Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Su-Bin Seong"'
Publikováno v:
Kidney Research and Clinical Practice, Vol 42, Iss 2, Pp 202-215 (2023)
Background Angiotensin II induces glomerular and podocyte injury via systemic and local vasoconstrictive or non-hemodynamic effects including oxidative stress. The release of reactive oxygen species (ROS) from podocytes may participate in the develop
Externí odkaz:
https://doaj.org/article/e2550ce7b8d04d2799c533fb6794f883
Publikováno v:
Cellular Physiology and Biochemistry, Vol 53, Iss 5, Pp 747-759 (2019)
Externí odkaz:
https://doaj.org/article/29f91d53a7dd43cab88bbe0b6fbdd808
Publikováno v:
Childhood Kidney Diseases, Vol 22, Iss 1, Pp 22-27 (2018)
Purpose Podocytes are important architectures that maintain the crucial roles of glomerular filtration barrier functions. Despite this structural importance, however, the mechanisms of the changes in podocytes that can be an important pathogenesis of
Externí odkaz:
https://doaj.org/article/77ec98c5d93a4ab5bf1baff6ba72d480
Publikováno v:
Journal of Ginseng Research, Vol 38, Iss 4, Pp 233-238 (2014)
Background: The actin cytoskeleton in podocytes is essential for the maintenance of its normal structure and function. Its disruption is a feature of podocyte foot-process effacement and is associated with proteinuria. α-Actinin-4 in podocytes serve
Externí odkaz:
https://doaj.org/article/1447a631746a4e2e8e1de6ae6ea7a1eb
Background: Interferon-gamma (IFN-γ) release assays (IGRAs) are useful for the diagnosis of Mycobacterium tuberculosis infection. Current IGRAs use either enzyme-linked immunosorbent assay (ELISA) or enzyme-linked immunospot assay, which require com
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::98ece68c2299c6f75a072d035fcb5e20
https://doi.org/10.21203/rs.3.rs-20838/v1
https://doi.org/10.21203/rs.3.rs-20838/v1
Publikováno v:
Childhood Kidney Diseases. 22:22-27
Publikováno v:
The International Journal of Biochemistry & Cell Biology. 79:370-381
Angiotensin II (Ang II) works as a paracrine or autocrine cytokine agent to regulate renal functions and promotes podocytes dysfunction directly or indirectly, causing proteinuria. The glomerular slit diaphragm (SD) serves as a size-selective barrier
Publikováno v:
Experimental Cell Research. 340:139-149
Puromycin aminonucleoside (PAN)-induced nephrosis is a widely studied animal model of human idiopathic nephrotic syndrome because PAN injection into rats results in increased glomerular permeability with the characteristic ultrastructural changes in
Publikováno v:
Journal of Ginseng Research
Journal of Ginseng Research, Vol 38, Iss 4, Pp 233-238 (2014)
Journal of Ginseng Research, Vol 38, Iss 4, Pp 233-238 (2014)
Background: The actin cytoskeleton in podocytes is essential for the maintenance of its normal structure and function. Its disruption is a feature of podocyte foot-process effacement and is associated with proteinuria. α-Actinin-4 in podocytes serve
Publikováno v:
Ha, T-S, Nam, J A, Seong, S-B, Saleem, M A, Park, S J & Shin, J I 2017, ' Montelukast improves the changes of cytoskeletal and adaptor proteins of human podocytes by interleukin-13 ', Inflammation Research, vol. 66, no. 9, pp. 793-802 . https://doi.org/10.1007/s00011-017-1058-y
OBJECTIVE AND DESIGN: Interleukin-13 (IL-13) has recently been reported to be a potential cytokine in the pathogenesis of minimal-change nephrotic syndrome (MCNS). However, the mechanistic insights associated with podocyte dysfunction mediated by IL-