Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Sue Monteith"'
Autor:
Yi Ping Bao, Joseph Cheriyan, Kaisa M. Mäki-Petäjä, Sharon M.L. Wallace, Sue Monteith, Carmel M. McEniery, Ian B. Wilkinson, Isla S. Mackenzie, Morris J. Brown
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology. 25(9)
Objective— Nitrates are used widely in clinical practice. However, the mechanism underlying the bioactivation of nitrates to release NO remains unclear. Recent animal data suggest that mitochondrial aldehyde dehydrogenase (ALDH2) plays a central ro
Autor:
Pankaj Sharma, Haiyan Jia, Sue Monteith, Chrysothemis Brown, Franco Ferraro, Morris J. Brown, Kevin M. O'Shaughnessy, Jennie Fatibene, David Clayton
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 35(6)
Abstract —We undertook a systematic search of the entire human genome with the affected sibling-pair model to identify major susceptibility loci to essential hypertension. Affected nuclear families (n=263) were recruited and divided according to de
Autor:
A Parsons, Aroon D. Hingorani, R Hopper, Morris J. Brown, Jenny Fatibene, Sue Monteith, Chia Fan Liang, Nigel G. Stephens, Kevin M. O'Shaughnessy, Amelia Lyon, Stephen F. Haydock
Publikováno v:
BASE-Bielefeld Academic Search Engine
Background —Endothelium-derived nitric oxide (NO) is synthesised from l -arginine by endothelial nitric oxide synthase (eNOS) encoded by the NOS 3 gene on chromosome 7. Because reduced NO synthesis has been implicated in the development of coronary
Publikováno v:
Clinical science (London, England : 1979). 88(5)
1. Chronic beta 1-adrenoceptor blocker therapy induces hyperresponsiveness of the beta 2-adrenoceptor in human atrium. To investigate whether the beta 2-adrenoceptor sensitization induced by beta 1-adrenoceptor blockade is associated with altered gen
Publikováno v:
Clinical Science. 88:24P-24P
Publikováno v:
Clinical Science. 81:14P-14P