Zobrazeno 1 - 10
of 28
pro vyhledávání: '"Suganya, Rangaswamy"'
Autor:
Wicker, Christina A., Takiar, Vinita, Suganya, Rangaswamy, Arnold, Susanne M., Brill, Yolanda M., Chen, Li, Horbinski, Craig M., Napier, Dana, Valentino, Joseph, Kudrimoti, Mahesh R., Yu, Guoqiang, Izumi, Tadahide
Publikováno v:
In Oral Oncology December 2020 111
Autor:
Haibo Wang, Suganya Rangaswamy, Manohar Kodavati, Joy Mitra, Wenting Guo, Erika N. Guerrero, Ludo Van Den Bosch, Muralidhar L. Hegde
Publikováno v:
Molecular Brain, Vol 12, Iss 1, Pp 1-5 (2019)
Abstract Amyotrophic lateral sclerosis (ALS) is a degenerative motor neuron disease that has been linked to defective DNA repair. Many familial ALS patients harbor autosomal dominant mutations in the gene encoding the RNA/DNA binding protein ‘fused
Externí odkaz:
https://doaj.org/article/f086d9d77d8f4fba9e51017ac1b15cf7
Autor:
Suganya, Rangaswamy, Chakraborty, Anirban, Miriyala, Sumitra, Hazra, Tapas K., Izumi, Tadahide
Publikováno v:
In DNA Repair March 2015 27:40-48
Autor:
Ludo Van Den Bosch, Suganya Rangaswamy, Muralidhar L. Hegde, Erika N. Guerrero, Manohar Kodavati, Wenting Guo, Haibo Wang, Joy Mitra
Publikováno v:
Molecular Brain, Vol 12, Iss 1, Pp 1-5 (2019)
Molecular Brain
Molecular Brain
Amyotrophic lateral sclerosis (ALS) is a degenerative motor neuron disease that has been linked to defective DNA repair. Many familial ALS patients harbor autosomal dominant mutations in the gene encoding the RNA/DNA binding protein 'fused in sarcoma
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::61c8b9f81c18cd59cf4f7f663e154d95
https://doi.org/10.1186/s13041-019-0526-4
https://doi.org/10.1186/s13041-019-0526-4
Autor:
Haibo Wang, Suganya Rangaswamy, Manohar Kodavati, Mitra, Joy, Wenting Guo, Guerrero, Erika, Bosch, Ludo, Muralidhar Hegde
Additional file 1: Table S1. List of DNA repair genes screened by RT2 profiler array in FUS knockout and knockdown cell. The histogram shows the number of total genes and genes that are consistently down regulated or up regulated in both FUS KO and K
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::393e248b9709727c05fd0a10d2294a4e
Autor:
Haibo Wang, Erika N. Guerrero, K. S. Rao, Muralidhar L. Hegde, Suganya Rangaswamy, Pavana M. Hegde, Priyadarshini Basu, Joy Mitra
Publikováno v:
Hum Mol Genet
Dominant mutations in the RNA/DNA-binding protein TDP-43 have been linked to amyotrophic lateral sclerosis (ALS). Here, we screened genomic DNA extracted from spinal cord specimens of sporadic ALS patients for mutations in the TARDBP gene and identif
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b39af9418782bff11998d8e6d4403b5f
https://pubmed.ncbi.nlm.nih.gov/31504503
https://pubmed.ncbi.nlm.nih.gov/31504503
Publikováno v:
Genes, Vol 8, Iss 7, p 175 (2017)
Genes
Genes
Genomic fidelity in the humans is continuously challenged by genotoxic reactive oxygen species (ROS) generated both endogenously during metabolic processes, and by exogenous agents. Mispairing of most ROS-induced oxidized base lesions during DNA repl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9aa22ff375728c9bb860d06a753c866c
http://www.mdpi.com/2073-4425/8/7/175
http://www.mdpi.com/2073-4425/8/7/175
Akademický článek
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Significance: Reactive oxygen species (ROS) are generated by exogenous and environmental genotoxins, but also arise from mitochondria as byproducts of respiration in the body. ROS generate DNA damage of which pathological consequence, including cance
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::27abffa41f9f32c87b1aa83a97fe8583
https://europepmc.org/articles/PMC3960848/
https://europepmc.org/articles/PMC3960848/
Autor:
Suganya Rangaswamy, Bithika Dhar, Timothy L. Scott, Hansruedi Büeler, Lianteng Zhi, Tadahide Izumi
Publikováno v:
Cancer Research. 73:622-622
The mammalian apurinic/apyrimidinic endonuclease 1 (APE1/Ref1) is a DNA repair/redox regulator protein of which cytoplasmic accumulation has been associated with tumorigenesis and poor survival outcome. APE1 is degraded through the ubiquitin/proteaso
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::240337dae3f14aa42947158ae1a97913
https://doi.org/10.1158/1538-7445.am2013-622
https://doi.org/10.1158/1538-7445.am2013-622