MO, USA.; Department of Mechanical Engineering and Materials Science, Washington University in St. Louis, St. Louis, MO, USA., Puapatanakul P; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USA., Jain S; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USA., Miner JH; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USA.; Department of Cell Biology and Physiology, Washington University School of Medicine, St. Louis, MO, USA., Genin GM; NSF Science and Technology Center for Engineering Mechanobiology, Washington University in St. Louis, St. Louis, MO, USA.; Department of Mechanical Engineering and Materials Science, Washington University in St. Louis, St. Louis, MO, USA., Suleiman HY; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, MO, USA.

Publikováno v: Science advances [Sci Adv] 2022 Sep 02; Vol. 8 (35), pp. eabn6027. Date of Electronic Publication: 2022 Aug 31.

Synaptopodin Is Dispensable for Normal Podocyte Homeostasis but Is Protective in the Context of Acute Podocyte Injury.

Autor: Ning L; Division of Nephrology, Washington University School of Medicine, St. Louis, Missouri., Suleiman HY; Division of Nephrology, Washington University School of Medicine, St. Louis, Missouri., Miner JH; Division of Nephrology, Washington University School of Medicine, St. Louis, Missouri minerj@wustl.edu.

Publikováno v: Journal of the American Society of Nephrology : JASN [J Am Soc Nephrol] 2020 Dec; Vol. 31 (12), pp. 2815-2832. Date of Electronic Publication: 2020 Sep 16.

Injury-induced actin cytoskeleton reorganization in podocytes revealed by super-resolution microscopy.

Autor: Suleiman HY; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Roth R; Washington University Center for Cellular Imaging, Washington University School of Medicine, St. Louis, Missouri, USA., Jain S; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Heuser JE; Institute for Integrated Cell-Material Sciences (iCeMS), Kyoto University, Kyoto, Japan., Shaw AS; Genentech, South San Francisco, California, USA., Miner JH; Division of Nephrology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA.

Publikováno v: JCI insight [JCI Insight] 2017 Aug 17; Vol. 2 (16). Date of Electronic Publication: 2017 Aug 17 (Print Publication: 2017).

The podocytes' inflammatory responses in experimental GN are independent of canonical MYD88-dependent toll-like receptor signaling.

Autor: Schömig, Thomas¹ (AUTHOR), Diefenhardt, Paul¹ (AUTHOR), Plagmann, Ingo¹ (AUTHOR), Trinsch, Bastian¹ (AUTHOR), Merz, Tim¹ (AUTHOR), Crispatzu, Giuliano^1,2 (AUTHOR), Unnersjö-Jess, David¹ (AUTHOR), Nies, Jasper¹ (AUTHOR), Pütz, David¹ (AUTHOR), Sierra Gonzalez, Claudio¹ (AUTHOR), Schermer, Bernhard^1,2 (AUTHOR), Benzing, Thomas^1,2 (AUTHOR), Brinkkoetter, Paul Thomas¹ (AUTHOR) paul.brinkkoetter@uk-koeln.de, Brähler, Sebastian¹ (AUTHOR) sebastian.braehler@uk-koeln.de

Publikováno v: Scientific Reports. 1/27/2024, Vol. 14 Issue 1, p1-12. 12p.