Zobrazeno 1 - 9 of 9 pro vyhledávání: '"Suad Alateeq"'
1
Akademický článek
Mitochondrial pathways in cancer
Autor: Suad Alateeq
Publikováno v: Saudi Journal of Medicine and Medical Sciences, Vol 8, Iss 1, Pp 1-2 (2020)
Externí odkaz: https://doaj.org/article/a74d940c9775415f92afca4f56990b5d
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2
Akademický článek
Identification of on-target mutagenesis during correction of a beta-thalassemia splice mutation in iPS cells with optimised CRISPR/Cas9-double nickase reveals potential safety concerns
Autor: Suad Alateeq, Dmitry Ovchinnikov, Timothy Tracey, Deanne Whitworth, Abdullah Al-Rubaish, Amein Al-Ali, Ernst Wolvetang
Publikováno v: APL Bioengineering, Vol 2, Iss 4, Pp 046103-046103-18 (2018)
Precise and accurate gene correction is crucial for enabling iPSC-based therapies, and Cas9-Nickase based approaches are increasingly considered for in vivo correction of diseases such as beta-thalassemia. Here, we generate footprint-free induced plu
Externí odkaz: https://doaj.org/article/80596bb0d4fd40f4acd855c999f2bb65
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3
Direct Comparison of Four Hematopoietic Differentiation Methods from Human Induced Pluripotent Stem Cells
Autor: Timothy J. Molloy, Ernst J. Wolvetang, Melinda L. Tursky, David D.F. Ma, Helen Tao, Suad Alateeq, To Ha Loi, Crisbel M. Artuz
Publikováno v: Stem Cell Reports
Summary Induced pluripotent stem cells (iPSCs) are an invaluable resource for the study of human disease. However, there are no standardized methods for differentiation into hematopoietic cells, and there is a lack of robust, direct comparisons of di
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::747e89915675680b11916934fdb484a2
https://doi.org/10.1016/j.stemcr.2020.07.009
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5
Identification of on-target mutagenesis during correction of a beta-thalassemia splice mutation in iPS cells with optimised CRISPR/Cas9-double nickase reveals potential safety concerns
Autor: Timothy J. Tracey, Ernst J. Wolvetang, Abdullah M. Al-Rubaish, Deanne J. Whitworth, Suad Alateeq, Amein K. Al-Ali, Dmitry A. Ovchinnikov
Publikováno v: APL Bioengineering
APL Bioengineering, Vol 2, Iss 4, Pp 046103-046103-18 (2018)
Precise and accurate gene correction is crucial for enabling iPSC-based therapies, and Cas9-Nickase based approaches are increasingly considered for in vivo correction of diseases such as beta-thalassemia. Here, we generate footprint-free induced plu
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::207d883d4ac893627f72e84c9a54389a
http://europepmc.org/articles/PMC6481731
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6
Advances in Reprogramming to Pluripotency
Autor: Ernst J. Wolvetang, Suad Alateeq, Patrick R.J. Fortuna
Publikováno v: Scopus-Elsevier
Ernst Wolvetang
ResearcherID
Pluripotent stem cells (PSCs) derived from somatic cells represent a powerful experimental tool for investigating the molecular mechanisms underlying the disease phenotype; with prospects to advance medical therapies. They also have significant poten
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::93c41a6468d9db7fe0b9f227f0cb3996
https://doi.org/10.2174/1574888x10666150220154820
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7
CRISPR/Cas9-mediated traceless gene correction and activation of the HBB locus in iPSCs with the beta thalassaemia mutation
Autor: Suad Alateeq
Beta thalassaemia is caused by mutations in the adult haemoglobin gene (HBB) and is one of the most prevalent monogenic blood disorders worldwide. The transplantation of haematopoietic stem cells derived from gene-corrected patient induced pluripoten
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::83b06c62be2e7efd3c5fe12f631c9b82
https://doi.org/10.14264/uql.2017.62
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8
Polymorphism in methylenetetrahydrofolate reductase, plasminogen activator inhibitor-1, and apolipoprotein E in hemodialysis patients
Autor: Al-Muhanna, F., Al-Mueilo, S., Al-Ali, A., Larbi, E., Rubaish, A., Abdulmohsen, M. F., Al-Zahrani, A., Suad Alateeq
Publikováno v: Scopus-Elsevier
Saudi Journal of Kidney Diseases and Transplantation, Vol 19, Iss 6, Pp 937-941 (2008)
The methylenetetrahydrofolate reductase (MTHFR) gene polymorphism, apolipoprotein E (apo s4) gene polymorphism and polymorphism of plasminogen activator inhibitor-1 (PAI-1) have been shown to be associated with end-stage renal disease (ESRD). To dete
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::89cda262f6dedf3de13ecf987a4db708
http://www.scopus.com/inward/record.url?eid=2-s2.0-59649088352&partnerID=MN8TOARS
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