Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Subhash M. Agarwal"'
Publikováno v:
ACS Omega, Vol 9, Iss 4, Pp 4528-4539 (2024)
Externí odkaz:
https://doaj.org/article/cf3ed1727b6e468da9298f5be942e20e
Publikováno v:
RSC advances. 12(26)
Double mutated epidermal growth factor receptor is a clinically important target for addressing drug resistance in lung cancer treatment. Therefore, discovering new inhibitors against the T790M/L858R (TMLR) resistant mutation is ongoing globally. In
Publikováno v:
Genes; Volume 14; Issue 3; Pages: 741
Endometrial cancer (EC) is among the most common gynecological disorders globally. As single nucleotide polymorphisms (SNPs) play an important role in the causation of EC, therefore, a comprehensive meta-analysis of 49 SNPs covering 25,446 cases and
Publikováno v:
Genomics. 114(3)
To study the risk of polymorphisms present in the non-coding regions of genes related with cervical cancer.The PubMed database was extensively searched using text-mining techniques to identify literature containing the association of single nucleotid
Autor:
Jagat Singh Chauhan, Sandeep Kumar Dhanda, Deepak Singla, Open Source Drug Discovery Consortium, Subhash M Agarwal, Gajendra P S Raghava
Publikováno v:
PLoS ONE, Vol 9, Iss 7, p e101079 (2014)
Overexpression of EGFR is responsible for causing a number of cancers, including lung cancer as it activates various downstream signaling pathways. Thus, it is important to control EGFR function in order to treat the cancer patients. It is well estab
Externí odkaz:
https://doaj.org/article/e7de3fb6c3244acc963798844644b989
Publikováno v:
Cytokine. 157
Cervical cancer is a leading women cancer globally with respect to both incidence and mortality. Its increased risk has been linked with HPV infection and genetic variations like single nucleotide polymorphisms (SNPs). Although, studies have been pub
Autor:
Akshit Arora, Saurav Gera, Tanuj Maheshwari, Dhwani Raghav, Md Jahoor Alam, R K Brojen Singh, Subhash M Agarwal
Publikováno v:
PLoS ONE, Vol 8, Iss 2, p e52736 (2013)
We construct a stress p53-Mdm2-p300-HDAC1 regulatory network that is activated and stabilised by two regulatory proteins, p300 and HDAC1. Different activation levels of [Formula: see text] observed due to these regulators during stress condition have
Externí odkaz:
https://doaj.org/article/b4b5a54d627b4b078ed95e3045fbfa3d